The extracellular adherence protein from Staphylococcus aureus abrogates angiogenic responses of endothelial cells by blocking Ras activation
暂无分享,去创建一个
K. Preissner | V. Orlova | M. Hussain | T. Chavakis | M. Herrmann | Uwe Schubert | G. Thiel | Athanasios N Athanasopoulos | Astrid C. S. Sobke | D. Selimovic | M. Hassan | Athanasios N. Athanasopoulos
[1] H. Augustin,et al. The extracellular adherence protein (Eap) of Staphylococcus aureus inhibits wound healing by interfering with host defense and repair mechanisms. , 2006, Blood.
[2] C. Wolz,et al. Role of Staphylococcus aureus Global Regulators sae and σB in Virulence Gene Expression during Device-Related Infection , 2005, Infection and Immunity.
[3] C. Wolz,et al. sae is essential for expression of the staphylococcal adhesins Eap and Emp. , 2005, Microbiology.
[4] U. Felbor,et al. Endostatin's heparan sulfate-binding site is essential for inhibition of angiogenesis and enhances in situ binding to capillary-like structures in bone explants. , 2005, Matrix biology : journal of the International Society for Matrix Biology.
[5] W. Stanford,et al. A Vascular Gene Trap Screen Defines RasGRP3 as an Angiogenesis-Regulated Gene Required for the Endothelial Response to Phorbol Esters , 2004, Molecular and Cellular Biology.
[6] J. Holgersson,et al. The Extracellular Adherence Protein from Staphylococcus aureus Inhibits Neutrophil Binding to Endothelial Cells , 2004, Infection and Immunity.
[7] C. Wolz,et al. Regulatory and genomic plasticity of Staphylococcus aureus during persistent colonization and infection. , 2004, International journal of medical microbiology : IJMM.
[8] M. Poncz,et al. Interactions of platelet factor 4 with the vessel wall. , 2004, Seminars in Thrombosis and Hemostasis.
[9] Jo Ann Buczek-Thomas,et al. Heparan sulphate proteoglycans modulate fibroblast growth factor-2 binding through a lipid raft-mediated mechanism. , 2004, The Biochemical journal.
[10] K. Harding,et al. Bacteria and wound healing , 2004, Current opinion in infectious diseases.
[11] I. Hwang,et al. Localization of VEGFR-2 and PLD2 in endothelial caveolae is involved in VEGF-induced phosphorylation of MEK and ERK. , 2004, American journal of physiology. Heart and circulatory physiology.
[12] R. Hebbel,et al. Binding and displacement of vascular endothelial growth factor (VEGF) by thrombospondin: Effect on human microvascular endothelial cell proliferation and angiogenesis , 2004, Angiogenesis.
[13] Hua-Lin Wu,et al. Angiostatin antagonizes the action of VEGF-A in human endothelial cells via two distinct pathways. , 2003, Biochemical and biophysical research communications.
[14] S. Mousa,et al. Signaling through JAM-1 and αvβ3 is required for the angiogenic action of bFGF: dissociation of the JAM-1 and αvβ3 complex , 2003 .
[15] S. Robbins,et al. Fibroblast Growth Factor-2-induced Signaling through Lipid Raft-associated Fibroblast Growth Factor Receptor Substrate 2 (FRS2)* , 2003, The Journal of Biological Chemistry.
[16] K. Nakagawa,et al. Functional role of Egr-1 mediating VEGF-induced tissue factor expression in the retinal capillary endothelium , 2002, Graefe's Archive for Clinical and Experimental Ophthalmology.
[17] E. Brown,et al. The Staphylococcus aureus Map protein is an immunomodulator that interferes with T cell-mediated responses. , 2002, The Journal of clinical investigation.
[18] Weicheng Wu,et al. Sphingosine Kinase Mediates Vascular Endothelial Growth Factor-Induced Activation of Ras and Mitogen-Activated Protein Kinases , 2002, Molecular and Cellular Biology.
[19] M. Bryckaert,et al. Platelet factor 4 inhibits FGF2-induced endothelial cell proliferation via the extracellular signal-regulated kinase pathway but not by the phosphatidylinositol 3-kinase pathway. , 2002, Blood.
[20] Walter Kolch,et al. Untying the regulation of the Raf-1 kinase. , 2002, Archives of biochemistry and biophysics.
[21] K. Preissner,et al. Staphylococcus aureus extracellular adherence protein serves as anti-inflammatory factor by inhibiting the recruitment of host leukocytes , 2002, Nature Medicine.
[22] D. Haskard,et al. bFGF and VEGF synergistically enhance endothelial cytoprotection via decay-accelerating factor induction. , 2002, American journal of physiology. Cell physiology.
[23] J. Lawler,et al. Thrombospondin‐1 as an endogenous inhibitor of angiogenesis and tumor growth , 2002, Journal of cellular and molecular medicine.
[24] L. Claesson‐Welsh,et al. VEGF receptor signal transduction. , 2003, Science's STKE : signal transduction knowledge environment.
[25] J. Schrenzel,et al. Identification and Characterization of a Novel 38.5-Kilodalton Cell Surface Protein of Staphylococcus aureus with Extended-Spectrum Binding Activity for Extracellular Matrix and Plasma Proteins , 2001, Journal of bacteriology.
[26] K. Becker,et al. Analogs of Eap Protein Are Conserved and Prevalent in Clinical Staphylococcus aureus Isolates , 2001, Clinical Diagnostic Laboratory Immunology.
