AhR and Arnt differentially regulate NF-κB signaling and chemokine responses in human bronchial epithelial cells
暂无分享,去创建一个
M. Lag | P. Schwarze | D. Lagadic-Gossmann | D. Gilot | M. Refsnes | R. Becher | J. Øvrevik | V. Lecureur | J. Holme | T. Skuland
[1] O. Fardel,et al. Calcium signaling and β2-adrenergic receptors regulate 1-nitropyrene induced CXCL8 responses in BEAS-2B cells. , 2014, Toxicology in vitro : an international journal published in association with BIBRA.
[2] A. Puga,et al. The Ah receptor recruits IKKα to its target binding motifs to phosphorylate serine-10 in histone H3 required for transcriptional activation. , 2014, Toxicological sciences : an official journal of the Society of Toxicology.
[3] A. Hoffmann,et al. Cross-talk between Aryl Hydrocarbon Receptor and the Inflammatory Response , 2013, The Journal of Biological Chemistry.
[4] Dalei Wu,et al. Aryl hydrocarbon receptor signaling regulates NF-κB RelB activation during dendritic-cell differentiation , 2013, Immunology and cell biology.
[5] M. Lag,et al. Mechanisms of chemokine responses by polycyclic aromatic hydrocarbons in bronchial epithelial cells: sensitization through toll-like receptor-3 priming. , 2013, Toxicology letters.
[6] M. Lag,et al. Differential chemokine induction by 1-nitropyrene and 1-aminopyrene in bronchial epithelial cells: importance of the TACE/TGF-α/EGFR-pathway. , 2013, Environmental toxicology and pharmacology.
[7] J. Lafitte,et al. Xenobiotic Metabolism and Disposition in Human Lung Cell Models: Comparison with In Vivo Expression Profiles , 2012, Drug Metabolism and Disposition.
[8] J. T. Chang,et al. Aryl hydrocarbon receptor in association with RelA modulates IL-6 expression in non-smoking lung cancer , 2012, Oncogene.
[9] Benjamin P. Seaver,et al. Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis. , 2012, Toxicological sciences : an official journal of the Society of Toxicology.
[10] M. Lag,et al. Differential effects of the particle core and organic extract of diesel exhaust particles. , 2012, Toxicology letters.
[11] A. Pivarcsi,et al. MicroRNAs in inflammation and response to injuries induced by environmental pollution. , 2011, Mutation research.
[12] J. Schneider,et al. RNA expressions of AHR, ARNT and CYP1B1 are influenced by AHR Arg554Lys polymorphism. , 2011, Molecular genetics and metabolism.
[13] Dalei Wu,et al. Interaction of aryl hydrocarbon receptor and NF-κB subunit RelB in breast cancer is associated with interleukin-8 overexpression. , 2011, Archives of biochemistry and biophysics.
[14] Dalei Wu,et al. AhR deficiency impairs expression of LPS-induced inflammatory genes in mice. , 2011, Biochemical and biophysical research communications.
[15] M. Lag,et al. TACE/TGF-α/EGFR regulates CXCL8 in bronchial epithelial cells exposed to particulate matter components , 2011, European Respiratory Journal.
[16] R. Scrivo,et al. Inflammation as "common soil" of the multifactorial diseases. , 2011, Autoimmunity reviews.
[17] Ann Richmond,et al. Chemokines in health and disease. , 2011, Experimental cell research.
[18] Y. Fujii‐Kuriyama,et al. A Role of the Aryl Hydrocarbon Receptor in Attenuation of Colitis , 2011, Digestive Diseases and Sciences.
[19] Shao-Cong Sun,et al. Non-canonical NF-κB signaling pathway , 2011, Cell Research.
[20] Youhua Liu,et al. Inhibition of Proinflammatory RANTES Expression by TGF-β1 Is Mediated by Glycogen Synthase Kinase-3β-dependent β-Catenin Signaling* , 2010, The Journal of Biological Chemistry.
[21] M. Böhm,et al. The cardiopulmonary continuum systemic inflammation as 'common soil' of heart and lung disease. , 2010, International journal of cardiology.
[22] A. Fasano,et al. Novel signaling interactions between proteinase-activated receptor 2 and Toll-like receptors in vitro and in vivo , 2010, Mucosal Immunology.
