β-amyloid deposition and other measures of neuropathology predict cognitive status in Alzheimer's disease
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Brian J Cummings | C. Cotman | R. Shankle | C. Pike | Christian J. Pike | Carl W. Cotman | Brian J. Cummings | Rodman Shankle
[1] I. Kanazawa,et al. Genetic association of the very low density lipoprotein (VLDL) receptor gene with sporadic Alzheimer's disease , 1995, Nature Genetics.
[2] Bradley T. Hyman,et al. Neurofibrillary tangles but not senile plaques parallel duration and severity of Alzheimer's disease , 1992, Neurology.
[3] Anthony F Jorm,et al. The Informant Questionnaire on Cognitive Decline in the Elderly (IQCODE): socio-demographic correlates, reliability, validity and some norms , 1989, Psychological Medicine.
[4] D. Schubert,et al. Amyloid beta-protein activates tachykinin receptors and inositol trisphosphate accumulation by synergy with glutamate. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[5] G. Lynch,et al. Induction of β-Amyloid-Containing Polypeptides in Hippocampus: Evidence for a Concomitant Loss of Synaptic Proteins and Interactions with an Excitotoxin , 1994, Experimental Neurology.
[6] J. Morris. The Clinical Dementia Rating (CDR) , 1993, Neurology.
[7] C. Duyckaerts,et al. Large amounts of neocortical βA4 deposits without neuritic plaques nor tangles in a psychometrically assessed, non-demented person , 1990, Neuroscience Letters.
[8] Paul J. Harrison,et al. Synaptophysin gene expression in human brain: A quantitative in situ hybridization and immunocytochemical study , 1994, Neuroscience.
[9] S. Younkin. The amyloid beta protein precursor mutations linked to familial Alzheimer's disease alter processing in a way that fosters amyloid deposition. , 1994, The Tohoku journal of experimental medicine.
[10] Carl W. Cotman,et al. Color image analysis in neuroanatomical research: Application to senile plaque subtype quantification in Alzheimer's disease , 1995, Neurobiology of Aging.
[11] E. Masliah,et al. Diffuse plaques do not accentuate synapse loss in Alzheimer's disease. , 1990, The American journal of pathology.
[12] C. Cotman,et al. β-Amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage , 1990, Brain Research.
[13] A. Klug,et al. Topographical relationship between beta-amyloid and tau protein epitopes in tangle-bearing cells in Alzheimer disease. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[14] K. Beyreuther,et al. Quantitative assessment of the synaptophysin immuno-reactivity of the cortical neuropil in various neurodegenerative disorders with dementia. , 1993, Dementia.
[15] W. Schlote,et al. Rating of the lesions in senile dementia of the Alzheimer type: concordance between laboratories A European multicenter study under the auspices of EURAGE , 1990, Journal of the Neurological Sciences.
[16] Brian J Cummings,et al. β-Amyloid-induced changes in cultured astrocytes parallel reactive astrocytosis associated with senile plaques in Alzheimer's disease , 1994, Neuroscience.
[17] C. Bieberich,et al. The Alzheimer's Aβ peptide induces neurodegeneration and apoptotic cell death in transgenic mice , 1995, Nature Genetics.
[18] Peter Davies,et al. Identification of normal and pathological aging in prospectively studied nondemented elderly humans , 1992, Neurobiology of Aging.
[19] J. Trojanowski,et al. Alzheimer disease A68 proteins injected into rat brain induce codeposits of beta-amyloid, ubiquitin, and alpha 1-antichymotrypsin. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[20] A. Smith,et al. Relative roles of plaques and tangles in the dementia of Alzheimer's disease: correlations using three sets of neuropathological criteria. , 1995, Dementia.
[21] K. Kosik,et al. Neuritic pathology and dementia in alzheimer's disease , 1991, Annals of neurology.
[22] J. Troncoso,et al. Differences in the pattern of hippocampal neuronal loss in normal ageing and Alzheimer's disease , 1994, The Lancet.
