Localization and modulation of α1D (Cav1.3) L-type Ca channel by protein kinase A

α1D L-type Ca channel was assumed to be of neuroendocrine origin only; however, α1D L-type Ca channel knockout mice exhibit sinus bradycardia and atrioventricular block, indicating a distinct role ...

[1]  W. Catterall,et al.  β-Adrenergic regulation requires direct anchoring of PKA to cardiac CaV1.2 channels via a leucine zipper interaction with A kinase-anchoring protein 15 , 2003, Proceedings of the National Academy of Sciences of the United States of America.

[2]  Jörg Striessnig,et al.  Functional role of L-type Cav1.3 Ca2+ channels in cardiac pacemaker activity , 2003, Proceedings of the National Academy of Sciences of the United States of America.

[3]  R. Kass,et al.  Requirement of subunit expression for cAMP-mediated regulation of a heart potassium channel , 2003, Proceedings of the National Academy of Sciences of the United States of America.

[4]  Zhao Zhang,et al.  Functional Roles of Ca(v)1.3 (alpha(1D)) calcium channel in sinoatrial nodes: insight gained using gene-targeted null mutant mice. , 2002, Circulation research.

[5]  Mohamed Chahine,et al.  Expression and Intracellular Localization of an SCN5A Double Mutant R1232W/T1620M Implicated in Brugada Syndrome , 2002, Circulation research.

[6]  M. Boutjdir,et al.  Gene Expression of SERCA2a and L- and T-type Ca Channels during Human Heart Development , 2001, Pediatric Research.

[7]  Weifeng Xu,et al.  Neuronal CaV1.3α1 L-Type Channels Activate at Relatively Hyperpolarized Membrane Potentials and Are Incompletely Inhibited by Dihydropyridines , 2001, The Journal of Neuroscience.

[8]  Aj Butcher,et al.  Biophysical Properties, Pharmacology, and Modulation of Human, Neuronal L-Type (α1D, CaV1.3) Voltage-Dependent Calcium Currents , 2001 .

[9]  S. Nattel,et al.  Ionic Remodeling in the Heart: Pathophysiological Significance and New Therapeutic Opportunities for Atrial Fibrillation , 2000, Circulation research.

[10]  J. Engel,et al.  Congenital Deafness and Sinoatrial Node Dysfunction in Mice Lacking Class D L-Type Ca2+ Channels , 2000, Cell.

[11]  M. Boutjdir,et al.  Gene expression of Na+/Ca2+ exchanger during development in human heart. , 2000, Cardiovascular research.

[12]  R. Tsien,et al.  Nomenclature of Voltage-Gated Calcium Channels , 2000, Neuron.

[13]  Gail Mandel,et al.  Nomenclature of Voltage-Gated Sodium Channels , 2000, Neuron.

[14]  J Jalife,et al.  Stable microreentrant sources as a mechanism of atrial fibrillation in the isolated sheep heart. , 2000, Circulation.

[15]  M. Lesh,et al.  Electrophysiologic Effects of Selective Right versus Left Atrial Linear Lesions in a Canine Model of Chronic Atrial Fibrillation , 1999, Journal of cardiovascular electrophysiology.

[16]  M. Bünemann,et al.  Functional Regulation of L-type Calcium Channels via Protein Kinase A-mediated Phosphorylation of the β2 Subunit* , 1999, The Journal of Biological Chemistry.

[17]  R F Bosch,et al.  Ionic mechanisms of electrical remodeling in human atrial fibrillation. , 1999, Cardiovascular research.

[18]  S. Nattel.  Ionic determinants of atrial fibrillation and Ca2+ channel abnormalities : cause, consequence, or innocent bystander? , 1999, Circulation research.

[19]  J. Nerbonne,et al.  Atrial L-type Ca2+ currents and human atrial fibrillation. , 1999, Circulation research.

[20]  M. Boutjdir,et al.  Serum and Immunoglobulin G from the Mother of a Child with Congenital Heart Block Induce Conduction Abnormalities and Inhibit L-Type Calcium Channels in a Rat Heart Model , 1998, Pediatric Research.

[21]  J. Nerbonne,et al.  Distribution, Splicing and Glucocorticoid-Induced Expression of Cardiacα1Candα1DVoltage-gated Ca2+Channel mRNAs , 1997 .

