A mouse model links asthma susceptibility to prenatal exposure to diesel exhaust.
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[1] J. Suen,et al. A common environmental pollutant, 4‐nonylphenol, promotes allergic lung inflammation in a murine model of asthma , 2013, Allergy.
[2] Shau-ku Huang,et al. PHAGOCYTES , GRANULOCYTES , AND MYELOPOIESIS Aryl hydrocarbon receptor controls murine mast cell homeostasis , 2013 .
[3] Inhibition of endotoxin‐induced perinatal asthma protection by pollutants in an experimental mouse model , 2013, Allergy.
[4] A. Comellas,et al. Protein kinase C-ζ mediates lung injury induced by diesel exhaust particles. , 2013, American journal of respiratory cell and molecular biology.
[5] F. Perera,et al. Early-life cockroach allergen and polycyclic aromatic hydrocarbon exposures predict cockroach sensitization among inner-city children. , 2013, The Journal of allergy and clinical immunology.
[6] E. Israel,et al. Lipoxin A4 Regulates Natural Killer Cell and Type 2 Innate Lymphoid Cell Activation in Asthma , 2013, Science Translational Medicine.
[7] W. M. Foster,et al. Maternal diesel inhalation increases airway hyperreactivity in ozone-exposed offspring. , 2012, American journal of respiratory cell and molecular biology.
[8] L. Akinbami,et al. National surveillance of asthma: United States, 2001-2010. , 2012, Vital & health statistics. Series 3, Analytical and epidemiological studies.
[9] M. Lommatzsch. Airway Hyperresponsiveness: New Insights into the Pathogenesis , 2012, Seminars in Respiratory and Critical Care Medicine.
[10] M. Colombo,et al. The Aryl Hydrocarbon Receptor Modulates Acute and Late Mast Cell Responses , 2012, The Journal of Immunology.
[11] Ling Lu,et al. IL-17A-producing NK cells were implicated in liver injury induced by ischemia and reperfusion. , 2012, International immunopharmacology.
[12] J. T. Chang,et al. Aryl hydrocarbon receptor in association with RelA modulates IL-6 expression in non-smoking lung cancer , 2012, Oncogene.
[13] D. Cook,et al. Prenatal and Passive Smoke Exposure and Incidence of Asthma and Wheeze: Systematic Review and Meta-analysis , 2012, Pediatrics.
[14] Antonia M. Calafat,et al. Prenatal Exposure to Bisphenol A and Child Wheeze from Birth to 3 Years of Age , 2012, Environmental health perspectives.
[15] Y. Fujitani,et al. Effects of nanoparticle-rich diesel exhaust particles on IL-17 production in vitro , 2012, Journal of immunotoxicology.
[16] Lidia Morawska,et al. Respiratory health effects of diesel particulate matter , 2012, Respirology.
[17] Liang Zhou,et al. The aryl hydrocarbon receptor regulates gut immunity through modulation of innate lymphoid cells. , 2012, Immunity.
[18] Hua Huang,et al. ERK1 is important for Th2 differentiation and development of experimental asthma , 2012, The FASEB Journal.
[19] E. Hobeika,et al. Natural Aryl Hydrocarbon Receptor Ligands Control Organogenesis of Intestinal Lymphoid Follicles , 2011, Science.
[20] D. Remick,et al. Cardiovascular , Pulmonary , and Renal Pathology Diesel Exhaust Particulates Exacerbate Asthma-Like Inflammation by Increasing CXC Chemokines , 2011 .
[21] Andrew Rundle,et al. Traffic density and stationary sources of air pollution associated with wheeze, asthma, and immunoglobulin E from birth to age 5 years among New York City children. , 2011, Environmental research.
[22] L. Boon,et al. Activation of the aryl hydrocarbon receptor suppresses sensitization in a mouse peanut allergy model. , 2011, Toxicological sciences : an official journal of the Society of Toxicology.
[23] Yongjian Xu,et al. Increased expression of aryl hydrocarbon receptor and interleukin 22 in patients with allergic asthma. , 2011, Asian Pacific journal of allergy and immunology.
[24] B. Levy,et al. NK Cells Are Effectors for Resolvin E1 in the Timely Resolution of Allergic Airway Inflammation , 2011, The Journal of Immunology.
[25] F. Perera,et al. Prenatal exposure to polycyclic aromatic hydrocarbons, environmental tobacco smoke and asthma. , 2011, Respiratory medicine.
