Eotaxin-Rich Proangiogenic Hematopoietic Progenitor Cells and CCR3+ Endothelium in the Atopic Asthmatic Response
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R. Dweik | S. Erzurum | A. Vasanji | M. Rothenberg | B. Anand-Apte | K. Asosingh | A. Janocha | Aaron Tipton | Kimberly Queisser | N. Wanner | D. Grandon
[1] T. Kanda,et al. Interleukin-25 expressed by brain capillary endothelial cells maintains blood-brain barrier function in a protein kinase Cϵ-dependent manner. , 2016, The Journal of Biological Chemistry.
[2] P. O'Byrne,et al. Thymic stromal lymphopoietin and IL-33 modulate migration of hematopoietic progenitor cells in patients with allergic asthma. , 2015, The Journal of allergy and clinical immunology.
[3] D. Laskowski,et al. Carboxyhemoglobin and Methemoglobin in Asthma , 2015, Lung.
[4] S. Erzurum,et al. Biology and flow cytometry of proangiogenic hematopoietic progenitors cells , 2015, Cytometry. Part A : the journal of the International Society for Analytical Cytology.
[5] Rosalind J Wright,et al. Future Research Directions in Asthma. An NHLBI Working Group Report. , 2004, American journal of respiratory and critical care medicine.
[6] R. Flavell,et al. TH2, allergy and group 2 innate lymphoid cells , 2013, Nature Immunology.
[7] S. Erzurum,et al. Nascent Endothelium Initiates Th2 Polarization of Asthma , 2013, The Journal of Immunology.
[8] W. Busse,et al. Future Research Directions in Asthma , 2012 .
[9] M. Schaller,et al. IL-25 induces type 2 cytokine production in a novel, steroid resistant IL-17RB+ myeloid population that exacerbates asthmatic pathology , 2012, Nature Medicine.
[10] M. Schaller,et al. Interleukin-25 induces type 2 cytokine production in a steroid-resistant interleukin-17 RB + myeloid population that exacerbates asthmatic pathology , 2012 .
[11] P. O'Byrne,et al. Lung homing of endothelial progenitor cells in humans with asthma after allergen challenge. , 2011, American journal of respiratory and critical care medicine.
[12] Margaret W Leigh,et al. An official ATS clinical practice guideline: interpretation of exhaled nitric oxide levels (FENO) for clinical applications. , 2011, American journal of respiratory and critical care medicine.
[13] S. Erzurum,et al. Pro-angiogenic hematopoietic progenitor cells and endothelial colony-forming cells in pathological angiogenesis of bronchial and pulmonary circulation , 2011, Angiogenesis.
[14] S. Nakae,et al. IL-33 and Airway Inflammation , 2011, Allergy, asthma & immunology research.
[15] R. Sehmi,et al. Modulating progenitor accumulation attenuates lung angiogenesis in a mouse model of asthma , 2010, European Respiratory Journal.
[16] S. Erzurum,et al. Allergen-induced, eotaxin-rich, proangiogenic bone marrow progenitors: a blood-borne cellular envoy for lung eosinophilia. , 2010, The Journal of allergy and clinical immunology.
[17] P. Teeling,et al. Differential expression of interleukin‐17 family cytokines in intact and complicated human atherosclerotic plaques , 2010, The Journal of pathology.
[18] 茂呂 和世. Innate production of T[H]2 cytokines by adipose tissue-associated c-Kit[+]Sca-1[+] lymphoid cells , 2010 .
[19] N. Lukacs,et al. Pulmonary IL-17E (IL-25) Production and IL-17RB+ Myeloid Cell-Derived Th2 Cytokine Production Are Dependent upon Stem Cell Factor-Induced Responses during Chronic Allergic Pulmonary Disease1 , 2009, The Journal of Immunology.
[20] T. Kanda,et al. Interleukin-25 Expressed by Brain Capillary Endothelial Cells Maintains Blood-Brain Barrier Function in a Protein Kinase Cϵ-dependent Manner* , 2009, The Journal of Biological Chemistry.