[22]  J. Jurevičius,et al.  Beta-2 adrenergic activation of L-type Ca++ current in cardiac myocytes. , 1997, The Journal of pharmacology and experimental therapeutics.

[23]  S. Green,et al.  cAMP-Dependent Regulation of Cardiac L-Type Ca2+ Channels Requires Membrane Targeting of PKA and Phosphorylation of Channel Subunits , 1997, Neuron.

[24]  M. Boutjdir,et al.  C2 Region–Derived Peptides of β-Protein Kinase C Regulate Cardiac Ca2+ Channels , 1997 .

[25]  H. Glossmann,et al.  Identification of PK-A phosphorylation sites in the carboxyl terminus of L-type calcium channel alpha 1 subunits. , 1996, Biochemistry.

[26]  N. Maruyama,et al.  Gene regulation of senescence marker protein-30 (SMP30): coordinated up-regulation with tissue maturation and gradual down-regulation with aging , 1996, Mechanisms of Ageing and Development.

[27]  N. Dascal,et al.  A potential site of functional modulation by protein kinase A in the cardiac Ca2+ channel α 1C subunit , 1996, FEBS letters.

[28]  F. Hofmann,et al.  On the regulation of the expressed L-type calcium channel by cAMP-dependent phosphorylation , 1995, Pflügers Archiv.

[29]  Douglas L. Jones,et al.  Chronic rapid atrial pacing. Structural, functional, and electrophysiological characteristics of a new model of sustained atrial fibrillation. , 1995, Circulation.

[30]  S. Green,et al.  Stable expression and coupling of cardiac L-type Ca2+ channels with beta 1-adrenoceptors. , 1995, Circulation research.

[31]  G. Yellen,et al.  Visual identification of individual transfected cells for electrophysiology using antibody-coated beads. , 1994, BioTechniques.

[32]  Jules C. Hancox,et al.  A method for isolating rabbit atrioventricular node myocytes which retain normal morphology and function. , 1993, The American journal of physiology.

[33]  H. Takeshima,et al.  Cyclic AMP‐dependent phosphorylation and regulation of the cardiac dihydropyridine‐sensitive Ca channel , 1992, Annals of the New York Academy of Sciences.

[34]  K. Itagaki,et al.  β-Subunit expression is required for cAMP-dependent increase of cloned cardiac and vascular calcium channel currents , 1992, Pflügers Archiv.

[35]  P. Lory,et al.  Cyclic AMP-dependent modulation of cardiac Ca channels expressed in Xenopus laevis oocytes. , 1992, Biochemical and biophysical research communications.

[36]  J. Bockaert,et al.  Serotonin increases calcium current in human atrial myocytes via the newly described 5-hydroxytryptamine4 receptors. , 1992, Molecular pharmacology.

[37]  J C Denyer,et al.  Rabbit sino‐atrial node cells: isolation and electrophysiological properties. , 1990, The Journal of physiology.

[38]  M. Caron,et al.  Beta-adrenergic receptors: biochemical mechanisms of physiological regulation. , 1984, Physiological reviews.

[39]  J. Daly,et al.  Forskolin: unique diterpene activator of adenylate cyclase in membranes and in intact cells. , 1981, Proceedings of the National Academy of Sciences of the United States of America.

[40]  K. Page,et al.  Biophysical properties, pharmacology, and modulation of human, neuronal L-type (alpha(1D), Ca(V)1.3) voltage-dependent calcium currents. , 2001, Journal of neurophysiology.

[41]  J. Nerbonne,et al.  Distribution, splicing and glucocorticoid-induced expression of cardiac alpha 1C and alpha 1D voltage-gated Ca2+ channel mRNAs. , 1997, Journal of molecular and cellular cardiology.

[42]  P. Lory,et al.  Molecular basis of the diversity of calcium channels in cardiovascular tissues. , 1997, European heart journal.

[43]  A. Harada,et al.  Atrial activation during chronic atrial fibrillation in patients with isolated mitral valve disease. , 1996, The Annals of thoracic surgery.

[44]  A. Brown,et al.  Comparison of the densities of 5-HT4 receptors, beta 1- and beta 2-adrenoceptors in human atrium: functional implications. , 1996, Naunyn-Schmiedeberg's archives of pharmacology.

[45]  H Reuter,et al.  Ion channels in cardiac cell membranes. , 1984, Annual review of physiology.

[46]  W. C. Randall,et al.  Nervous control of cardiovascular function , 1984 .