Correction: α-MSH Stimulates Glucose Uptake in Mouse Muscle and Phosphorylates Rab-GTPase-Activating Protein TBC1D1 Independently of AMPK
暂无分享,去创建一个
B. Wulff | J. Wojtaszewski | M. Cowley | S. Litwak | K. Raun | T. Jensen | P. Enriori | Møller | R. Kjøbsted | C. Garcia-Rudaz | Cathrine Laustrup | Cecilia Garcia-Rudaz
[1] F. Ross,et al. PT-1 selectively activates AMPK-γ1 complexes in mouse skeletal muscle, but activates all three γ subunit complexes in cultured human cells by inhibiting the respiratory chain. , 2015, The Biochemical journal.
[2] Nolan J. Hoffman,et al. The RabGAP TBC1D1 Plays a Central Role in Exercise-Regulated Glucose Metabolism in Skeletal Muscle , 2015, Diabetes.
[3] A. Butler,et al. Melanocortin-3 receptors and metabolic homeostasis. , 2013, Progress in molecular biology and translational science.
[4] M. Khaksari,et al. Abdominal fat analyzed by DEXA scan reflects visceral body fat and improves the phenotype description and the assessment of metabolic risk in mice. , 2012, American journal of physiology. Endocrinology and metabolism.
[5] B. Holst,et al. Characterization of murine melanocortin receptors mediating adipocyte lipolysis and examination of signalling pathways involved , 2011, Molecular and Cellular Endocrinology.
[6] B. Lowell,et al. Melanocortin-4 receptors expressed by cholinergic neurons regulate energy balance and glucose homeostasis. , 2011, Cell metabolism.
[7] D. Wasserman,et al. Mice with AS160/TBC1D4-Thr649Ala Knockin Mutation Are Glucose Intolerant with Reduced Insulin Sensitivity and Altered GLUT4 Trafficking , 2011, Cell metabolism.
[8] L. Goodyear,et al. Contraction regulates site-specific phosphorylation of TBC1D1 in skeletal muscle , 2010, Biochemical Journal.
[9] K. Mountjoy. Functions for pro-opiomelanocortin-derived peptides in obesity and diabetes. , 2010, The Biochemical journal.
[10] N. Fujii,et al. TBC1D1 Regulates Insulin- and Contraction-Induced Glucose Transport in Mouse Skeletal Muscle , 2010, Diabetes.
[11] B. Budnik,et al. Insulin-stimulated Phosphorylation of the Rab GTPase-activating Protein TBC1D1 Regulates GLUT4 Translocation* , 2009, The Journal of Biological Chemistry.
[12] D. Hardie,et al. Genetic disruption of AMPK signaling abolishes both contraction- and insulin-stimulated TBC1D1 phosphorylation and 14-3-3 binding in mouse skeletal muscle. , 2009, American journal of physiology. Endocrinology and metabolism.
[13] B. Hansen,et al. A-769662 activates AMPK beta1-containing complexes but induces glucose uptake through a PI3-kinase-dependent pathway in mouse skeletal muscle. , 2009, American journal of physiology. Cell physiology.
[14] Rob C Hoeben,et al. Tbc1d1 mutation in lean mouse strain confers leanness and protects from diet-induced obesity , 2008, Nature Genetics.
[15] L. Goodyear,et al. Signaling mechanisms in skeletal muscle: Acute responses and chronic adaptations to exercise , 2008, IUBMB life.
[16] J. Klein,et al. Melanocortin crosstalk with adipose functions: ACTH directly induces insulin resistance, promotes a pro-inflammatory adipokine profile and stimulates UCP-1 in adipocytes. , 2008, The Journal of endocrinology.
[17] C. K. Song,et al. Differential activation of the sympathetic innervation of adipose tissues by melanocortin receptor stimulation. , 2007, Endocrinology.
[18] S. O’Rahilly,et al. The central melanocortin system directly controls peripheral lipid metabolism. , 2007, The Journal of clinical investigation.
[19] L. E. Pritchard,et al. Printed in U.S.A. Copyright © 2007 by The Endocrine Society doi: 10.1210/en.2006-1686 Minireview: Neuropeptide Processing and Its Impact on , 2022 .
[20] Y. Watanabe,et al. Adrenaline increases glucose transport via a Rap1‐p38MAPK pathway in rat vascular smooth muscle cells , 2007, British journal of pharmacology.
[21] G. Lienhard,et al. Substrate specificity and effect on GLUT4 translocation of the Rab GTPase-activating protein Tbc1d1. , 2007, The Biochemical journal.
[22] J. Baik,et al. Peripheral effect of alpha-melanocyte-stimulating hormone on fatty acid oxidation in skeletal muscle. , 2007, The Journal of biological chemistry.
[23] J. Wojtaszewski,et al. Predominant α2/β2/γ3 AMPK activation during exercise in human skeletal muscle , 2006, The Journal of physiology.