[23] M. Lag,et al. Differential effects of nitro-PAHs and amino-PAHs on cytokine and chemokine responses in human bronchial epithelial BEAS-2B cells. , 2010, Toxicology and applied pharmacology.
[24] M. Lag,et al. Signalling pathways involved in 1-nitropyrene (1-NP)-induced and 3-nitrofluoranthene (3-NF)-induced cell death in Hepa1c1c7 cells. , 2009, Mutagenesis.
[25] Y. Fujii‐Kuriyama,et al. Hypersensitivity of Aryl Hydrocarbon Receptor-Deficient Mice to Lipopolysaccharide-Induced Septic Shock , 2009, Molecular and Cellular Biology.
[26] Y. Fujii‐Kuriyama,et al. Aryl hydrocarbon receptor in combination with Stat1 regulates LPS-induced inflammatory responses , 2009, The Journal of experimental medicine.
[27] M. Lag,et al. Cytokine and chemokine expression patterns in lung epithelial cells exposed to components characteristic of particulate air pollution. , 2009, Toxicology.
[28] D. Leduc,et al. Anthrax Lethal Toxin Impairs IL-8 Expression in Epithelial Cells through Inhibition of Histone H3 Modification , 2009, PLoS pathogens.
[29] K. Hensley,et al. Inhalation of environmental stressors & chronic inflammation: autoimmunity and neurodegeneration. , 2009, Mutation research.
[30] Fumio Matsumura,et al. A new cross-talk between the aryl hydrocarbon receptor and RelB, a member of the NF-kappaB family. , 2009, Biochemical pharmacology.
[31] A. Bhatnagar. Could dirty air cause diabetes? , 2009, Circulation.
[32] R. Sapolsky. Faculty Opinions recommendation of The aryl hydrocarbon nuclear translocator alters CD30-mediated NF-kappaB-dependent transcription. , 2009 .
[33] C. Duckett,et al. The Aryl Hydrocarbon Nuclear Translocator Alters CD30-Mediated NF-κB–Dependent Transcription , 2009, Science.
[34] T. Thatcher,et al. The Aryl Hydrocarbon Receptor Attenuates Tobacco Smoke-induced Cyclooxygenase-2 and Prostaglandin Production in Lung Fibroblasts through Regulation of the NF-κB Family Member RelB* , 2008, Journal of Biological Chemistry.
[35] S. Momohara,et al. A role for the aryl hydrocarbon receptor and the dioxin TCDD in rheumatoid arthritis. , 2008, Rheumatology.
[36] O. Fardel,et al. Interleukin-8 induction by the environmental contaminant benzo(a)pyrene is aryl hydrocarbon receptor-dependent and leads to lung inflammation. , 2008, Toxicology letters.
[37] F. Matsumura,et al. RelB, a new partner of aryl hydrocarbon receptor-mediated transcription. , 2007, Molecular endocrinology.
[38] M. Mathieu,et al. Differential regulation of RANTES and IL-8 expression in lung adenocarcinoma cells. , 2007, Lung cancer.
[39] D. Lagadic-Gossmann,et al. Effects of nitrated-polycyclic aromatic hydrocarbons and diesel exhaust particle extracts on cell signalling related to apoptosis: possible implications for their mutagenic and carcinogenic effects. , 2007, Toxicology.
[40] T. Thatcher,et al. Aryl Hydrocarbon Receptor-Deficient Mice Develop Heightened Inflammatory Responses to Cigarette Smoke and Endotoxin Associated with Rapid Loss of the Nuclear Factor-κB Component RelB , 2007 .
[41] T. Thatcher,et al. Aryl hydrocarbon receptor-deficient mice develop heightened inflammatory responses to cigarette smoke and endotoxin associated with rapid loss of the nuclear factor-kappaB component RelB. , 2007, The American journal of pathology.
[42] M. Lag,et al. Mechanisms of silica-induced IL-8 release from A549 cells: initial kinase-activation does not require EGFR activation or particle uptake. , 2006, Toxicology.
[43] M. Kawaguchi,et al. Synthetic double‐stranded RNA induces multiple genes related to inflammation through Toll‐like receptor 3 depending on NF‐κB and/or IRF‐3 in airway epithelial cells , 2006, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[44] H. Aburatani,et al. Constitutive Expression of Aryl Hydrocarbon Receptor in Keratinocytes Causes Inflammatory Skin Lesions , 2005, Molecular and Cellular Biology.