[21] S. Erzurum,et al. Angioplasticity in asthma. , 2009, Biochemical Society transactions.
[22] Justine R. Smith,et al. CCR3 is a target for age-related macular degeneration diagnosis and therapy , 2009, Nature.
[23] C. Lam,et al. Intracellular JNK, p38 MAPK and NF-kappaB regulate IL-25 induced release of cytokines and chemokines from costimulated T helper lymphocytes. , 2007, Immunology letters.
[24] Yong‐jun Liu,et al. IL-25 augments type 2 immune responses by enhancing the expansion and functions of TSLP-DC–activated Th2 memory cells , 2007, The Journal of experimental medicine.
[25] Yong‐jun Liu,et al. Interleukin 25 promotes the initiation of proallergic type 2 responses , 2007, The Journal of experimental medicine.
[26] S. Erzurum,et al. Th1- and Th2-Dependent Endothelial Progenitor Cell Recruitment and Angiogenic Switch in Asthma1 , 2007, The Journal of Immunology.
[27] L. Hassman,et al. A central regulatory role for eosinophils and the eotaxin/CCR3 axis in chronic experimental allergic airway inflammation , 2006, Proceedings of the National Academy of Sciences.
[28] T. Mcclanahan,et al. Interleukin 25 regulates type 2 cytokine-dependent immunity and limits chronic inflammation in the gastrointestinal tract , 2006, The Journal of experimental medicine.
[29] Niamh E Mangan,et al. Identification of an interleukin (IL)-25–dependent cell population that provides IL-4, IL-5, and IL-13 at the onset of helminth expulsion , 2006, The Journal of experimental medicine.
[30] N. Lukacs,et al. A closer look at chemokines and their role in asthmatic responses. , 2006, European journal of pharmacology.
[31] B. Aronow,et al. Eotaxin-3 and a uniquely conserved gene-expression profile in eosinophilic esophagitis. , 2006, The Journal of clinical investigation.
[32] J Fernando Bazan,et al. IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines. , 2005, Immunity.
[33] M. Karow,et al. The Eotaxin Chemokines and CCR3 Are Fundamental Regulators of Allergen-Induced Pulmonary Eosinophilia1 , 2005, The Journal of Immunology.
[34] S. Peters,et al. Airway remodeling contributes to the progressive loss of lung function in asthma: an overview. , 2005, The Journal of allergy and clinical immunology.
[35] Nikolaos M. Nikolaidis,et al. Identification of a Cooperative Mechanism Involving Interleukin-13 and Eotaxin-2 in Experimental Allergic Lung Inflammation* , 2005, Journal of Biological Chemistry.
[36] S. Orkin,et al. A Critical Role for Eosinophils in Allergic Airways Remodeling , 2004, Science.
[37] Raed A Dweik,et al. Temporal association of nitric oxide levels and airflow in asthma after whole lung allergen challenge. , 2003, Journal of applied physiology.
[38] Kotaro Suzuki,et al. Mast cells produce interleukin-25 upon FcεRI-mediated activation , 2003 .
[39] L. Claesson‐Welsh,et al. A novel function for tissue inhibitor of metalloproteinases-3 (TIMP3): inhibition of angiogenesis by blockage of VEGF binding to VEGF receptor-2 , 2003, Nature Medicine.
[40] Concerted expression of eotaxin-1, eotaxin-2, and eotaxin-3 in human bronchial epithelial cells. , 2003, Cellular immunology.
[41] James J. Lee,et al. Ablation of eosinophils leads to a reduction of allergen-induced pulmonary pathology. , 2003, American journal of physiology. Lung cellular and molecular physiology.
[42] Kotaro Suzuki,et al. Mast cells produce interleukin-25 upon Fc epsilon RI-mediated activation. , 2003, Blood.
[43] R. Coffman,et al. New IL-17 Family Members Promote Th1 or Th2 Responses in the Lung: In Vivo Function of the Novel Cytokine IL-251 , 2002, The Journal of Immunology.
