The stretch responsive microRNA miR‐148a‐3p is a novel repressor of IKBKB, NF‐κB signaling, and inflammatory gene expression in human aortic valve cells
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V. Nizet | G. Hardiman | R. Sásik | A. Hinton | S. Nigam | Vishal Nigam | Thomas F. Gallegos | A. Hollands | S. A. Mohamed | Katrina Carrion | J. Dyo | V. Patel | Emma Leire | C. King
[1] M. Daniels,et al. Architectural Trends in the Human Normal and Bicuspid Aortic Valve Leaflet and Its Relevance to Valve Disease , 2014, Annals of Biomedical Engineering.
[2] Vishal Nigam,et al. Bicuspid Aortic Valves Experience Increased Strain as Compared to Tricuspid Aortic Valves , 2013, World journal for pediatric & congenital heart surgery.
[3] S. Kauppinen,et al. Treatment of HCV infection by targeting microRNA. , 2013, The New England journal of medicine.
[4] Libing Song,et al. miR-486 sustains NF-κB activity by disrupting multiple NF-κB-negative feedback loops , 2012, Cell Research.
[5] S. Verma,et al. miRNA-141 is a novel regulator of BMP-2-mediated calcification in aortic stenosis. , 2012, The Journal of thoracic and cardiovascular surgery.
[6] O. Bukulmez,et al. Endometrial miR-200c is Altered During Transformation into Cancerous States and Targets the Expression of ZEBs, VEGFA, FLT1, IKKβ, KLF9, and FBLN5 , 2012, Reproductive Sciences.
[7] Chris B Schaffer,et al. Cyclic strain anisotropy regulates valvular interstitial cell phenotype and tissue remodeling in three-dimensional culture. , 2012, Acta biomaterialia.
[8] Norbert Gretz,et al. miRWalk - Database: Prediction of possible miRNA binding sites by "walking" the genes of three genomes , 2011, J. Biomed. Informatics.
[9] Robert M Nerem,et al. Discovery of shear- and side-specific mRNAs and miRNAs in human aortic valvular endothelial cells. , 2011, American journal of physiology. Heart and circulatory physiology.
[10] Jun Li,et al. miR-218 inhibits the invasive ability of glioma cells by direct downregulation of IKK-β. , 2010, Biochemical and biophysical research communications.
[11] S. Hirota,et al. Increased interleukin-18 expression in nonrheumatic aortic valve stenosis. , 2010, International journal of cardiology.
[12] D. Srivastava,et al. Altered microRNAs in bicuspid aortic valve: a comparison between stenotic and insufficient valves. , 2010, The Journal of heart valve disease.
[13] Michael A Lopez,et al. Mechanical Stretch Up-regulates MicroRNA-26a and Induces Human Airway Smooth Muscle Hypertrophy by Suppressing Glycogen Synthase Kinase-3β* , 2010, The Journal of Biological Chemistry.
[14] Kathryn E. Smith,et al. Cyclic strain inhibits acute pro-inflammatory gene expression in aortic valve interstitial cells , 2010, Biomechanics and modeling in mechanobiology.
[15] Mark D. Robinson,et al. edgeR: a Bioconductor package for differential expression analysis of digital gene expression data , 2009, Bioinform..
[16] K. Waters,et al. MicroRNA 132 regulates nutritional stress-induced chemokine production through repression of SirT1. , 2009, Molecular endocrinology.
[17] Nectarios Koziris,et al. DIANA-microT web server: elucidating microRNA functions through target prediction , 2009, Nucleic Acids Res..
[18] Cole Trapnell,et al. Ultrafast and memory-efficient alignment of short DNA sequences to the human genome , 2009, Genome Biology.
[19] Ajit P Yoganathan,et al. Elevated cyclic stretch alters matrix remodeling in aortic valve cusps: implications for degenerative aortic valve disease. , 2009, American journal of physiology. Heart and circulatory physiology.
[20] R. Chen,et al. Regulation of IKKβ by miR-199a affects NF-κB activity in ovarian cancer cells , 2008, Oncogene.
[21] Xiaowei Wang. miRDB: a microRNA target prediction and functional annotation database with a wiki interface. , 2008, RNA.
[22] G. Nickenig,et al. Differential profile of the OPG/RANKL/RANK-system in degenerative aortic native and bioprosthetic valves. , 2008, The Journal of heart valve disease.
[23] Michael Kertesz,et al. The role of site accessibility in microRNA target recognition , 2007, Nature Genetics.
[24] Younghee Lee,et al. Enhancement of NF-kappaB expression and activity upon differentiation of human embryonic stem cell line SNUhES3. , 2007, Stem cells and development.
[25] M. Amrani,et al. Role of Human Valve Interstitial Cells in Valve Calcification and Their Response to Atorvastatin , 2006, Circulation.
[26] Y. Suematsu,et al. Age-associated aortic stenosis in apolipoprotein E-deficient mice. , 2005, Journal of the American College of Cardiology.
[27] B. Iung,et al. Extracellular matrix remodelling in human aortic valve disease: the role of matrix metalloproteinases and their tissue inhibitors. , 2005, European heart journal.
