Phospho-Akt pathway activation and inhibition depends on N-cadherin or phospho-EGFR expression in invasive human bladder cancer cell lines.

[1]  M. Loda,et al.  Identification and Prognostic Significance of an Epithelial-Mesenchymal Transition Expression Profile in Human Bladder Tumors , 2007, Clinical Cancer Research.

[2]  H. Wallerand,et al.  Gefitinib Inhibits the Growth and Invasion of Urothelial Carcinoma Cell Lines in which Akt and MAPK Activation Is Dependent on Constitutive Epidermal Growth Factor Receptor Activation , 2006, Clinical Cancer Research.

[3]  H. Wallerand,et al.  N-Cadherin as a Novel Prognostic Marker of Progression in Superficial Urothelial Tumors , 2006, Clinical Cancer Research.

[4]  R. Weinberg,et al.  Exploring a new twist on tumor metastasis. , 2006, Cancer research.

[5]  A. Toker,et al.  Akt signaling and cancer: surviving but not moving on. , 2006, Cancer research.

[6]  J. Minna,et al.  Restoring E-cadherin expression increases sensitivity to epidermal growth factor receptor inhibitors in lung cancer cell lines. , 2006, Cancer research.

[7]  I. Lascombe,et al.  Expression of E-Cadherin and α-, β-, γ-Catenins in Patients With Bladder Cancer Identification of γ-Catenin as a New Prognostic Marker of Neoplastic Progression in T1 Superficial Urothelial Tumors , 2006 .

[8]  J. Brugge,et al.  Distinct roles of Akt1 and Akt2 in regulating cell migration and epithelial–mesenchymal transition , 2005, The Journal of cell biology.

[9]  Avri Ben-Ze'ev,et al.  Novel expression of N-cadherin elicits in vitro bladder cell invasion via the Akt signaling pathway , 2004, Oncogene.

[10]  R. Hazan,et al.  Cadherin Switch in Tumor Progression , 2004, Annals of the New York Academy of Sciences.

[11]  S. Groshen,et al.  Radical cystectomy in the treatment of invasive bladder cancer: long-term results in 1,054 patients. , 2001, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.

[12]  K. Rieger-Christ,et al.  Expression of classic cadherins type I in urothelial neoplastic progression. , 2001, Human pathology.

[13]  C. Abbou,et al.  Low E-cadherin expression in bladder cancer at the transcriptional and protein level provides prognostic information , 2000, British Journal of Cancer.

[14]  Stuart A. Aaronson,et al.  Exogenous Expression of N-Cadherin in Breast Cancer Cells Induces Cell Migration, Invasion, and Metastasis , 2000, The Journal of cell biology.

[15]  M. Namiki,et al.  Dominant role of E-cadherin in the progression of bladder cancer. , 1999, The Journal of urology.

[16]  Gerhard Christofori,et al.  A causal role for E-cadherin in the transition from adenoma to carcinoma , 1998, Nature.

[17]  R. Hazan,et al.  N-cadherin promotes adhesion between invasive breast cancer cells and the stroma. , 1997, Cell adhesion and communication.

[18]  R. Kerbel,et al.  Ha-ras induction of the invasive phenotype results in up-regulation of epidermal growth factor receptors and altered responsiveness to epidermal growth factor in human papillary transitional cell carcinoma cells. , 1991, Cancer research.

[19]  E. Messing,et al.  Growth stimulating activity produced by human bladder cancer cells. , 1984, The Journal of urology.