IL-8 enhancement of amyloid-beta (Aβ1-42)-induced expression and production of pro-inflammatory cytokines and COX-2 in cultured human microglia
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Hyun B Choi | Seung U. Kim | S. Franciosi | Hyun B. Choi | James G. McLarnon | Sonia Franciosi | J. Mclarnon | J. McLarnon
[1] D W Dickson,et al. Cytokine production by human fetal microglia and astrocytes. Differential induction by lipopolysaccharide and IL-1 beta. , 1993, Journal of immunology.
[2] R. Veerhuis,et al. The role of cyclo-oxygenase 1 and 2 activity in prostaglandin E2 secretion by cultured human adult microglia: Implications for Alzheimer’s disease , 2002, Brain Research.
[3] Seung U. Kim,et al. Differential activation of subtype purinergic receptors modulates Ca2+ mobilization and COX‐2 in human microglia , 2003, Glia.
[4] L. Haverkamp,et al. Senile plaques stimulate microglia to release a neurotoxin found in Alzheimer brain , 1995, Neurochemistry International.
[5] B. Hyman,et al. Microglial response to amyloid plaques in APPsw transgenic mice. , 1998, The American journal of pathology.
[6] L. Lue,et al. Gene expression profiling of amyloid beta peptide-stimulated human post-mortem brain microglia , 2001, Neurobiology of Aging.
[7] M. V. van Breemen,et al. Amyloid β plaque-associated proteins C1q and SAP enhance the Aβ1–42 peptide-induced cytokine secretion by adult human microglia in vitro , 2003, Acta Neuropathologica.
[8] B. Hyman,et al. Interleukin-8 receptor B immunoreactivity in brain and neuritic plaques of Alzheimer's disease. , 1997, The American journal of pathology.
[9] E. Scarpini,et al. Proinflammatory profile of cytokine production by human monocytes and murine microglia stimulated with β-amyloid[25–35] , 1999, Journal of Neuroimmunology.
[10] D. Schubert,et al. The amyloid beta-protein of Alzheimer's disease is chemotactic for mononuclear phagocytes. , 1992, Biochemical and biophysical research communications.
[11] Seung-Up Kim,et al. Generation and Characterization of Immortalized Human Microglial Cell Lines: Expression of Cytokines and Chemokines , 2001, Neurobiology of Disease.
[12] G. Murphy,et al. Macrophage Colony-stimulating Factor Augments β-Amyloid-induced Interleukin-1, Interleukin-6, and Nitric Oxide Production by Microglial Cells* , 1998, The Journal of Biological Chemistry.
[13] M. Woodroofe,et al. Chemokines induce migration and changes in actin polymerization in adult rat brain microglia and a human fetal microglial cell line in vitro , 1999, Journal of neuroscience research.
[14] M. Giovannini,et al. β-Amyloid-Induced Inflammation and Cholinergic Hypofunction in the Rat Brain in Vivo: Involvement of the p38MAPK Pathway , 2002, Neurobiology of Disease.
[15] J. Satoh,et al. T-cell costimulatory molecules B7-1 (CD80) and B7-2 (CD86) are expressed in human microglia but not in astrocytes in culture , 1995, Brain Research.
[16] H. Hampel,et al. Inflammatory repertoire of Alzheimer's disease and nondemented elderly microglia in vitro , 2001, Glia.
[17] P. Mcgeer,et al. The inflammatory response system of brain: implications for therapy of Alzheimer and other neurodegenerative diseases , 1995, Brain Research Reviews.
[18] Shuxian Hu,et al. Cytokine regulation of human microglial cell IL-8 production. , 1998, Journal of immunology.
[19] A. Hofman,et al. Nonsteroidal antiinflammatory drugs and the risk of Alzheimer's disease. , 2001, The New England journal of medicine.
[20] E. Benveniste. Inflammatory cytokines within the central nervous system: sources, function, and mechanism of action. , 1992, The American journal of physiology.
[21] A. Nagai,et al. Cytokines, chemokines, and cytokine receptors in human microglia , 2002, Journal of neuroscience research.
[22] D. Giulian,et al. Brain glia release factors with opposing actions upon neuronal survival , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[23] W. Griffin,et al. Glial‐Neuronal Interactions in Alzheimer's Disease: The Potential Role of a ‘Cytokine Cycle’ in Disease Progression , 1998, Brain pathology.
[24] K. Matsushima,et al. IL‐8 induces T cell Chemotaxis, suppresses IL‐4, and up‐regulates IL‐8 production by CD4+ T cells , 1996, Journal of leukocyte biology.
[25] J. Ryu,et al. Inhibition of lipopolysaccharide‐induced cyclooxygenase‐2, tumor necrosis factor‐α and [Ca2+]i responses in human microglia by the peripheral benzodiazepine receptor ligand PK11195 , 2002, Journal of neurochemistry.
[26] R. Mrak,et al. Interleukin‐1 in the genesis and progression of and risk for development of neuronal degeneration in Alzheimer’s disease , 2002, Journal of leukocyte biology.
[27] A. Kaszniak,et al. Clinical trial of indomethacin in Alzheimer's disease , 1993, Neurology.
[28] P. Scheltens,et al. Chemokines in serum and cerebrospinal fluid of Alzheimer's disease patients , 2003, Annals of neurology.
[29] D. Walker,et al. Complement and cytokine gene expression in cultured microglia derived from postmortem human brains , 1995, Journal of neuroscience research.
[30] Douglas R. McDonald,et al. Amyloid Fibrils Activate Tyrosine Kinase-Dependent Signaling and Superoxide Production in Microglia , 1997, The Journal of Neuroscience.
[31] K. Matsushima,et al. Essential involvement of interleukin‐8 (IL‐8) in acute inflammation , 1994, Journal of leukocyte biology.
[32] P. Aisen,et al. Regional distribution of cyclooxygenase‐2 in the hippocampal formation in Alzheimer's disease , 1999, Journal of neuroscience research.
[33] A. Ford-hutchinson,et al. Expression of mRNA for cyclooxygenase‐1 and cyclooxygenase‐2 in human tissues , 1993, FEBS letters.
[34] D. Ferrari,et al. Activation of microglial cells by β-amyloid protein and interferon-γ , 1995, Nature.
[35] C. Plata-salamán,et al. Inflammation and Alzheimer’s disease , 2000, Neurobiology of Aging.
[36] A. Mantovani,et al. IL‐8 induces a specific transcriptional profile in human neutrophils: synergism with LPS for IL‐1 production , 2004, European journal of immunology.
[37] P. Aisen,et al. Cyclooxygenase-2 expression is increased in frontal cortex of Alzheimer's disease brain , 1998, Neuroscience.
[38] D. Feinstein,et al. Noradrenergic depletion increases inflammatory responses in brain: effects on IκB and HSP70 expression , 2003, Journal of neurochemistry.
[39] J. Johnston,et al. Serum amyloid A induces calcium mobilization and chemotaxis of human monocytes by activating a pertussis toxin-sensitive signaling pathway. , 1995, Journal of immunology.
[40] E. Ling,et al. The origin and nature of ramified and amoeboid microglia: A historical review and current concepts , 1993, Glia.
[41] R. Veerhuis,et al. Cyclooxygenase expression in microglia and neurons in Alzheimer's disease and control brain , 2000, Acta Neuropathologica.
[42] P. Mcgeer,et al. Human neurons generate C-reactive protein and amyloid P: upregulation in Alzheimer’s disease , 2000, Brain Research.