Endocrine Disruptor Bisphenol A (BPA) Triggers Systemic Para-Inflammation and is Sufficient to Induce Airway Allergic Sensitization in Mice
暂无分享,去创建一个
S. Berdnikovs | Lucas Fedele Loffredo | Mackenzie Elyse Coden | Sergejs Berdnikovs | Mackenzie E. Coden | L. Loffredo | Lucas F Loffredo
[1] D. Medhat,et al. Urinary bisphenol A concentrations in relation to asthma in a sample of Egyptian children , 2018, Human & experimental toxicology.
[2] S. Dey,et al. Canonical Wnt signaling is critical to estrogen-mediated uterine growth. , 2004, Molecular endocrinology.
[3] Nasruddin,et al. 17β‐Estradiol administration promotes delayed cutaneous wound healing in 40‐week ovariectomised female mice , 2016, International wound journal.
[4] E. Raz,et al. Dust mite‐derived Der f 3 activates a pro‐inflammatory program in airway epithelial cells via PAR‐1 and PAR‐2 , 2019, Molecular immunology.
[5] I. C. Arroyo,et al. Non-reproductive effects of sex steroids: their immunoregulatory role. , 2011, Current topics in medicinal chemistry.
[6] H. Takano,et al. Exposure to low-dose bisphenol A during the juvenile period of development disrupts the immune system and aggravates allergic airway inflammation in mice , 2018, International journal of immunopathology and pharmacology.
[7] S. Nakae,et al. IL-33, IL-25 and TSLP contribute to development of fungal-associated protease-induced innate-type airway inflammation , 2018, Scientific Reports.
[8] J. Mei,et al. TSLP induced by estrogen stimulates secretion of MCP-1 and IL-8 and growth of human endometrial stromal cells through JNK and NF-κB signal pathways. , 2014, International journal of clinical and experimental pathology.
[9] J. Rosen,et al. Wnt and mammary stem cells: hormones cannot fly wingless. , 2010, Current opinion in pharmacology.
[10] R. Medzhitov,et al. Stress, inflammation, and defense of homeostasis. , 2014, Molecular cell.
[11] P. Howarth,et al. Defective epithelial barrier function in asthma. , 2011, The Journal of allergy and clinical immunology.
[12] C. Lloyd,et al. CC Chemokine Ligand 1 Promotes Recruitment of Eosinophils But Not Th2 Cells During the Development of Allergic Airways Disease1 , 2003, The Journal of Immunology.
[13] A. Theocharis,et al. Cross-talk between estradiol receptor and EGFR/IGF-IR signaling pathways in estrogen-responsive breast cancers: focus on the role and impact of proteoglycans. , 2014, Matrix biology : journal of the International Society for Matrix Biology.
[14] A. Just,et al. Prenatal and Postnatal Bisphenol A Exposure and Asthma Development Among Inner-City Children , 2013, Pediatrics.
[15] D. Edwards,et al. Receptor mechanisms mediating non-genomic actions of sex steroids. , 2007, Seminars in reproductive medicine.
[16] P. Sly,et al. High IFN-γ production by CD8+ T cells and early sensitization among infants at high risk of atopy , 2004 .
[17] L. Baskin,et al. Endocrine disruptors, genital development, and hypospadias. , 2008, Journal of andrology.
[18] P. Grigsby,et al. Notch-Deficient Skin Induces a Lethal Systemic B-Lymphoproliferative Disorder by Secreting TSLP, a Sentinel for Epidermal Integrity , 2008, PLoS biology.
[19] Yukari Nakajima,et al. Topical estrogen application to wounds promotes delayed cutaneous wound healing in 80-week-old female mice , 2019, PloS one.
[20] M. Fessler,et al. Association of urinary levels of bisphenols F and S used as bisphenol A substitutes with asthma and hay fever outcomes. , 2019, Environmental research.
[21] Luting Xu,et al. Expression of interleukin-4 in the epidermis of transgenic mice results in a pruritic inflammatory skin disease: an experimental animal model to study atopic dermatitis. , 2001, The Journal of investigative dermatology.
[22] R. Ghosh,et al. Untangling the association between environmental endocrine disruptive chemicals and the etiology of male genitourinary cancers. , 2019, Biochemical pharmacology.
[23] U. Kintscher,et al. Metabolic impact of estrogen signalling through ERalpha and ERbeta , 2010, The Journal of Steroid Biochemistry and Molecular Biology.
[24] J. Lobaccaro,et al. Environmental estrogen exposure during fetal life: a time bomb for prostate cancer. , 2014, Endocrinology.
[25] W. Huo,et al. Prenatal exposure to bisphenol A and risk of allergic diseases in early life , 2017, Pediatric Research.
[26] G. Risbridger,et al. Estrogen-regulated development and differentiation of the prostate. , 2008, Differentiation; research in biological diversity.
[27] A. Muñoz,et al. Nuclear receptors: Genomic and non-genomic effects converge , 2009, Cell cycle.
[28] Laura N. Vandenberg,et al. Human exposure to bisphenol A (BPA). , 2007, Reproductive toxicology.
[29] J. Gustafsson,et al. Biological functions and clinical implications of oestrogen receptors alfa and beta in epithelial tissues , 2008, Journal of internal medicine.
[30] T. Shibamoto,et al. Exposure to bisphenol A enhanced lung eosinophilia in adult male mice , 2016, Allergy, Asthma & Clinical Immunology.
