Effect of nutritional counseling and long term isomaltulose based liquid formula (MHN-01) intake on metabolic syndrome
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H. Maegawa | H. Yamanaka-Okumura | E. Takeda | Y. Taketani | N. Inagaki | H. Sasaki | A. Kawashima | M. Hosokawa | K. Kondo | T. Matsubara | E. Kawasaki | T. Katayama | Y. Fujinaka | K. Shide | H. Aonuma | Shoko Shinozaki | Keiko Kondo
[1] E. Ortega,et al. Inverse association between serum phospholipid oleic acid and insulin resistance in subjects with primary dyslipidaemia. , 2011, Clinical nutrition.
[2] M. Martínez-González,et al. The role of diet in the prevention of type 2 diabetes. , 2011, Nutrition, metabolism, and cardiovascular diseases : NMCD.
[3] S. Kashyap,et al. Randomized trial on the effects of a 7-d low-glycemic diet and exercise intervention on insulin resistance in older obese humans. , 2009, The American journal of clinical nutrition.
[4] A. Avenell,et al. Systematic Review of Long-term Lifestyle Interventions to Prevent Weight Gain and Morbidity in Adults. Authors: , 2009 .
[5] T. Doi,et al. Improvement of Glucose Metabolism in Patients with Impaired Glucose Tolerance or Diabetes by Long-Term Administration of a Palatinose-Based Liquid Formula as a Part of Breakfast , 2009, Journal of clinical biochemistry and nutrition.
[6] Elizabeth J Elliott,et al. Low glycaemic index, or low glycaemic load, diets for diabetes mellitus. , 2009, The Cochrane database of systematic reviews.
[7] S. López,et al. Distinctive postprandial modulation of beta cell function and insulin sensitivity by dietary fats: monounsaturated compared with saturated fatty acids. , 2008, The American journal of clinical nutrition.
[8] E. Takeda,et al. Palatinose and oleic acid act together to prevent pancreatic islet disruption in nondiabetic obese Zucker rats. , 2008, The journal of medical investigation : JMI.
[9] Jing-ping Zhao,et al. Lifestyle intervention and metformin for treatment of antipsychotic-induced weight gain: a randomized controlled trial. , 2008, JAMA.
[10] T. Doi,et al. Dietary palatinose and oleic acid ameliorate disorders of glucose and lipid metabolism in Zucker fatty rats. , 2007, The Journal of nutrition.
[11] K. Chad,et al. Effects of exercise and nutritional counseling in women with polycystic ovary syndrome. , 2006, Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme.
[12] F. Tinahones,et al. Dietary fatty acids and insulin secretion: a population-based study , 2006, European Journal of Clinical Nutrition.
[13] T. Doi,et al. Effect of a novel palatinose-based liquid balanced formula (MHN-01) on glucose and lipid metabolism in male Sprague-Dawley rats after short- and long-term ingestion. , 2004, Metabolism: clinical and experimental.
[14] Merlin C. Thomas,et al. Improved islet morphology after blockade of the renin- angiotensin system in the ZDF rat. , 2004, Diabetes.
[15] T. Nakagami,et al. Hyperglycaemia and mortality from all causes and from cardiovascular disease in five populations of Asian origin , 2004, Diabetologia.
[16] P. Wilson,et al. Carbohydrate nutrition, insulin resistance, and the prevalence of the metabolic syndrome in the Framingham Offspring Cohort. , 2004, Diabetes care.
[17] Markolf Hanefeld,et al. Acarbose treatment and the risk of cardiovascular disease and hypertension in patients with impaired glucose tolerance: the STOP-NIDDM trial. , 2003, JAMA.
[18] C. Cobelli,et al. Effects of dietary macronutrient content on glucose metabolism in children. , 2002, The Journal of clinical endocrinology and metabolism.
[19] D. Ludwig,et al. The glycemic index: physiological mechanisms relating to obesity, diabetes, and cardiovascular disease. , 2002, JAMA.
[20] S. Schwartz,et al. Therapy focused on lowering postprandial glucose, not fasting glucose, may be superior for lowering HbA1c. IOEZ Study Group. , 2000, Diabetes care.
[21] A Sekikawa,et al. Impaired glucose tolerance is a risk factor for cardiovascular disease, but not impaired fasting glucose. The Funagata Diabetes Study. , 1999, Diabetes care.
[22] A. Avignon,et al. Nonfasting Plasma Glucose is a Better Marker of Diabetic Control Than Fasting Plasma Glucose in Type 2 Diabetes , 1997, Diabetes Care.
[23] G. Boden,et al. Evidence for a Circadian Rhythm of Insulin Sensitivity in Patients With NIDDM Caused by Cyclic Changes in Hepatic Glucose Production , 1996, Diabetes.
[24] Y. Okuda,et al. Usefulness of Palatinose as a Caloric Sweetener for Diabetic Patients , 1989, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.
[25] Y. Okuda,et al. Effects of parenteral palatinose on glucose metabolism in normal and streptozotocin diabetic rats. , 1986, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.
[26] G. Bolli,et al. The "dawn phenomenon"--a common occurrence in both non-insulin-dependent and insulin-dependent diabetes mellitus. , 1984, The New England journal of medicine.
[27] T. Wolever,et al. Glycemic index of foods: a physiological basis for carbohydrate exchange. , 1981, The American journal of clinical nutrition.
[28] T. Doi,et al. Effects of a palatinose-based liquid diet (Inslow) on glycemic control and the second-meal effect in healthy men. , 2007, Metabolism: clinical and experimental.
[29] T. Md. Hyperglycaemia and mortality from all causes and from cardiovascular disease in five populations of Asian origin , 2004, Diabetologia.
[30] E. Feskens,et al. Glucose tolerance and cardiovascular mortality: comparison of fasting and 2-hour diagnostic criteria. , 2001, Archives of internal medicine.