Signaling through the interleukin-18 receptor α attenuates inflammation in cisplatin-induced acute kidney injury.

[1]  S. Holdsworth,et al.  Endogenous Tim-1 (Kim-1) promotes T-cell responses and cell-mediated injury in experimental crescentic glomerulonephritis. , 2012, Kidney international.

[2]  U. Panzer,et al.  CCR6 recruits regulatory T cells and Th17 cells to the kidney in glomerulonephritis. , 2010, Journal of the American Society of Nephrology : JASN.

[3]  K. Meldrum,et al.  IL-18 neutralization ameliorates obstruction-induced epithelial-mesenchymal transition and renal fibrosis. , 2009, Kidney international.

[4]  C. Dinarello,et al.  Increased Cytokine Production in Interleukin-18 Receptor α-deficient Cells Is Associated with Dysregulation of Suppressors of Cytokine Signaling* , 2009, The Journal of Biological Chemistry.

[5]  J. O’Shea,et al.  Cytokine signaling modules in inflammatory responses. , 2008, Immunity.

[6]  S. Holdsworth,et al.  T-bet deficiency attenuates renal injury in experimental crescentic glomerulonephritis. , 2008, Journal of the American Society of Nephrology : JASN.

[7]  C. Edelstein,et al.  Cisplatin-Induced Acute Renal Failure Is Associated with an Increase in the Cytokines Interleukin (IL)-1β, IL-18, IL-6, and Neutrophil Infiltration in the Kidney , 2007, Journal of Pharmacology and Experimental Therapeutics.

[8]  C. Dinarello,et al.  Responses of IL-18- and IL-18 receptor-deficient pancreatic islets with convergence of positive and negative signals for the IL-18 receptor , 2006, Proceedings of the National Academy of Sciences.

[9]  K. Kinoshita,et al.  All-trans-retinoic acid suppresses interferon-γ and tumor necrosis factor-α; a possible therapeutic agent for rheumatoid arthritis , 2006, Rheumatology International.

[10]  K. Kinoshita,et al.  Anti-inflammatory effect of all-trans-retinoic acid in inflammatory arthritis. , 2006, Clinical immunology.

[11]  R. Colvin,et al.  A pathophysiologic role for T lymphocytes in murine acute cisplatin nephrotoxicity. , 2006, Journal of the American Society of Nephrology : JASN.

[12]  Marek Ancukiewicz,et al.  Urine IL-18 is an early diagnostic marker for acute kidney injury and predicts mortality in the intensive care unit. , 2005, Journal of the American Society of Nephrology : JASN.

[13]  S. Chadban,et al.  Macrophage-derived interleukin-18 in experimental renal allograft rejection. , 2005, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[14]  K. Kinoshita,et al.  Blockade of IL-18 Receptor Signaling Delays the Onset of Autoimmune Disease in MRL-Faslpr Mice1 , 2004, The Journal of Immunology.

[15]  S. Akira,et al.  Contrasting action of IL-12 and IL-18 in the development of dextran sodium sulphate colitis in mice. , 2003, Scandinavian journal of gastroenterology.

[16]  Hua Liu,et al.  Cytochrome P450 2E1 null mice provide novel protection against cisplatin-induced nephrotoxicity and apoptosis. , 2003, Kidney international.

[17]  M. Kubo,et al.  SOCS1/JAB is a negative regulator of LPS-induced macrophage activation. , 2002, Immunity.

[18]  F. Pociot,et al.  Suppressor of cytokine signaling 3 (SOCS-3) protects β-cells against interleukin-1β- and interferon-γ-mediated toxicity , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[19]  Pawan Kumar,et al.  Interleukin-18 Induces Rheumatoid Arthritis Synovial Fibroblast CXC Chemokine Production through NFκB Activation , 2001, Laboratory Investigation.

[20]  G. Fantuzzi,et al.  Neutralization of interleukin-18 reduces severity in murine colitis and intestinal IFN-γ and TNF-α production , 2001 .

[21]  S. Swain.  Interleukin 18: tipping the balance towards a T helper cell 1 response. , 2001 .

[22]  A. Hishida,et al.  Attenuation of cisplatin-induced acute renal failure is associated with less apoptotic cell death. , 1999, The Journal of laboratory and clinical medicine.

[23]  J. Megyesi,et al.  Induction of p21WAF1/CIP1/SDI1 in kidney tubule cells affects the course of cisplatin-induced acute renal failure. , 1998, The Journal of clinical investigation.

[24]  M. Su,et al.  Activation of Interferon-γ Inducing Factor Mediated by Interleukin-1β Converting Enzyme , 1997, Science.

[25]  P. Tipping,et al.  P‐selectin directs T lymphocyte‐mediated injury in delayed‐type hypersensitivity responses: studies in glomerulonephritis and cutaneous delayed‐type hypersensitivity , 1996, European journal of immunology.

[26]  C. Manthey,et al.  Targeting renal macrophage accumulation via c-fms kinase reduces tubular apoptosis but fails to modify progressive fibrosis in the obstructed rat kidney. , 2009, American journal of physiology. Renal physiology.

[27]  P. Baeuerle,et al.  Function and activation of NF-kappa B in the immune system. , 1994, Annual review of immunology.

[28]  J. Derry,et al.  Inflammation and Inflammatory Bowel Disease , 2022 .