Chemokine receptor expression on human eosinophils from peripheral blood and bronchoalveolar lavage fluid after segmental antigen challenge.
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[1] P. Foster,et al. Chemokines in asthma: cooperative interaction between chemokines and IL-13. , 2003, The Journal of allergy and clinical immunology.
[2] D. Taub,et al. Stem cell factor induces eosinophil activation and degranulation: mediator release and gene array analysis. , 2002, Blood.
[3] Lin Ying Liu,et al. Decreased Expression of Membrane IL-5 Receptor α on Human Eosinophils: I. Loss of Membrane IL-5 Receptor α on Airway Eosinophils and Increased Soluble IL-5 Receptor α in the Airway After Allergen Challenge1 , 2002, The Journal of Immunology.
[4] Dominique Schols,et al. AMD3100, a CxCR4 antagonist, attenuates allergic lung inflammation and airway hyperreactivity. , 2002, The American journal of pathology.
[5] R. Geha,et al. CCR3 is essential for skin eosinophilia and airway hyperresponsiveness in a murine model of allergic skin inflammation. , 2002, The Journal of clinical investigation.
[6] D. Friend,et al. The murine CCR3 receptor regulates both the role of eosinophils and mast cells in allergen-induced airway inflammation and hyperresponsiveness , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[7] K. Ohta,et al. Chemokine receptor expression profile of eosinophils at inflamed tissue sites: Decreased CCR3 and increased CXCR4 expression by lung eosinophils. , 2001, The Journal of allergy and clinical immunology.
[8] A. Kaser,et al. Functional expression of chemokine receptor 2 by normal human eosinophils. , 2001, The Journal of allergy and clinical immunology.
[9] S. Lira,et al. Requirement for the Chemokine Receptor Ccr6 in Allergic Pulmonary Inflammation , 2001, The Journal of experimental medicine.
[10] E. Israel,et al. Eotaxin expression after segmental allergen challenge in subjects with atopic asthma. , 2001, American journal of respiratory and critical care medicine.
[11] R. Breuer,et al. Eotaxin-3 but not eotaxin gene expression is upregulated in asthmatics 24 hours after allergen challenge. , 2001, American journal of respiratory cell and molecular biology.
[12] C. Bandeira‐Melo,et al. Eotaxins. Contributing to the diversity of eosinophil recruitment and activation. , 2001, American journal of respiratory cell and molecular biology.
[13] Y. Kato,et al. Chemokines induce eosinophil degranulation through CCR-3. , 2000, The Journal of allergy and clinical immunology.
[14] W. Busse,et al. Inhaled budesonide decreases airway inflammatory response to allergen. , 2000, American journal of respiratory and critical care medicine.
[15] Sha Quan,et al. CXCR3 Expression and Activation of Eosinophils: Role of IFN-γ-Inducible Protein-10 and Monokine Induced by IFN-γ1 , 2000, The Journal of Immunology.
[16] Kazuhiko Yamamoto,et al. Expression of CXCR4 in Eosinophils: Functional Analyses and Cytokine-Mediated Regulation1 , 2000, The Journal of Immunology.
[17] C. Murdoch,et al. Chemokine receptors and their role in inflammation and infectious diseases. , 2000, Blood.
[18] P. Weller,et al. Lymph node trafficking and antigen presentation by endobronchial eosinophils. , 2000, The Journal of clinical investigation.
[19] R. Nickel,et al. Chemokines and allergic disease. , 1999, The Journal of allergy and clinical immunology.
[20] K. Ohta,et al. Eosinophil chemotaxis by chemokines: a study by a simple photometric assay , 1999, Allergy.
[21] I. Pavord,et al. Increases in eotaxin‐positive cells in induced sputum from atopic asthmatic subjects after inhalational allergen challenge , 1999, Allergy.
[22] M. Rothenberg,et al. CC Chemokine Receptor-3 Undergoes Prolonged Ligand-induced Internalization* , 1999, The Journal of Biological Chemistry.
[23] S. Bel,et al. Differential regulation of eosinophil chemokine signaling via CCR3 and non-CCR3 pathways. , 1999, Journal of immunology.
[24] O. Götze,et al. The Biologic Role of Interleukin-8: Functional Analysis and Expression of CXCR1 and CXCR2 on Human Eosinophils , 1999 .
[25] A. Bellini,et al. Kinetics of eotaxin expression and its relationship to eosinophil accumulation and activation in bronchial biopsies and bronchoalveolar lavage (BAL) of asthmatic patients after allergen inhalation , 1998, Clinical and experimental immunology.
[26] S. Holgate,et al. Release of RANTES, MIP-1 alpha, and MCP-1 into asthmatic airways following endobronchial allergen challenge. , 1997, American journal of respiratory and critical care medicine.
[27] C. Mackay,et al. Chemokine receptor usage by human eosinophils. The importance of CCR3 demonstrated using an antagonistic monoclonal antibody. , 1997, The Journal of clinical investigation.
[28] H. Kita,et al. Eosinophil recruitment is associated with IL-5, but not with RANTES, twenty-four hours after allergen challenge. , 1996, The Journal of allergy and clinical immunology.
[29] J. Demartino,et al. Cloning, expression, and characterization of the human eosinophil eotaxin receptor , 1996, The Journal of experimental medicine.
[30] E. Butcher,et al. L-selectin and very late antigen-4 integrin promote eosinophil rolling at physiological shear rates in vivo. , 1994, Journal of immunology.
[31] P. Zanen,et al. RANTES- and interleukin-8-induced responses in normal human eosinophils: effects of priming with interleukin-5. , 1994, Blood.
[32] W. Busse,et al. Increased airway inflammation with segmental versus aerosol antigen challenge. , 1993, The American review of respiratory disease.