[28] Rosario V. Freeman,et al. Spectrum of Calcific Aortic Valve Disease: Pathogenesis, Disease Progression, and Treatment Strategies , 2005, Circulation.
[29] K. Gunsalus,et al. Combinatorial microRNA target predictions , 2005, Nature Genetics.
[30] S. Hagl,et al. Inflammatory regulation of extracellular matrix remodeling in calcific aortic valve stenosis. , 2005, Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology.
[31] C. Burge,et al. Conserved Seed Pairing, Often Flanked by Adenosines, Indicates that Thousands of Human Genes are MicroRNA Targets , 2005, Cell.
[32] Anton J. Enright,et al. Human MicroRNA Targets , 2004, PLoS biology.
[33] M. Bernardo,et al. Differential Inhibition of Membrane Type 3 (MT3)-Matrix Metalloproteinase (MMP) and MT1-MMP by Tissue Inhibitor of Metalloproteinase (TIMP)-2 and TIMP-3 Regulates Pro-MMP-2 Activation* , 2004, Journal of Biological Chemistry.
[34] S. Hagl,et al. Interleukin-1 beta promotes matrix metalloproteinase expression and cell proliferation in calcific aortic valve stenosis. , 2003, Atherosclerosis.
[35] T. Salo,et al. Evidence for an altered balance between matrix metalloproteinase-9 and its inhibitors in calcific aortic stenosis. , 2003, The Annals of thoracic surgery.
[36] R. Levy,et al. Progression of aortic valve stenosis: TGF-beta1 is present in calcified aortic valve cusps and promotes aortic valve interstitial cell calcification via apoptosis. , 2003, The Annals of thoracic surgery.
[37] B. Janerot-Sjöberg,et al. T lymphocyte infiltration in non-rheumatic aortic stenosis: a comparative descriptive study between tricuspid and bicuspid aortic valves , 2002, Heart.
[38] S. Verma,et al. Clinical and Pathophysiological Implications of a Bicuspid Aortic Valve , 2002, Circulation.
[39] Y. Soini,et al. Expression of MMP2, MMP9, MT1‐MMP, TIMP1, and TIMP2 mRNA in valvular lesions of the heart , 2001, The Journal of pathology.
[40] J. Shirani,et al. Matrix metalloproteinase expression in nonrheumatic aortic stenosis. , 2000, Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology.
[41] C. Ward. Clinical significance of the bicuspid aortic valve , 2000, Heart.
[42] C. Brinckerhoff,et al. Nuclear factor kappaB/p50 activates an element in the distal matrix metalloproteinase 1 promoter in interleukin-1beta-stimulated synovial fibroblasts. , 1998, Arthritis and rheumatism.
[43] T. Lüscher,et al. Pulsatile Stretch Stimulates Superoxide Production and Activates Nuclear Factor-κB in Human Coronary Smooth Muscle , 1997 .
[44] E. Mohler,et al. Detection of osteopontin in calcified human aortic valves. , 1997, Arteriosclerosis, thrombosis, and vascular biology.
[45] N. Mori,et al. Transactivation of the interleukin-1alpha promoter by human T-cell leukemia virus type I and type II Tax proteins. , 1996, Blood.
[46] M. Davies,et al. Demographic characteristics of patients undergoing aortic valve replacement for stenosis: relation to valve morphology. , 1996, Heart.
[47] B. Sumpio,et al. Cyclic strain causes heterogeneous induction of transcription factors, AP-1, CRE binding protein and NF-kB, in endothelial cells: species and vascular bed diversity. , 1995, Journal of biomechanics.
[48] M. Ferguson,et al. Osteopontin is expressed in human aortic valvular lesions. , 1995, Circulation.
[49] A. Gown,et al. Characterization of the Early Lesion of ‘Degenerative’ Valvular Aortic Stenosis: Histological and Immunohistochemical Studies , 1994, Circulation.
[50] M. Rosenqvist,et al. Accumulation of T lymphocytes and expression of interleukin-2 receptors in nonrheumatic stenotic aortic valves. , 1994, Journal of the American College of Cardiology.
[51] J. Hiscott,et al. Characterization of a functional NF-kappa B site in the human interleukin 1 beta promoter: evidence for a positive autoregulatory loop , 1993, Molecular and cellular biology.
[52] S. Beppu,et al. Rapidity of progression of aortic stenosis in patients with congenital bicuspid aortic valves. , 1993, The American journal of cardiology.
[53] A. Brasier,et al. Optimized use of the firefly luciferase assay as a reporter gene in mammalian cell lines. , 1989, BioTechniques.
[54] J. Cleveland,et al. Expression of functional Toll-like receptors 2 and 4 in human aortic valve interstitial cells: potential roles in aortic valve inflammation and stenosis. , 2008, American journal of physiology. Cell physiology.
[55] H. S. Jung,et al. Optimized THP-1 differentiation is required for the detection of responses to weak stimuli , 2007, Inflammation Research.
[56] M. Thubrikar,et al. The congenitally bicuspid aortic valve: how does it function? Why does it fail? , 2004, The Annals of thoracic surgery.