Involvement of integrins, MAPK, and NF-kappaB in regulation of the shear stress-induced MMP-9 expression in endothelial cells.
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Mei Zhang | Yun Zhang | Mei Zhang | Hui-Wen Sun | Chang-Jiang Li | Huai-Qing Chen | Hui-Li Lin | Hui-Xia Lv | Yun Zhang | H. Lv | Huaiqing Chen | Chang-jiang Li | Hui-wen Sun | Hui Lin
[1] Zhe Sun,et al. Integrins and Regulation of the Microcirculation: From Arterioles to Molecular Studies using Atomic Force Microscopy , 2005, Microcirculation.
[2] S. Im,et al. Involvement of matrix metalloproteinase‐9 in platelet‐activating factor‐induced angiogenesis , 2005, FEBS letters.
[3] F. Grosveld,et al. Atherosclerotic Lesion Size and Vulnerability Are Determined by Patterns of Fluid Shear Stress , 2006, Circulation.
[4] N. Parinandi,et al. Redox Regulation of 4-Hydroxy-2-nonenal-mediated Endothelial Barrier Dysfunction by Focal Adhesion, Adherens, and Tight Junction Proteins* , 2006, Journal of Biological Chemistry.
[5] B. Sumpio,et al. Shear stress-stimulated endothelial cells induce smooth muscle cell chemotaxis via platelet-derived growth factor-BB and interleukin-1alpha. , 2005, Journal of vascular surgery.
[6] Z. Galis,et al. Expression of Matrix Metalloproteinase-9 in Endothelial Cells Is Differentially Regulated by Shear Stress , 2003, Journal of Biological Chemistry.
[7] John M Tarbell,et al. Asynchronous Shear Stress and Circumferential Strain Reduces Endothelial NO Synthase and Cyclooxygenase-2 but Induces Endothelin-1 Gene Expression in Endothelial Cells , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[8] Andrés J. García,et al. Model of integrin-mediated cell adhesion strengthening. , 2007, Journal of biomechanics.
[9] C Kleinstreuer,et al. Hemodynamics simulation and identification of susceptible sites of atherosclerotic lesion formation in a model abdominal aorta. , 2003, Journal of biomechanics.
[10] T. Hunter,et al. Fluid Shear Stress Activation of Focal Adhesion Kinase , 1997, The Journal of Biological Chemistry.
[11] G. Garcı́a-Cardeña,et al. Endothelial Dysfunction, Hemodynamic Forces, and Atherogenesis a , 2000, Annals of the New York Academy of Sciences.
[12] P. Carmeliet,et al. Function of the plasminogen/plasmin and matrix metalloproteinase systems after vascular injury in mice with targeted inactivation of fibrinolytic system genes. , 1998, Arteriosclerosis, thrombosis, and vascular biology.
[13] Nikos Stergiopulos,et al. Arterial Wall Response to ex vivo Exposure to Oscillatory Shear Stress , 2005, Journal of Vascular Research.
[14] V. Fuster,et al. Human monocyte-derived macrophages induce collagen breakdown in fibrous caps of atherosclerotic plaques. Potential role of matrix-degrading metalloproteinases and implications for plaque rupture. , 1995, Circulation.
[15] S. Alper,et al. Hemodynamic shear stress and its role in atherosclerosis. , 1999, JAMA.
[16] B. Sumpio,et al. Role of PP2A in the regulation of p38 MAPK activation in bovine aortic endothelial cells exposed to cyclic strain , 2003, Journal of cellular physiology.
[17] Martin Chalfie,et al. Genetics of sensory mechanotransduction. , 2002, Annual review of genetics.
[18] D. Ku,et al. Transmural pressure induces matrix-degrading activity in porcine arteries ex vivo. , 1999, American journal of physiology. Heart and circulatory physiology.
[19] V. Gahtan,et al. Localization of atherosclerosis: role of hemodynamics. , 1999, Archives of surgery.
[20] C. Zarins,et al. Arterial enlargement in response to high flow requires early expression of matrix metalloproteinases to degrade extracellular matrix. , 2002, Experimental and molecular pathology.
[21] Q. Xu,et al. Biomechanical-stress-induced signaling and gene expression in the development of arteriosclerosis. , 2000, Trends in cardiovascular medicine.
[22] J. H. Wang,et al. An Introductory Review of Cell Mechanobiology , 2006, Biomechanics and modeling in mechanobiology.
[23] A. Morgan,et al. Haplotypic analysis of the MMP-9 gene in relation to coronary artery disease , 2003, Journal of Molecular Medicine.
[24] Shu Chien,et al. Role of integrins in endothelial mechanosensing of shear stress. , 2002, Circulation research.
[25] A. J. Valente,et al. Interleukin-18-induced Human Coronary Artery Smooth Muscle Cell Migration Is Dependent on NF-κB- and AP-1-mediated Matrix Metalloproteinase-9 Expression and Is Inhibited by Atorvastatin* , 2006, Journal of Biological Chemistry.
[26] S. Kawashima,et al. Shear stress enhances glutathione peroxidase expression in endothelial cells. , 2000, Biochemical and biophysical research communications.
[27] Jiahuai Han,et al. PKCε modulates NF-κB and AP-1 via mitogen-activated protein kinases in adult rabbit cardiomyocytes , 2000, American Journal of Physiology-Heart and Circulatory Physiology.
[28] P. Libby,et al. Increased expression of matrix metalloproteinases and matrix degrading activity in vulnerable regions of human atherosclerotic plaques. , 1994, The Journal of clinical investigation.
[29] M. U. Naik,et al. Junctional adhesion molecule-A-induced endothelial cell migration on vitronectin is integrin αvβ3 specific , 2006, Journal of Cell Science.
[30] S. Verma,et al. Biomarkers of Vascular Disease Linking Inflammation to Endothelial Activation: Part II , 2003, Circulation.
[31] S. Lehoux,et al. Bases cellulaires de la mécanotransduction dans la cellule endothéliale , 2004 .