[26] P. Foster,et al. An Alternate STAT6-Independent Pathway Promotes Eosinophil Influx into Blood during Allergic Airway Inflammation , 2011, PloS one.
[27] Joseph C. Sun,et al. Homeostatic proliferation generates long-lived natural killer cells that respond against viral infection , 2011, The Journal of experimental medicine.
[28] M. Veldhoen,et al. The aryl hydrocarbon receptor: fine-tuning the immune-response. , 2010, Current opinion in immunology.
[29] S. Jeong,et al. Asian dust and titanium dioxide particles–induced inflammation and oxidative DNA damage in C57BL/6 mice , 2010, Inhalation toxicology.
[30] D. Raulet,et al. Mature natural killer cells reset their responsiveness when exposed to an altered MHC environment , 2010, The Journal of experimental medicine.
[31] C. Ple,et al. Natural Killer Cells Accumulate in Lung‐Draining Lymph Nodes and Regulate Airway Eosinophilia in a Murine Model of Asthma , 2010, Scandinavian journal of immunology.
[32] M. Caligiuri,et al. Interleukin-1beta selectively expands and sustains interleukin-22+ immature human natural killer cells in secondary lymphoid tissue. , 2010, Immunity.
[33] Zhi Ning,et al. Ambient ultrafine particles provide a strong adjuvant effect in the secondary immune response: implication for traffic-related asthma flares. , 2010, American journal of physiology. Lung cellular and molecular physiology.
[34] R. Luebke,et al. Effects of prenatal diesel exhaust inhalation on pulmonary inflammation and development of specific immune responses. , 2010, Toxicology letters.
[35] F. Perera,et al. Intrauterine exposure to polycyclic aromatic hydrocarbons, fine particulate matter and early wheeze. Prospective birth cohort study in 4‐year olds , 2010, Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology.
[36] F. Perera,et al. Prenatal allergen and diesel exhaust exposure and their effects on allergy in adult offspring mice , 2010, Allergy, asthma, and clinical immunology : official journal of the Canadian Society of Allergy and Clinical Immunology.
[37] R. Alam,et al. Uncoordinated 119 Preferentially Induces Th2 Differentiation and Promotes the Development of Asthma , 2010, The Journal of Immunology.
[38] Kiros Berhane,et al. Childhood Incident Asthma and Traffic-Related Air Pollution at Home and School , 2010, Environmental health perspectives.
[39] C. Hunter,et al. IL-6 Promotes NK Cell Production of IL-17 during Toxoplasmosis , 2010, The Journal of Immunology.
[40] F. Muskens,et al. Both Conventional and Interferon Killer Dendritic Cells Have Antigen-Presenting Capacity during Influenza Virus Infection , 2009, PloS one.
[41] Emily A. Stevens,et al. The aryl hydrocarbon receptor: a perspective on potential roles in the immune system , 2009, Immunology.
[42] D. Jarvis,et al. Association between modelled traffic-related air pollution and asthma score in the ECRHS , 2009, European Respiratory Journal.
[43] N. Goplen,et al. Combined sensitization of mice to extracts of dust mite, ragweed, and Aspergillus species breaks through tolerance and establishes chronic features of asthma. , 2009, The Journal of allergy and clinical immunology.
[44] Petros Koutrakis,et al. Symptoms and Medication Use in Children with Asthma and Traffic-Related Sources of Fine Particle Pollution , 2009, Environmental health perspectives.
[45] M. E. Hahn,et al. Regulation of constitutive and inducible AHR signaling: complex interactions involving the AHR repressor. , 2009, Biochemical pharmacology.
[46] M. Veldhoen,et al. Natural agonists for aryl hydrocarbon receptor in culture medium are essential for optimal differentiation of Th17 T cells , 2009, The Journal of experimental medicine.
[47] J. Douwes,et al. Farm exposure in utero may protect against asthma, hay fever and eczema , 2008, European Respiratory Journal.
[48] J. Buer,et al. The aryl hydrocarbon receptor links TH17-cell-mediated autoimmunity to environmental toxins , 2008, Nature.
[49] Eric Vivier,et al. Functions of natural killer cells , 2008, Nature Immunology.
[50] Lester Kobzik,et al. Pulmonary exposure to particles during pregnancy causes increased neonatal asthma susceptibility. , 2008, American journal of respiratory cell and molecular biology.
[51] Pamela Ohman-Strickland,et al. Respiratory effects of exposure to diesel traffic in persons with asthma. , 2007, The New England journal of medicine.