[27] K. Shitara,et al. Heterogeneity of the signal transduction pathways for VEGF‐induced MAPKs activation in human vascular endothelial cells , 2001, Journal of cellular physiology.
[28] D. Mukhopadhyay,et al. Cell surface glypicans are low-affinity endostatin receptors. , 2001, Molecular cell.
[29] Sathyanarayana,et al. Antibody neutralization of vascular endothelial growth factor inhibits wound granulation tissue formation. , 2001, The Journal of surgical research.
[30] C. Hecquet,et al. Two distinct signalling pathways are involved in FGF2-stimulated proliferation of choriocapillary endothelial cells: A comparative study with VEGF , 2001, Oncogene.
[31] I. Zachary,et al. Vascular endothelial growth factor-induced prostacyclin production is mediated by a protein kinase C (PKC)-dependent activation of extracellular signal-regulated protein kinases 1 and 2 involving PKC-delta and by mobilization of intracellular Ca2+. , 2001, The Biochemical journal.
[32] T. Veikkola,et al. Interaction of endostatin with integrins implicated in angiogenesis. , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[33] T. Sasaki,et al. Endostatin-induced tyrosine kinase signaling through the Shb adaptor protein regulates endothelial cell apoptosis. , 2000, Blood.
[34] A. Jahreis,et al. Effects of two novel cationic staphylococcal proteins (NP‐taseand p70) and enterotoxin B on IgE synthesis and interleukin‐4 and interferon‐γ production in patients with atopic dermatitis , 2000, The British journal of dermatology.
[35] W. Brodbeck,et al. Decay-accelerating factor: CD55, DAF , 2000 .
[36] J. Martial,et al. 16K human prolactin inhibits vascular endothelial growth factor-induced activation of Ras in capillary endothelial cells. , 1999, Molecular endocrinology.
[37] M. Shibuya,et al. VEGF activates protein kinase C-dependent, but Ras-independent Raf-MEK-MAP kinase pathway for DNA synthesis in primary endothelial cells , 1999, Oncogene.
[38] R. Soldi,et al. Role of αvβ3 integrin in the activation of vascular endothelial growth factor receptor‐2 , 1999, The EMBO journal.
[39] P. Francioli,et al. Prevalence and risk factors for nosocomial infections in four university hospitals in Switzerland. , 1999, Infection control and hospital epidemiology.
[40] H. Granger,et al. Protein Kinase G Mediates Vascular Endothelial Growth Factor-induced Raf-1 Activation and Proliferation in Human Endothelial Cells* , 1998, The Journal of Biological Chemistry.
[41] R. Gamelli,et al. Vascular endothelial growth factor mediates angiogenic activity during the proliferative phase of wound healing. , 1998, The American journal of pathology.
[42] A. Bikfalvi,et al. Platelet factor 4 modulates fibroblast growth factor 2 (FGF-2) activity and inhibits FGF-2 dimerization. , 1998, Blood.
[43] S. Batsford,et al. Glomerular injury induced by cationic 70‐kD staphylococcal protein; specific immune response is not involved in early phase in rats , 1998, The Journal of pathology.
[44] R. Klemke,et al. Integrin αvβ3 Requirement for Sustained Mitogen-activated Protein Kinase Activity during Angiogenesis , 1998, The Journal of cell biology.
[45] A. Ljungh,et al. Staphylococci Bind Heparin-Binding Host Growth Factors , 1996, Current Microbiology.
[46] V Nehls,et al. A novel, microcarrier-based in vitro assay for rapid and reliable quantification of three-dimensional cell migration and angiogenesis. , 1995, Microvascular research.
[47] G. Neufeld,et al. Platelet Factor-4 Inhibits the Mitogenic Activity of VEGF121 and VEGF165 Using Several Concurrent Mechanisms (*) , 1995, The Journal of Biological Chemistry.
[48] A. Vogt,et al. Staphylococcal neutral phosphatase , 1994 .
[49] A. Vogt,et al. Staphylococcal neutral phosphatase. A highly cationic molecule with binding properties for immunoglobulin. , 1994, APMIS : acta pathologica, microbiologica, et immunologica Scandinavica.
[50] R. Gaynes,et al. An overview of nosocomial infections, including the role of the microbiology laboratory , 1993, Clinical Microbiology Reviews.
[51] R. Sosnowski,et al. Interference with endogenous ras function inhibits cellular responses to wounding , 1993, Journal of Cell Biology.
[52] R. Gaynes,et al. Major trends in the microbial etiology of nosocomial infection. , 1991, The American journal of medicine.
[53] J. Herbert,et al. Tumor-promoting phorbol esters stimulate bovine cerebral cortex capillary endothelial cell growth in vitro. , 1989, Biochemical and biophysical research communications.
[54] P. Blackshear,et al. Tumor-promoting phorbol esters induce angiogenesis in vivo. , 1988, The American journal of physiology.
[55] A. Schor,et al. The isolation and culture of endothelial cells and pericytes from the bovine retinal microvasculature: a comparative study with large vessel vascular cells. , 1986, Microvascular research.
[56] L. Orci,et al. Tumor-promoting phorbol esters induce angiogenesis in vitro , 1985, Cell.
[57] E. Jaffe,et al. Culture of human endothelial cells derived from umbilical veins. Identification by morphologic and immunologic criteria. , 1973, The Journal of clinical investigation.
[58] I. Zachary,et al. Vascular endothelial growth factor-induced prostacyclin production is mediated by a protein kinase C ( PKC )-dependent activation of extracellular signal-regulated protein kinases 1 and 2 involving PKC-δ and by mobilization of intracellular Ca 2 + , 2022 .