[23] C. Duyckaerts,et al. Does amyloid precede paired helical filaments in the senile plaque? A study of 15 cases with graded intellectual status in aging and Alzheimer disease , 1988, Neuroscience Letters.
[24] Brian J Cummings,et al. β-Amyloid Accumulation Correlates with Cognitive Dysfunction in the Aged Canine , 1996, Neurobiology of Learning and Memory.
[25] L. Thal,et al. Alzheimer's disease , 1986, Neurology.
[26] M. Roth,et al. The Association Between Quantitative Measures of Dementia and of Senile Change in the Cerebral Grey Matter of Elderly Subjects , 1968, British Journal of Psychiatry.
[27] N. Milgram,et al. Cognitive functions and aging in the dog: acquisition of nonspatial visual tasks. , 1994, Behavioral neuroscience.
[28] S. Prusiner,et al. Formic acid pretreatment enhances immunostaining of cerebral and systemic amyloids. , 1987, Laboratory investigation; a journal of technical methods and pathology.
[29] R. Mantegna,et al. Quantitative analysis of senile plaques in Alzheimer disease: observation of log-normal size distribution and molecular epidemiology of differences associated with apolipoprotein E genotype and trisomy 21 (Down syndrome). , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[30] Brian J Cummings,et al. Constructional apraxia in Alzheimer's disease correlates with neuritic neuropathology in occipital cortex , 1996, Brain Research.
[31] D. Sparks,et al. Quantitative assessment of synaptic density in the outer molecular layer of the hippocampal dentate gyrus in Alzheimer's disease. , 1996, Dementia.
[32] S. DeKosky,et al. Synapse loss in frontal cortex biopsies in Alzheimer's disease: Correlation with cognitive severity , 1990, Annals of neurology.
[33] S. Rapoport,et al. β-amyloid increases choline conductance of PC12 cells: possible mechanism of toxicity in Alzheimer's disease , 1994, Brain Research.
[34] R. McConnell,et al. Microwave antigen retrieval for immunocytochemistry on formalin‐fixed, paraffin‐embedded post‐mortem CNS tissue , 1995, The Journal of pathology.
[35] P. Davies,et al. Hydrofluoric acid-treated tau PHF proteins display the same biochemical properties as normal tau. , 1992, The Journal of biological chemistry.
[36] P. Davies,et al. A preparation of Alzheimer paired helical filaments that displays distinct tau proteins by polyacrylamide gel electrophoresis. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[37] V. Hachinski,et al. A NEW DEFINITION OF ALZHEIMER'S DISEASE: A HIPPOCAMPAL DEMENTIA , 1985, The Lancet.
[38] C. Glabe,et al. Intracellular Aβ1-42 Aggregates Stimulate the Accumulation of Stable, Insoluble Amyloidogenic Fragments of the Amyloid Precursor Protein in Transfected Cells (*) , 1995, The Journal of Biological Chemistry.
[39] Brian J Cummings,et al. beta-Amyloid induces neuritic dystrophy in vitro: similarities with Alzheimer pathology. , 1992, Neuroreport.
[40] C. Duyckaerts,et al. COMPARISON OF SEVEN STAINING METHODS FOR SENILE PLAQUES AND NEUROFIBRILLARY TANGLES IN A PROSPECTIVE SERIES OF 15 ELDERLY PATIENTS , 1989, Neuropathology and applied neurobiology.
[41] L. Lapham,et al. Clinicopathologic Correlates in Alzheimer Disease: Assessment of Clinical and Pathologic Diagnostic Criteria , 1993, Alzheimer disease and associated disorders.
[42] L. Iversen,et al. The toxicity in vitro of beta-amyloid protein. , 1995, The Biochemical journal.
[43] D. Selkoe,et al. Mutation of the β-amyloid precursor protein in familial Alzheimer's disease increases β-protein production , 1992, Nature.
[44] S. M. Sumi,et al. The Consortium to Establish a Registry for Alzheimer's Disease (CERAD) , 1991, Neurology.
[45] L. Mucke,et al. Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein , 1995, Nature.