[52] T. Nutman,et al. Filarial Parasites Induce NK Cell Activation, Type 1 and Type 2 Cytokine Secretion, and Subsequent Apoptotic Cell Death1 , 2007, The Journal of Immunology.
[53] M. Gaestel,et al. MK2 controls the level of negative feedback in the NF-κB pathway and is essential for vascular permeability and airway inflammation , 2007, The Journal of experimental medicine.
[54] Charles Ris,et al. U.S. EPA Health Assessment for Diesel Engine Exhaust: A Review , 2007, Inhalation toxicology.
[55] M. Fujii,et al. Polycyclic aromatic hydrocarbons aggravate antigen-induced nasal blockage in experimental allergic rhinitis. , 2007, Journal of pharmacological sciences.
[56] K. Bock,et al. Ah receptor: dioxin-mediated toxic responses as hints to deregulated physiologic functions. , 2006, Biochemical pharmacology.
[57] M. Madden,et al. Effects of diesel exhaust particles on human alveolar macrophage ability to secrete inflammatory mediators in response to lipopolysaccharide. , 2006, Toxicology in vitro : an international journal published in association with BIBRA.
[58] B. Wallaert,et al. Diesel exhaust exposure favors TH2 cell recruitment in nonatopic subjects by differentially regulating chemokine production. , 2006, The Journal of allergy and clinical immunology.
[59] B. Brunekreef,et al. Prenatal farm exposure is related to the expression of receptors of the innate immunity and to atopic sensitization in school-age children. , 2006, The Journal of allergy and clinical immunology.
[60] Kiros Berhane,et al. Traffic, Susceptibility, and Childhood Asthma , 2006, Environmental health perspectives.
[61] T. Sandström,et al. Airway antioxidant and inflammatory responses to diesel exhaust exposure in healthy humans , 2006, European Respiratory Journal.
[62] Masayuki Yamamoto,et al. Effects of Aryl Hydrocarbon Receptor Signaling on the Modulation of Th1/Th2 Balance1 , 2005, The Journal of Immunology.
[63] F. Gonzalez,et al. Over‐production of IFN‐γ and IL‐12 in AhR‐null mice , 2005 .
[64] H. Aburatani,et al. Constitutive Expression of Aryl Hydrocarbon Receptor in Keratinocytes Causes Inflammatory Skin Lesions , 2005, Molecular and Cellular Biology.
[65] T. Matsushita,et al. Abnormal natural killer cell function in systemic sclerosis: altered cytokine production and defective killing activity. , 2005, The Journal of investigative dermatology.
[66] D. Bernstein,et al. Is it traffic type, volume, or distance? Wheezing in infants living near truck and bus traffic. , 2005, The Journal of allergy and clinical immunology.
[67] D. Warshawsky,et al. Metabolic activation of polycyclic and heterocyclic aromatic hydrocarbons and DNA damage: a review. , 2005, Toxicology and applied pharmacology.
[68] C. Akdis,et al. Different natural killer (NK) receptor expression and immunoglobulin E (IgE) regulation by NK1 and NK2 cells , 2005, Clinical and experimental immunology.
[69] R. Sun,et al. Involvement of human natural killer cells in asthma pathogenesis: natural killer 2 cells in type 2 cytokine predominance. , 2005, The Journal of allergy and clinical immunology.
[70] M. Kadiiska,et al. Synergistic production of lung free radicals by diesel exhaust particles and endotoxin. , 2005, American journal of respiratory and critical care medicine.
[71] Ning Li,et al. Nrf2 Is a Key Transcription Factor That Regulates Antioxidant Defense in Macrophages and Epithelial Cells: Protecting against the Proinflammatory and Oxidizing Effects of Diesel Exhaust Chemicals1 , 2004, The Journal of Immunology.
[72] F. Lauer,et al. Environmental polycyclic aromatic hydrocarbons, benzo(a) pyrene (BaP) and BaP-quinones, enhance IgE-mediated histamine release and IL-4 production in human basophils. , 2003, Clinical immunology.
[73] R. Luebke,et al. Suppression of allergic immune responses to house dust mite (HDM) in rats exposed to 2,3,7,8-TCDD. , 2001, Toxicological sciences : an official journal of the Society of Toxicology.
[74] T. Sandström,et al. Diesel exhaust enhances airway responsiveness in asthmatic subjects. , 2001, The European respiratory journal.
[75] A. Nel,et al. In vivo nasal challenge with diesel exhaust particles enhances expression of the CC chemokines rantes, MIP-1alpha, and MCP-3 in humans. , 2000, Clinical immunology.
