Soluble CLEC-2 is generated independently of ADAM10 and is increased in plasma in acute coronary syndrome: comparison with soluble GPVI

[1]  S. Watson,et al.  Functional significance of the platelet immune receptors GPVI and CLEC-2. , 2019, The Journal of clinical investigation.

[2]  T. You,et al.  Prognostic Significance of Plasma CLEC-2 (C-Type Lectin-Like Receptor 2) in Patients With Acute Ischemic Stroke , 2019, Stroke.

[3]  S. Watson,et al.  Fibrin and D-dimer bind to monomeric GPVI. , 2017, Blood advances.

[4]  K. Suzuki-Inoue,et al.  Physiologic and pathophysiologic roles of interaction between C‐type lectin‐like receptor 2 and podoplanin: partners from in utero to adulthood , 2017, Journal of thrombosis and haemostasis : JTH.

[5]  S. Nomura.  Extracellular vesicles and blood diseases , 2017, International Journal of Hematology.

[6]  J. Carberry,et al.  A-Disintegrin-And-Metalloproteinase (ADAM) 10 Activity on Resting and Activated Platelets. , 2016, Biochemistry.

[7]  Y. Hosaka,et al.  Properties of soluble glycoprotein VI, a potential platelet activation biomarker , 2015, Platelets.

[8]  Y. Ozaki,et al.  Measurement of soluble C-type lectin-like receptor 2 in human plasma , 2015, Platelets.

[9]  S. Watson,et al.  CLEC-2 expression is maintained on activated platelets and on platelet microparticles. , 2014, Blood.

[10]  A. Sudo,et al.  Elevated soluble platelet glycoprotein VI is a useful marker for DVT in postoperative patients treated with edoxaban , 2014, International Journal of Hematology.

[11]  S. Coughlin,et al.  Podoplanin maintains high endothelial venule integrity by interacting with platelet CLEC-2 , 2013, Nature.

[12]  P. Seizer,et al.  Platelets and matrix metalloproteinases , 2013, Thrombosis and Haemostasis.

[13]  Y. Kanda,et al.  Investigation of the freely available easy-to-use software ‘EZR' for medical statistics , 2012, Bone Marrow Transplantation.

[14]  Y. Yatomi,et al.  Platelet Activation Receptor CLEC-2 Regulates Blood/Lymphatic Vessel Separation by Inhibiting Proliferation, Migration, and Tube Formation of Lymphatic Endothelial Cells* , 2012, The Journal of Biological Chemistry.

[15]  C. Ward,et al.  Pathologic shear triggers shedding of vascular receptors: a novel mechanism for down-regulation of platelet glycoprotein VI in stenosed coronary vessels. , 2012, Blood.

[16]  M. Al-Tamimi,et al.  Focusing on plasma glycoprotein VI , 2012, Thrombosis and Haemostasis.

[17]  K. Shimada,et al.  Increased circulating plasma lysophosphatidic acid in patients with acute coronary syndrome. , 2012, Clinica chimica acta; international journal of clinical chemistry.

[18]  René M. Botnar,et al.  Sandwich immunoassay for soluble glycoprotein VI in patients with symptomatic coronary artery disease. , 2011, Clinical chemistry.

[19]  J. Scheller,et al.  Differentially regulated GPVI ectodomain shedding by multiple platelet-expressed proteinases. , 2010, Blood.

[20]  M. Lu,et al.  Platelets regulate lymphatic vascular development through CLEC-2-SLP-76 signaling. , 2010, Blood.

[21]  Li Li,et al.  A mouse knockout library for secreted and transmembrane proteins , 2010, Nature Biotechnology.

[22]  Y. Tomiyama,et al.  Essential in Vivo Roles of the C-type Lectin Receptor CLEC-2 , 2010, The Journal of Biological Chemistry.

[23]  Y. Ozaki,et al.  Novel synthetic collagen fibers, poly(PHG), stimulate platelet aggregation through glycoprotein VI , 2009, FEBS letters.

[24]  M. Grandoch,et al.  Preliminary evidence for a matrix metalloproteinase-2 (MMP-2)-dependent shedding of soluble CD40 ligand (sCD40L) from activated platelets , 2009, Platelets.

[25]  M. Moroi,et al.  Measuring soluble platelet glycoprotein VI in human plasma by ELISA , 2009, Platelets.

[26]  T. Tsuruo,et al.  Platelet aggregation in the formation of tumor metastasis. , 2008, Proceedings of the Japan Academy. Series B, Physical and biological sciences.

[27]  S. Watson,et al.  Renal cells activate the platelet receptor CLEC-2 through podoplanin. , 2008, The Biochemical journal.

[28]  M. Moroi,et al.  Snake venom metalloproteinases, crotarhagin and alborhagin, induce ectodomain shedding of the platelet collagen receptor, glycoprotein VI , 2007, Thrombosis and Haemostasis.

[29]  K. Mishima,et al.  Involvement of the Snake Toxin Receptor CLEC-2, in Podoplanin-mediated Platelet Activation, by Cancer Cells*♦ , 2007, Journal of Biological Chemistry.

[30]  M. Berndt,et al.  Controlled shedding of platelet glycoprotein (GP)VI and GPIb–IX–V by ADAM family metalloproteinases , 2007, Journal of thrombosis and haemostasis : JTH.

[31]  Gemma L. J. Fuller,et al.  DC-SIGN and CLEC-2 Mediate Human Immunodeficiency Virus Type 1 Capture by Platelets , 2006, Journal of Virology.

[32]  Yukio Ozaki,et al.  Laminin stimulates spreading of platelets through integrin α6β1–dependent activation of GPVI , 2006 .

[33]  Gemma L. J. Fuller,et al.  A novel Syk-dependent mechanism of platelet activation by the C-type lectin receptor CLEC-2. , 2006, Blood.

[34]  M. Miyasaka,et al.  Lymphocyte trafficking across high endothelial venules: dogmas and enigmas , 2004, Nature Reviews Immunology.

[35]  S. Watson,et al.  Platelet-collagen interaction: is GPVI the central receptor? , 2003, Blood.

[36]  David Gurney,et al.  A reliable plasma marker of platelet activation: Does it exist? , 2002, American journal of hematology.

[37]  H. Takayama,et al.  Physical and Functional Association of the Src Family Kinases Fyn and Lyn with the Collagen Receptor Glycoprotein Vi-fc Receptor ␥ Chain Complex on Human Platelets , 1998 .