[44] A. Gurney,et al. Forced Expression of Murine IL-17E Induces Growth Retardation, Jaundice, a Th2-Biased Response, and Multiorgan Inflammation in Mice , 2001, The Journal of Immunology.
[45] M. Leach,et al. IL-25 induces IL-4, IL-5, and IL-13 and Th2-associated pathologies in vivo. , 2001, Immunity.
[46] G Salvato,et al. Quantitative and morphological analysis of the vascular bed in bronchial biopsy specimens from asthmatic and non-asthmatic subjects , 2001, Thorax.
[47] J. Ward,et al. Eotaxin (CCL11) Induces In Vivo Angiogenic Responses by Human CCR3+ Endothelial Cells1 , 2001, The Journal of Immunology.
[48] T. Williams,et al. Eotaxin and the attraction of eosinophils to the asthmatic lung , 2001, Respiratory research.
[49] R. Djukanović,et al. Bronchial angiogenesis in severe glucocorticoid-dependent asthma. , 2000, The European respiratory journal.
[50] M. Jordana,et al. IL-10 is necessary for the expression of airway hyperresponsiveness but not pulmonary inflammation after allergic sensitization. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[51] M. Humbert,et al. Eosinophil chemotactic chemokines (eotaxin, eotaxin-2, RANTES, monocyte chemoattractant protein-3 (MCP-3), and MCP-4), and C-C chemokine receptor 3 expression in bronchial biopsies from atopic and nonatopic (Intrinsic) asthmatics. , 1999, Journal of immunology.
[52] M. Rothenberg,et al. Eotaxin. An essential mediator of eosinophil trafficking into mucosal tissues. , 1999, American journal of respiratory cell and molecular biology.
[53] R. Coffman,et al. Depletion of eosinophils in mice through the use of antibodies specific for C‐C chemokine receptor 3 (CCR3) , 1999, Journal of leukocyte biology.
[54] J. Drazen,et al. Contribution of Nitric Oxide Synthases 1, 2, and 3 to Airway Hyperresponsiveness and Inflammation in a Murine Model of Asthma , 1999, The Journal of experimental medicine.
[55] C. Mackay,et al. Enhanced expression of eotaxin and CCR3 mRNA and protein in atopic asthma. Association with airway hyperresponsiveness and predominant co‐localization of eotaxin mRNA to bronchial epithelial and endothelial cells , 1997, European journal of immunology.
[56] J. Wilson,et al. Increased vascularity of the bronchial mucosa in mild asthma. , 1997, American journal of respiratory and critical care medicine.
[57] C. Mackay,et al. Chemokine receptor usage by human eosinophils. The importance of CCR3 demonstrated using an antagonistic monoclonal antibody. , 1997, The Journal of clinical investigation.
[58] A. Luster,et al. Identification of a mouse eosinophil receptor for the CC chemokine eotaxin. , 1996, Biochemical and biophysical research communications.
[59] C. Mackay,et al. Molecular cloning and characterization of a human eotaxin receptor expressed selectively on eosinophils , 1996, The Journal of experimental medicine.
[60] J. Demartino,et al. Cloning, expression, and characterization of the human eosinophil eotaxin receptor , 1996, The Journal of experimental medicine.
[61] P. Leder,et al. Human eotaxin is a specific chemoattractant for eosinophil cells and provides a new mechanism to explain tissue eosinophilia , 1996, Nature Medicine.
[62] W Newman,et al. Cloning of the human eosinophil chemoattractant, eotaxin. Expression, receptor binding, and functional properties suggest a mechanism for the selective recruitment of eosinophils. , 1996, The Journal of clinical investigation.
[63] C. Combadière,et al. Cloning and Functional Expression of a Human Eosinophil CC Chemokine Receptor (*) , 1995, The Journal of Biological Chemistry.
[64] D. Kioussis,et al. Direct derivation of conditionally immortal cell lines from an H-2Kb-tsA58 transgenic mouse. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[65] D. Kioussis,et al. H‐2Kb‐tsA58トランスジェニックマウスから直接に無限増殖細胞系を得る方法 , 1991 .