[45] Wenge Lu,et al. The effects and comparative differences of neutrophil specific chemokines on neutrophil chemotaxis of the neonate. , 2005, Cytokine.
[46] P. Schmid‐Grendelmeier,et al. Chemokines and their receptors in the pathogenesis of allergic asthma: progress and perspective , 2005, Current opinion in pulmonary medicine.
[47] M. Schmitz,et al. Constitutive and interleukin-1-inducible phosphorylation of p65 NF-{kappa}B at serine 536 is mediated by multiple protein kinases including I{kappa}B kinase (IKK)-{alpha}, IKK{beta}, IKK{epsilon}, TRAF family member-associated (TANK)-binding kinase 1 (TBK1), and an unknown kinase and couples p65 to , 2004, The Journal of biological chemistry.
[48] N. Suttorp,et al. Streptococcus pneumoniae-induced p38 MAPK-dependent Phosphorylation of RelA at the Interleukin-8 Promotor* , 2004, Journal of Biological Chemistry.
[49] S. Ghosh,et al. RelB Forms Transcriptionally Inactive Complexes with RelA/p65* , 2003, Journal of Biological Chemistry.
[50] David E. Misek,et al. RANTES expression is a predictor of survival in stage I lung adenocarcinoma. , 2002, Clinical cancer research : an official journal of the American Association for Cancer Research.
[51] O. Dittrich‐Breiholz,et al. Multiple control of interleukin‐8 gene expression , 2002, Journal of leukocyte biology.
[52] M. Gallo,et al. Ah receptor and NF-κB interactions: mechanisms and physiological implications , 2002 .
[53] Govinda Rao,et al. IRF3 mediates a TLR3/TLR4-specific antiviral gene program. , 2002, Immunity.
[54] Jong Soon Kang,et al. Aryl hydrocarbon receptor-dependent inhibition of AP-1 activity by 2,3,7,8-tetrachlorodibenzo-p-dioxin in activated B cells. , 2002, Toxicology and applied pharmacology.
[55] M. Gallo,et al. Ah receptor and NF-kappaB interactions: mechanisms and physiological implications. , 2002, Chemico-biological interactions.
[56] K. Xie,et al. Interleukin-8 and human cancer biology. , 2001, Cytokine & growth factor reviews.
[57] G. Bren,et al. Transcription of the RelB gene is regulated by NF-kappaB. , 2001, Oncogene.
[58] D. W. Kim,et al. The RelA NF-κB subunit and the aryl hydrocarbon receptor (AhR) cooperate to transactivate the c-myc promoter in mammary cells , 2000, Oncogene.
[59] A. Yuan,et al. Interleukin-8 messenger ribonucleic acid expression correlates with tumor progression, tumor angiogenesis, patient survival, and timing of relapse in non-small-cell lung cancer. , 2000, American journal of respiratory and critical care medicine.
[60] H. Sakurai,et al. IκB Kinases Phosphorylate NF-κB p65 Subunit on Serine 536 in the Transactivation Domain* , 1999, The Journal of Biological Chemistry.
[61] M. Halterman,et al. Eradication of pre-established lymphoma using herpes simplex virus amplicon vectors. , 1999, Blood.
[62] M. Gallo,et al. Ah Receptor and NF-κB Interactions, a Potential Mechanism for Dioxin Toxicity* , 1999, The Journal of Biological Chemistry.
[63] H. Sakurai,et al. IkappaB kinases phosphorylate NF-kappaB p65 subunit on serine 536 in the transactivation domain. , 1999, The Journal of biological chemistry.
[64] M. Gallo,et al. Ah receptor and NF-kappaB interactions, a potential mechanism for dioxin toxicity. , 1999, The Journal of biological chemistry.
[65] W. Murphy,et al. Prospects for cytokine and chemokine biotherapy. , 1997, Clinical cancer research : an official journal of the American Association for Cancer Research.
[66] J. Gustafsson,et al. Aryl hydrocarbon receptor-mediated signal transduction. , 1997, Critical reviews in toxicology.
[67] D. Goeddel,et al. RANTES secretion by gene-modified tumor cells results in loss of tumorigenicity in vivo: role of immune cell subpopulations. , 1996, Human gene therapy.
[68] O. Gotoh,et al. Interindividual difference in expression of human Ah receptor and related P450 genes. , 1994, Carcinogenesis.