MIP-3 (cid:1) neutralizing monoclonal antibody protects against TNBS-induced colonic injury and inflammation in mice
暂无分享,去创建一个
[1] S. Brand,et al. Cell differentiation dependent expressed CCR6 mediates ERK‐1/2, SAPK/JNK, and Akt signaling resulting in proliferation and migration of colorectal cancer cells , 2006, Journal of cellular biochemistry.
[2] S. Jalkanen,et al. Lymphocyte homing to the gut: attraction, adhesion, and commitment , 2005, Immunological reviews.
[3] P. Allavena,et al. Intestinal immune homeostasis is regulated by the crosstalk between epithelial cells and dendritic cells , 2005, Nature Immunology.
[4] N. Hosoe,et al. Increased lymphocyte trafficking to colonic microvessels is dependent on MAdCAM‐1 and C‐C chemokine mLARC/CCL20 in DSS‐induced mice colitis , 2005, Clinical and experimental immunology.
[5] M. Kagnoff,et al. Chemokine receptor CCR6 transduces signals that activate p130Cas and alter cAMP-stimulated ion transport in human intestinal epithelial cells. , 2005, American journal of physiology. Cell physiology.
[6] R. Newberry,et al. Inhibition of indoleamine 2,3-dioxygenase augments trinitrobenzene sulfonic acid colitis in mice. , 2003, Gastroenterology.
[7] J. Flores,et al. CCR6 has a non‐redundant role in the development of inflammatory bowel disease , 2003, European journal of immunology.
[8] J. Van Damme,et al. The CC chemokine CCL20 and its receptor CCR6. , 2003, Cytokine & growth factor reviews.
[9] I. Williams,et al. CCR6 expression distinguishes mouse myeloid and lymphoid dendritic cell subsets: demonstration using a CCR6 EGFP knock‐in mouse , 2002, European journal of immunology.
[10] L. Lefrançois,et al. Isolation of mouse small intestinal intraepithelial lymphocytes, Peyer's patch, and lamina propria cells. , 2001, Current protocols in immunology.
[11] M. Leach,et al. Characterization of chemokines and chemokine receptors in two murine models of inflammatory bowel disease: IL‐10– / – mice and Rag‐2– / – mice reconstituted with CD4+CD45RBhigh T cells , 2001, European journal of immunology.
[12] P. Ricciardi-Castagnoli,et al. Dendritic cells express tight junction proteins and penetrate gut epithelial monolayers to sample bacteria , 2001, Nature Immunology.
[13] M. Kagnoff,et al. Regulated MIP-3alpha/CCL20 production by human intestinal epithelium: mechanism for modulating mucosal immunity. , 2001, American journal of physiology. Gastrointestinal and liver physiology.
[14] C. Martínez-A,et al. CCR6-deficient mice have impaired leukocyte homeostasis and altered contact hypersensitivity and delayed-type hypersensitivity responses. , 2001, The Journal of clinical investigation.
[15] S. Caughman,et al. Cutting Edge: C-C Chemokine Receptor 6 Is Essential for Arrest of a Subset of Memory T Cells on Activated Dermal Microvascular Endothelial Cells Under Physiologic Flow Conditions In Vitro , 2000, The Journal of Immunology.
[16] W. Gong,et al. Expression of CCR6 and CD83 by cytokine-activated human neutrophils. , 2000, Blood.
[17] C. Kelly,et al. Interleukin 16 is up-regulated in Crohn's disease and participates in TNBS colitis in mice. , 2000, Gastroenterology.
[18] C. Caux,et al. Macrophage Inflammatory Protein 3α Is Expressed at Inflamed Epithelial Surfaces and Is the Most Potent Chemokine Known in Attracting Langerhans Cell Precursors , 2000, The Journal of experimental medicine.
[19] J. Mcghee,et al. Mice deficient in Th1- and Th2-type cytokines develop distinct forms of hapten-induced colitis. , 2000, Gastroenterology.
[20] H. Greenberg,et al. CCR6 mediates dendritic cell localization, lymphocyte homeostasis, and immune responses in mucosal tissue. , 2000, Immunity.
[21] Y. Tanaka,et al. Selective expression of liver and activation‐regulated chemokine (LARC) in intestinal epithelium in mice and humans , 1999, European journal of immunology.
[22] C. Caux,et al. Selective Recruitment of Immature and Mature Dendritic Cells by Distinct Chemokines Expressed in Different Anatomic Sites , 1998, The Journal of experimental medicine.
[23] E. Butcher,et al. Chemokines and the arrest of lymphocytes rolling under flow conditions. , 1998, Science.
[24] A. Mantovani,et al. Cloning and Characterization of a Specific Receptor for the Novel CC Chemokine MIP-3α from Lung Dendritic Cells , 1997, The Journal of experimental medicine.
[25] J. Gutiérrez-Ramos,et al. Neurotactin, a membrane-anchored chemokine upregulated in brain inflammation , 1997, Nature.
[26] Wei Wang,et al. A new class of membrane-bound chemokine with a CX3C motif , 1997, Nature.
[27] J. Mcghee,et al. Hapten-induced model of murine inflammatory bowel disease: mucosa immune responses and protection by tolerance. , 1996, Journal of immunology.
[28] A. Ben-Baruch,et al. Signals and Receptors Involved in Recruitment of Inflammatory Cells (*) , 1995, The Journal of Biological Chemistry.
[29] D. Largaespada,et al. Lymphotactin: a cytokine that represents a new class of chemokine. , 1994, Science.
[30] L. Mayer,et al. Stimulation of lamina propria lymphocytes by intestinal epithelial cells: evidence for recognition of nonclassical restriction elements , 1994, The Journal of experimental medicine.
[31] H. Tilg,et al. Increased Expression of CCL20 in Human Inflammatory Bowel Disease , 2004, Journal of Clinical Immunology.
[32] T. Matsui,et al. Production of macrophage inflammatory protein 3alpha (MIP-3alpha) (CCL20) and MIP-3beta (CCL19) by human peripheral blood neutrophils in response to microbial pathogens. , 2003, Infection and immunity.
[33] R. Colman,et al. Experimental models of inflammatory bowel disease. , 2003, Archivum immunologiae et therapiae experimentalis.
[34] L. Mayer,et al. Colonic epithelial cells are a major site of macrophage inflammatory protein 3alpha (MIP-3alpha) production in normal colon and inflammatory bowel disease. , 2002, Gut.
[35] R. Blumberg,et al. The immunology of mucosal models of inflammation. , 2002, Annual review of immunology.
[36] P. Allavena,et al. Neutrophils produce biologically active macrophage inflammatory protein-3alpha (MIP-3alpha)/CCL20 and MIP-3beta/CCL19. , 2001, European journal of immunology.
[37] R. Rabin,et al. CC-chemokine receptor 6 is expressed on diverse memory subsets of T cells and determines responsiveness to macrophage inflammatory protein 3 alpha. , 1999, Journal of immunology.