[24] J. Hebebrand,et al. Several mutations in the melanocortin 4 receptor gene are associated with obesity in Chinese children and adolescents , 2006, Journal of endocrinological investigation.
[25] N. Fujii,et al. Distinct Signals Regulate AS160 Phosphorylation in Response to Insulin, AICAR, and Contraction in Mouse Skeletal Muscle , 2006, Diabetes.
[26] C. K. Song,et al. Melanocortin-4 receptor mRNA is expressed in sympathetic nervous system outflow neurons to white adipose tissue. , 2005, American journal of physiology. Regulatory, integrative and comparative physiology.
[27] S. Kane,et al. AS160, the Akt substrate regulating GLUT4 translocation, has a functional Rab GTPase-activating protein domain. , 2005, The Biochemical journal.
[28] R. Cone. Anatomy and regulation of the central melanocortin system , 2005, Nature Neuroscience.
[29] J. Bryan,et al. Hypothalamic sensing of circulating fatty acids is required for glucose homeostasis , 2005, Nature Medicine.
[30] O. Pedersen,et al. Prevalence of mutations and functional analyses of melanocortin 4 receptor variants identified among 750 men with juvenile-onset obesity. , 2005, The Journal of clinical endocrinology and metabolism.
[31] M. Elia,et al. Plasma concentrations of α‐MSH, AgRP and leptin in lean and obese men and their relationship to differing states of energy balance perturbation , 2004, Clinical endocrinology.
[32] N. Hoggard,et al. Regulation of adipose tissue leptin secretion by alpha-melanocyte-stimulating hormone and agouti-related protein: further evidence of an interaction between leptin and the melanocortin signalling system. , 2004, Journal of molecular endocrinology.
[33] J. Stephens,et al. Agouti expression in human adipose tissue: functional consequences and increased expression in type 2 diabetes. , 2003, Diabetes.
[34] John M Asara,et al. Insulin-stimulated Phosphorylation of a Rab GTPase-activating Protein Regulates GLUT4 Translocation* , 2003, The Journal of Biological Chemistry.
[35] Tim Cheetham,et al. Clinical spectrum of obesity and mutations in the melanocortin 4 receptor gene. , 2003, The New England journal of medicine.
[36] S. O’Rahilly,et al. Mutations in the human melanocortin-4 receptor gene associated with severe familial obesity disrupts receptor function through multiple molecular mechanisms. , 2003, Human molecular genetics.
[37] M. Bucan,et al. A role for AMP-activated protein kinase in contraction- and hypoxia-regulated glucose transport in skeletal muscle. , 2001, Molecular cell.
[38] K. Clément,et al. Melanocortin-4 receptor mutations are a frequent and heterogeneous cause of morbid obesity. , 2000, The Journal of clinical investigation.
[39] B. Boston. The Role of Melanocortins in Adipocyte Function , 1999, Annals of the New York Academy of Sciences.
[40] Z. Abdel‐Malek,et al. The Melanocortin‐1 Receptor and Human Pigmentation , 1999, Annals of the New York Academy of Sciences.
[41] A. Grüters,et al. Severe early-onset obesity, adrenal insufficiency and red hair pigmentation caused by POMC mutations in humans , 1998, Nature Genetics.
[42] A. Klip,et al. GLUT4 translocation by insulin in intact muscle cells: detection by a fast and quantitative assay , 1998, FEBS letters.
[43] S. Manna,et al. Alpha-melanocyte-stimulating hormone inhibits the nuclear transcription factor NF-kappa B activation induced by various inflammatory agents. , 1998, Journal of immunology.
[44] J. Wojtaszewski,et al. Perfused rat hindlimb is suitable for skeletal muscle glucose transport measurements. , 1998, American journal of physiology. Endocrinology and metabolism.
[45] Ximena Opitz-Araya,et al. Exocrine Gland Dysfunction in MC5-R-Deficient Mice: Evidence for Coordinated Regulation of Exocrine Gland Function by Melanocortin Peptides , 1997, Cell.
[46] V. Hruby,et al. Biological and conformational examination of stereochemical modifications using the template melanotropin peptide, Ac-Nle-c[Asp-His-Phe-Arg-Trp-Ala-Lys]-NH2, on human melanocortin receptors. , 1997, Journal of medicinal chemistry.
[47] R. Cone,et al. Targeted Disruption of the Melanocortin-4 Receptor Results in Obesity in Mice , 1997, Cell.
[48] R. Cone,et al. Characterization of melanocortin receptor subtype expression in murine adipose tissues and in the 3T3-L1 cell line. , 1996, Endocrinology.
[49] J. Holloszy,et al. Suitability of 2-deoxyglucose for in vitro measurement of glucose transport activity in skeletal muscle. , 1994, Journal of applied physiology.
[50] E. Richter,et al. Kinetics of glucose transport in rat muscle: effects of insulin and contractions. , 1987, The American journal of physiology.
[51] D. Yaffe. Retention of differentiation potentialities during prolonged cultivation of myogenic cells. , 1968, Proceedings of the National Academy of Sciences of the United States of America.