[76] P. Cullinan,et al. Airway inflammation after controlled exposure to diesel exhaust particulates. , 2000, American journal of respiratory and critical care medicine.
[77] M. Li‐Weber,et al. The environmental pollutant pyrene induces the production of IL-4. , 2000, The Journal of allergy and clinical immunology.
[78] S T Holgate,et al. Acute inflammatory responses in the airways and peripheral blood after short-term exposure to diesel exhaust in healthy human volunteers. , 1999, American journal of respiratory and critical care medicine.
[79] H. Ljunggren,et al. Natural Killer Cells Determine Development of Allergen-induced Eosinophilic Airway Inflammation in Mice , 1999, The Journal of experimental medicine.
[80] A E Nel,et al. Enhancement of allergic inflammation by the interaction between diesel exhaust particles and the immune system. , 1998, The Journal of allergy and clinical immunology.
[81] S. Cammisuli,et al. IL-5 production by NK cells contributes to eosinophil infiltration in a mouse model of allergic inflammation. , 1998, Journal of immunology.
[82] A. Litonjua,et al. Parental history and the risk for childhood asthma. Does mother confer more risk than father? , 1998, American journal of respiratory and critical care medicine.
[83] H. Takano,et al. Diesel exhaust enhances allergic airway inflammation and hyperresponsiveness in mice. , 1998, American journal of respiratory and critical care medicine.
[84] R. Hagemann,et al. The adjuvant activity of diesel exhaust particles and carbon black on systemic IgE production to ovalbumin in mice after intranasal instillation. , 1997, Toxicology.
[85] D. Diat-Sanchez,et al. The role of diesel exhaust particles and their associated polyaromatic hydrocarbons in the induction of allergic airway disease , 1997 .
[86] R. Bravo,et al. Monocyte chemotactic protein-3 (MCP-3)/fibroblast-induced cytokine (FIC) in eosinophilic inflammation of the airways and the inhibitory effects of an anti-MCP-3/FIC antibody. , 1997, Journal of immunology.
[87] S. Durham,et al. Upregulation of αGM-CSF-receptor in nonatopic asthma but not in atopic asthma ☆ ☆☆ ★ ★★ , 1997 .
[88] A. Saxon,et al. Combined diesel exhaust particulate and ragweed allergen challenge markedly enhances human in vivo nasal ragweed-specific IgE and skews cytokine production to a T helper cell 2-type pattern. , 1997, Journal of immunology.
[89] S. Durham,et al. Upregulation of alpha GM-CSF-receptor in nonatopic asthma but not in atopic asthma. , 1997, The Journal of allergy and clinical immunology.
[90] M. Sears,et al. Parental and neonatal risk factors for atopy, airway hyper-responsiveness, and asthma. , 1996, Archives of disease in childhood.
[91] J. Reddy,et al. Characterization of a murine Ahr null allele: involvement of the Ah receptor in hepatic growth and development. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[92] T. Suzuki,et al. Adjuvant activities of pyrene, anthracene, fluoranthene and benzo(a)pyrene in production of anti-IgE antibody to Japanese cedar pollen allergen in mice. , 1996, Journal of clinical & laboratory immunology.
[93] H. Fujimaki,et al. Intranasal instillation of diesel exhaust particulates and antigen in mice modulated cytokine productions in cervical lymph node cells. , 1995, International archives of allergy and immunology.
[94] H. Fujimaki,et al. IL-4 production in mediastinal lymph node cells in mice intratracheally instilled with diesel exhaust particulates and antigen. , 1994, Toxicology.
[95] T. Suzuki,et al. The adjuvant activity of pyrene in diesel exhaust on IgE antibody production in mice. , 1993, Arerugi = [Allergy].
[96] D. M. Kemény,et al. Higher risk of infantile atopic dermatitis from maternal atopy than from paternal atopy , 1992, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[97] T. Miyamoto,et al. Adjuvant activity of diesel-exhaust particulates for the production of IgE antibody in mice. , 1986, The Journal of allergy and clinical immunology.
[98] P. Srivastava,et al. Fetal translocation and metabolism of PAH obtained from coal fly ash given intratracheally to pregnant rats. , 1986, Journal of toxicology and environmental health.
[99] F. Dixon,et al. IMMUNOLOGIC UNRESPONSIVENESS INDUCED BY PROTEIN ANTIGENS , 1955, The Journal of experimental medicine.
[100] P. Medawar,et al. ‘Actively Acquired Tolerance’ of Foreign Cells , 1953, Nature.