[46] J. C. Chisholm,et al. The Ca2+ influx induced by beta-amyloid peptide 25-35 in cultured hippocampal neurons results from network excitation. , 1995, Journal of neurobiology.
[47] Brian J Cummings,et al. Aggregation of the amyloid precursor protein within degenerating neurons and dystrophic neurites in alzheimer's disease , 1992, Neuroscience.
[48] S. DeKosky,et al. APOE*4-associated Alzheimer's disease risk is modified by alpha 1-antichymotrypsin polymorphism. , 1995, Nature genetics.
[49] J. Miller,et al. Neuropathological indexes of Alzheimer's disease in demented and nondemented persons aged 80 years and older. , 1993, Archives of neurology.
[50] R. Mohs,et al. Consortium to establish a registry for Alzheimer's disease (CERAD) clinical and neuropsychological assessment of Alzheimer's disease. , 2002, Psychopharmacology bulletin.
[51] K. Jellinger,et al. Synaptic Pathology of Alzheimer's Disease a , 1993, Annals of the New York Academy of Sciences.
[52] Brian J Cummings,et al. The canine as an animal model of human aging and dementia , 1996, Neurobiology of Aging.
[53] E. Masliah,et al. A simple dot-immunobinding assay for quantification of synaptophysin-like immunoreactivity in human brain. , 1994, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[54] D. Sparks,et al. Quantitative assessment of synaptic density in the entorhinal cortex in Alzheimer's disease , 1993, Annals of neurology.
[55] D. Dickson,et al. Primitive plaques and memory dysfunction in normal and impaired elderly persons. , 1987, The New England journal of medicine.
[56] J. Growdon,et al. Clinical and Neuropathological Correlates of Apolipoprotein E Genotype in Alzheimer's Disease ‐ Window on Molecular Epidemiology a , 1996, Annals of the New York Academy of Sciences.
[57] J. Rostas,et al. A rapid method for isolation of synaptosomes on Percoll gradients , 1986, Brain Research.
[58] G. K. Wilcock,et al. Plaques, tangles and dementia A quantitative study , 1982, Journal of the Neurological Sciences.
[59] D. Salmon,et al. Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment , 1991, Annals of neurology.
[60] M. Ilyas Kamboh,et al. A4POE*4-associated Alzheimer's disease risk is modified by α1–antichymotrypsin polymorphism , 1995, Nature Genetics.
[61] S. Folstein,et al. "Mini-mental state". A practical method for grading the cognitive state of patients for the clinician. , 1975, Journal of psychiatric research.
[62] B. Hyman,et al. The Lack of Accumulation of Senile Plaques or Amyloid Burden in Alzheimer's Disease Suggests a Dynamic Balance Between Amyloid Deposition and Resolution , 1993, Journal of neuropathology and experimental neurology.
[63] K. Wisniewski,et al. Ageing in higher functioning adults with Down's syndrome: an interim report in a longitudinal study. , 2008, Journal of intellectual disability research : JIDR.
[64] Paul D. Coleman,et al. Neuron numbers and dendritic extent in normal aging and Alzheimer's disease , 1987, Neurobiology of Aging.
[65] N. Akaike,et al. Amyloid β protein‐induced irreversible current in rat cortical neurones , 1994 .
[66] W. Müller,et al. beta-Amyloid peptide decreases membrane fluidity. , 1995, Brain research.
[67] Jacob Cohen,et al. Applied multiple regression/correlation analysis for the behavioral sciences , 1979 .
[68] R. Katzman.,et al. Clinical, pathological, and neurochemical changes in dementia: A subgroup with preserved mental status and numerous neocortical plaques , 1988, Annals of neurology.
[69] Brian J Cummings,et al. Spatial learning and memory as a function of age in the dog. , 1995, Behavioral neuroscience.
[70] J. Hauw,et al. Differential correlation between neurochemical deficits, neuropathology, and cognitive status in Alzheimer's disease , 1995, Neurobiology of Aging.
[71] D. Salmon,et al. Longitudinal evaluation of dementia of the Alzheimer type: a comparison of 3 standardized mental status examinations , 1991 .
