TMT-based proteomics profile reveals changes of the entorhinal cortex in a kainic acid model of epilepsy in mice
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Yuanlin Ma | Jie Liu | Fenglin Tang | Yin Yan | Danmei Hu | Zhijuan Zhang | Zhi-juan Zhang
[1] Minoru Kanehisa,et al. KEGG: integrating viruses and cellular organisms , 2020, Nucleic Acids Res..
[2] A. Sickmann,et al. Tandem Mass Tags for Comparative and Discovery Proteomics. , 2021, Methods in molecular biology.
[3] B. Bernhardt,et al. Connectome biomarkers of drug‐resistant epilepsy , 2020, Epilepsia.
[4] E. Buffalo,et al. Anatomy and Function of the Primate Entorhinal Cortex. , 2020, Annual review of vision science.
[5] J. McNamara,et al. TrkB-Shc Signaling Protects against Hippocampal Injury Following Status Epilepticus , 2019, The Journal of Neuroscience.
[6] E. Verdaguer,et al. JNK1 inhibition by Licochalcone A leads to neuronal protection against excitotoxic insults derived of kainic acid , 2017, Neuropharmacology.
[7] Ruth C Lovering,et al. Exploring autophagy with Gene Ontology , 2016, Autophagy.
[8] B. Krammer,et al. NTRK2 (TrkB gene) variants and temporal lobe epilepsy: A genetic association study , 2017, Epilepsy Research.
[9] Joshua E. Elias,et al. Relative Protein Quantification Using Tandem Mass Tag Mass Spectrometry. , 2017, Methods in molecular biology.
[10] Peng Xie,et al. Quantitative proteomics analysis of the liver reveals immune regulation and lipid metabolism dysregulation in a mouse model of depression , 2016, Behavioural Brain Research.
[11] M. Méndez-Armenta,et al. Oxidative Stress Associated with Neuronal Apoptosis in Experimental Models of Epilepsy , 2014, Oxidative medicine and cellular longevity.
[12] G. Sills,et al. Advantages of Repeated Low Dose against Single High Dose of Kainate in C57BL/6J Mouse Model of Status Epilepticus: Behavioral and Electroencephalographic Studies , 2014, PloS one.
[13] S. Legartová,et al. Epigenetic aspects of HP1 exchange kinetics in apoptotic chromatin. , 2013, Biochimie.
[14] R. Libby,et al. BCL2L1 (BCL-X) promotes survival of adult and developing retinal ganglion cells , 2012, Molecular and Cellular Neuroscience.
[15] B. Jeon,et al. Clusterin interaction with Bcl-xL is associated with seizure-induced neuronal death , 2012, Epilepsy Research.
[16] E. Bertram,et al. Temporal lobe epilepsy induces intrinsic alterations in Na channel gating in layer II medial entorhinal cortex neurons , 2011, Neurobiology of Disease.
[17] B. Jeon,et al. Protein kinase Cdelta is associated with 14-3-3 phosphorylation in seizure-induced neuronal death , 2010, Epilepsy Research.
[18] D. Hochstrasser,et al. From relative to absolute quantification of tryptic peptides with tandem mass tags: application to cerebrospinal fluid. , 2010, Chimia.
[19] Natalie L. M. Cappaert,et al. The anatomy of memory: an interactive overview of the parahippocampal–hippocampal network , 2009, Nature Reviews Neuroscience.
[20] Edward H. Bertram,et al. Temporal lobe epilepsy: Where do the seizures really begin? , 2009, Epilepsy & Behavior.
[21] S. Elmore. Apoptosis: A Review of Programmed Cell Death , 2007, Toxicologic pathology.
[22] P. Buckmaster,et al. Hyperexcitability, Interneurons, and Loss of GABAergic Synapses in Entorhinal Cortex in a Model of Temporal Lobe Epilepsy , 2006, The Journal of Neuroscience.
[23] R. Simon,et al. Epilepsy and Apoptosis Pathways , 2005, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[24] N. Gavaldà,et al. Brain‐derived neurotrophic factor prevents changes in Bcl‐2 family members and caspase‐3 activation induced by excitotoxicity in the striatum , 2005, Journal of neurochemistry.
[25] P. Rakic,et al. A critical role of neural-specific JNK3 for ischemic apoptosis , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[26] S. Willis,et al. The Bcl-2-regulated apoptotic pathway , 2003, Journal of Cell Science.
[27] N. Belluardo,et al. Increase in Bcl‐2 phosphorylation and reduced levels of BH3‐only Bcl‐2 family proteins in kainic acid‐mediated neuronal death in the rat brain , 2003, The European journal of neuroscience.
[28] R. Clark,et al. To die or not to die for neurons in ischemia, traumatic brain injury and epilepsy: a review on the stress-activated signaling pathways and apoptotic pathways , 2003, Progress in Neurobiology.
[29] R. Schwarcz,et al. Neurons in Layer III of the Entorhinal Cortex: A Role in Epileptogenesis and Epilepsy? , 2000, Annals of the New York Academy of Sciences.
[30] Teiichi Furuichi,et al. Inositol 1,4,5-Trisphosphate Receptor Type 1 Is a Substrate for Caspase-3 and Is Cleaved during Apoptosis in a Caspase-3-dependent Manner* , 1999, The Journal of Biological Chemistry.
[31] H. Horvitz,et al. Genetic control of programmed cell death in the nematode Caenorhabditis elegans. , 1999, Cancer research.
[32] Y. Lazebnik,et al. Caspases: enemies within. , 1998, Science.
[33] F. Edward Dudek,et al. Recurrent spontaneous motor seizures after repeated low-dose systemic treatment with kainate: assessment of a rat model of temporal lobe epilepsy , 1998, Epilepsy Research.
[34] R. Schwarcz,et al. Neuronal damage after the injection of amino-oxyacetic acid into the rat entorhinal cortex: a silver impregnation study , 1997, Neuroscience.
[35] K. Mikoshiba. The InsP3 receptor and intracellular Ca2+ signaling , 1997, Current Opinion in Neurobiology.
[36] S. Powell,et al. Distinct Neurodevelopmental Patterns of Bcl‐2 and Bcl‐x Expression Are Altered in Glioneuronal Hamartias of the Human Temporal Lobe , 1997, Journal of neuropathology and experimental neurology.
[37] E. Cavalheiro,et al. Developmental aspects of the pilocarpine model of epilepsy , 1996, Epilepsy Research.
[38] R. Schwarcz,et al. Preferential neuronal loss in layer III of the medial entorhinal cortex in rat models of temporal lobe epilepsy , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[39] H. Horvitz,et al. C. elegans cell survival gene ced-9 encodes a functional homolog of the mammalian proto-oncogene bcl-2 , 1994, Cell.
[40] Shai Shaham,et al. The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1β-converting enzyme , 1993, Cell.
[41] M. Berridge. Inositol trisphosphate and calcium signalling , 1993, Nature.
[42] M. Hengartner,et al. Caenorhabditis elegans gene ced-9 protects cells from programmed cell death , 1992, Nature.
[43] Z. Bortolotto,et al. Long‐Term Effects of Pilocarpine in Rats: Structural Damage of the Brain Triggers Kindling and Spontaneous I Recurrent Seizures , 1991, Epilepsia.
[44] H. Horvitz,et al. Genetic control of programmed cell death in the nematode C. elegans , 1986, Cell.