[31] S. Woo,et al. Filaggrin knockdown and Toll‐like receptor 3 (TLR3) stimulation enhanced the production of thymic stromal lymphopoietin (TSLP) from epidermal layers , 2011, Experimental dermatology.
[32] K. Gronert,et al. Estrogen negatively regulates epithelial wound healing and protective lipid mediator circuits in the cornea , 2012, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[33] B. Gilchrest,et al. Biology of estrogens in skin: implications for skin aging , 2006, Experimental dermatology.
[34] A. Musti,et al. Crosstalk between Notch, HIF-1α and GPER in Breast Cancer EMT , 2018, International journal of molecular sciences.
[35] J. Morales-Montor,et al. Non-reproductive effects of sex steroids: their immunoregulatory role. , 2011, Current topics in medicinal chemistry.
[36] L. Hummelshoj,et al. Allergic sensitization: host-immune factors , 2014, Clinical and Translational Allergy.
[37] R. Wolf,et al. Abnormal epidermal barrier in the pathogenesis of atopic dermatitis. , 2012, Clinics in dermatology.
[38] Aldehyde dehydrogenase and estrogen receptor define a hierarchy of cellular differentiation in the normal human mammary epithelium , 2014, Breast Cancer Research.
[39] G. Ashcroft,et al. Estrogen, not intrinsic aging, is the major regulator of delayed human wound healing in the elderly , 2008, Genome Biology.
[40] Bruce Blumberg,et al. Endocrine disrupting chemicals and disease susceptibility , 2011, The Journal of Steroid Biochemistry and Molecular Biology.
[41] T. Fukuyama,et al. Oral Administration of Bisphenol A Directly Exacerbates Allergic Airway Inflammation but Not Allergic Skin Inflammation in Mice , 2018, Toxicological sciences : an official journal of the Society of Toxicology.
[42] H. Sampson,et al. Intestinal permeability in children with food allergy on specific elimination diets , 2013, Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology.
[43] H. Takano,et al. Oral exposure to low dose bisphenol A aggravates allergic airway inflammation in mice , 2019, Toxicology reports.
[44] A. Misharin,et al. Phenotypic plasticity and targeting of Siglec‐FhighCD11clow eosinophils to the airway in a murine model of asthma , 2016, Allergy.
[45] C. Zouboulis,et al. Nuclear hormone receptors in human skin. , 2007, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.
[46] F. Minutolo,et al. Risks and benefits related to alimentary exposure to xenoestrogens , 2017, Critical reviews in food science and nutrition.
[47] J. Gustafsson,et al. Estrogen receptors and the metabolic network. , 2011, Cell metabolism.
[48] R. Schleimer,et al. Etiology of epithelial barrier dysfunction in patients with type 2 inflammatory diseases , 2017, The Journal of allergy and clinical immunology.
[49] C. Perrier,et al. Gut permeability and food allergies , 2011, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[50] P. Sly,et al. High IFN-gamma production by CD8+ T cells and early sensitization among infants at high risk of atopy. , 2004, The Journal of allergy and clinical immunology.
[51] Yiping Zhang,et al. Overexpression Of ERβ Participates In The Progression Of Liver Cancer Via Inhibiting The Notch Signaling Pathway , 2019, OncoTargets and therapy.
[52] Frederick S vom Saal,et al. Large effects from small exposures. III. Endocrine mechanisms mediating effects of bisphenol A at levels of human exposure. , 2006, Endocrinology.
[53] J. Rochester. Bisphenol A and human health: a review of the literature. , 2013, Reproductive toxicology.
[54] S. Alshahrani,et al. Diet: A Source of Endocrine Disruptors. , 2020, Endocrine, metabolic & immune disorders drug targets.
[55] T. Takai. Expression : Cellular Sources , Triggers , and Regulatory Mechanisms , 2010 .
[56] A. Shiohama,et al. A homozygous frameshift mutation in the murine filaggrin gene facilitates enhanced percutaneous allergen priming , 2009, Nature Genetics.
[57] T. Simoncini,et al. Extra‐nuclear signaling of estrogen receptors , 2008, IUBMB life.
[58] R. Medzhitov. Origin and physiological roles of inflammation , 2008, Nature.
[59] R. Geha,et al. Desloratadine inhibits allergen-induced airway inflammation and bronchial hyperresponsiveness and alters T-cell responses in murine models of asthma. , 2003, The Journal of allergy and clinical immunology.
[60] T. Midoro-Horiuti,et al. Fetal exposure to bisphenol A as a risk factor for the development of childhood asthma: an animal model study , 2012, Environmental Health.
[61] T. Midoro-Horiuti,et al. Estrogen effects in allergy and asthma , 2013, Current opinion in allergy and clinical immunology.
[62] K. Dahlman-Wright,et al. The diversity of sex steroid action: regulation of metabolism by estrogen signaling. , 2012, The Journal of endocrinology.
[63] J. McLachlan,et al. Epigenetics, evolution, endocrine disruption, health, and disease. , 2006, Endocrinology.
[64] D. Postma,et al. Down-Regulation of E-Cadherin in Human Bronchial Epithelial Cells Leads to Epidermal Growth Factor Receptor-Dependent Th2 Cell-Promoting Activity1 , 2007, The Journal of Immunology.