[72] Brian J Cummings,et al. Beta-amyloid-induced changes in cultured astrocytes parallel reactive astrocytosis associated with senile plaques in Alzheimer's disease. , 1994, Neuroscience.
[73] L Carlin,et al. Neocortical neurofibrillary tangles correlate with dementia severity in Alzheimer's disease. , 1995, Archives of neurology.
[74] H. Soininen,et al. Loss of synaptophysin-like immunoreactivity in the hippocampal formation is an early phenomenon in alzheimer's disease , 1995, Neuroscience.
[75] W. Müller,et al. β-amyloid peptide decreases membrane fluidity , 1995, Brain Research.
[76] W. Honer,et al. Correlations of synaptic and pathological markers with cognition of the elderly , 1995, Neurobiology of Aging.
[77] C. Cotman,et al. Apoptosis is induced by beta-amyloid in cultured central nervous system neurons. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[78] Brian J Cummings,et al. Subpopulations of dystrophic neurites [correction of neuritis] in Alzheimer's brain with distinct immunocytochemical and argentophilic characteristics. , 1994, Brain research.
[79] B. Crain,et al. Interlaboratory Comparison of Neuropathology Assessments in Alzheimer's Disease: A Study of the Consortium to Establish a Registry for Alzheimer's Disease (CERAD) , 1994, Journal of neuropathology and experimental neurology.
[80] H. Wiśniewski,et al. Alzheimer’s Disease, a Cerebral Form of Amyloidosis , 1988 .
[81] B. Yankner,et al. β-Amyloid fibrils induce tau phosphorylation and loss of microtubule binding , 1995, Neuron.
[82] D. Selkoe,et al. Mutation of the beta-amyloid precursor protein in familial Alzheimer's disease increases beta-protein production. , 1992, Nature.
[83] Brian J Cummings,et al. Image analysis of β-amyloid load in Alzheimer's disease and relation to dementia severity , 1995, The Lancet.
[84] D. Troost,et al. Tissue fixation methods alter the immunohistochemical demonstrability of neurofilament proteins, synaptophysin, and glial fibrillary acidic protein in human cerebellum. , 1993, Acta histochemica.
[85] Brian J Cummings,et al. β-Amyloid accumulation in aged canine brain: A model of early plaque formation in Alzheimer's disease , 1993, Neurobiology of Aging.
[86] C. Cotman,et al. Senile plaques as aberrant sprout-stimulating structures , 1986, Experimental Neurology.
[87] J. Wegiel,et al. Review: David Oppenheimer Memorial Lecture 1995: Some neuropathological aspects of Alzheimer's disease and its relevance to other disciplines * , 1996, Neuropathology and applied neurobiology.
[88] H. Wiśniewski,et al. AGE-ASSOCIATED DEVELOPMENT OF DIFFUSE AND THIOFLAVINE-S-POSITIVE PLAQUES IN DOWN SYNDROME , 1994 .
[89] C. Duyckaerts,et al. Alzheimer's lesions labelled by anti‐ubiquitin antibodies: comparison with other staining techniques. A study of 15 cases with graded intellectual status in ageing and Alzheimer's disease , 1993, Neuropathology and applied neurobiology.
[90] N. Akaike,et al. Flow cytometric analysis on cytotoxic action of amyloid β protein fragment 25–35 on brain neurons dissociated from the rats , 1994, Brain Research.
[91] B. Tycko,et al. Synergistic Effects of Traumatic Head Injury and Apolipoprotein-epsilon4 in Patients With Alzheimer's Disease , 1995, Neurology.
[92] P. Yates,et al. THE TOPOGRAPHY OF PLAQUES AND TANGLES IN DOWN'S SYNDROME PATIENTS OF DIFFERENT AGES , 1986, Neuropathology and applied neurobiology.
[93] M. Mattson,et al. Amyloid beta-peptide impairs ion-motive ATPase activities: evidence for a role in loss of neuronal Ca2+ homeostasis and cell death , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.