TIPARP is involved in the regulation of intraocular pressure
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Yuhong Chen | Y. Lei | Y. Bi | M. Song | Youjia Zhang | Xinghuai Sun
[1] D. M. Peters,et al. Pathogenesis of glaucoma: Extracellular matrix dysfunction in the trabecular meshwork‐A review , 2022, Clinical & experimental ophthalmology.
[2] Tim J. Wigle,et al. PARP7 negatively regulates the type I interferon response in cancer cells and its inhibition triggers antitumor immunity. , 2021, Cancer cell.
[3] D. Grant,et al. 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD)-Inducible Poly-ADP-Ribose Polymerase (TIPARP/PARP7) Catalytic Mutant Mice (TiparpH532A) Exhibit Increased Sensitivity to TCDD-Induced Hepatotoxicity and Lethality , 2021, Toxicological sciences : an official journal of the Society of Toxicology.
[4] W. Kraus,et al. MARTs and MARylation in the Cytosol: Biological Functions, Mechanisms of Action, and Therapeutic Potential , 2021, Cells.
[5] J. Vranka,et al. Normal and glaucomatous outflow regulation , 2020, Progress in Retinal and Eye Research.
[6] Ji Cao,et al. TiPARP forms nuclear condensates to degrade HIF-1α and suppress tumorigenesis , 2020, Proceedings of the National Academy of Sciences.
[7] Xinghuai Sun,et al. ABCA1 Regulates IOP by Modulating Cav1/eNOS/NO Signaling Pathway , 2020, Investigative ophthalmology & visual science.
[8] Jingyi Zhu,et al. Rottlerin acts as a therapeutic in primary open-angle glaucoma by targeting the trabecular meshwork via activation of Rap1 signaling. , 2020, Pharmacological research.
[9] A. Clark,et al. Cross-linked actin networks (CLANs) in glaucoma. , 2017, Experimental eye research.
[10] A. Clark,et al. The many faces of the trabecular meshwork cell , 2017, Experimental eye research.
[11] S. Akira,et al. Mitochondrial damage elicits a TCDD-inducible poly(ADP-ribose) polymerase-mediated antiviral response , 2017, Proceedings of the National Academy of Sciences.
[12] J. Matthews. AHR toxicity and signaling: Role of TIPARP and ADP-ribosylation , 2017 .
[13] C. Toris,et al. The exit strategy: Pharmacological modulation of extracellular matrix production and deposition for better aqueous humor drainage. , 2016, European journal of pharmacology.
[14] G. Damonte,et al. From DNA damage to functional changes of the trabecular meshwork in aging and glaucoma , 2016, Ageing Research Reviews.
[15] Xinghuai Sun,et al. eNOS Activity in CAV1 Knockout Mouse Eyes. , 2016, Investigative ophthalmology & visual science.
[16] A. Takada,et al. Constitutive aryl hydrocarbon receptor signaling constrains type I interferon–mediated antiviral innate defense , 2016, Nature Immunology.
[17] J. Vranka,et al. Extracellular matrix in the trabecular meshwork: intraocular pressure regulation and dysregulation in glaucoma. , 2015, Experimental eye research.
[18] Steven L Salzberg,et al. HISAT: a fast spliced aligner with low memory requirements , 2015, Nature Methods.
[19] E. Poeschla,et al. RhoA GTPase-induced ocular hypertension in a rodent model is associated with increased fibrogenic activity in the trabecular meshwork. , 2015, The American journal of pathology.
[20] W. Huber,et al. Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2 , 2014, Genome Biology.
[21] T. Wong,et al. Common variants near ABCA1 and in PMM2 are associated with primary open-angle glaucoma , 2014, Nature Genetics.
[22] Paul Theodor Pyl,et al. HTSeq—a Python framework to work with high-throughput sequencing data , 2014, bioRxiv.
[23] R. Maddala,et al. Regulation of Plasticity and Fibrogenic Activity of Trabecular Meshwork Cells by Rho GTPase Signaling , 2014, Journal of cellular physiology.
[24] Tin Aung,et al. Genome-wide association analyses identify multiple loci associated with central corneal thickness and keratoconus , 2013, Nature Genetics.
[25] J. McPherson,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin poly(ADP-ribose) polymerase (TiPARP, ARTD14) is a mono-ADP-ribosyltransferase and repressor of aryl hydrocarbon receptor transactivation , 2012, Nucleic acids research.
[26] A. Bosserhoff,et al. Connective tissue growth factor causes glaucoma by modifying the actin cytoskeleton of the trabecular meshwork. , 2012, The American journal of pathology.
[27] C. O'brien,et al. Hypoxia regulated gene transcription in human optic nerve lamina cribrosa cells in culture. , 2012, Investigative ophthalmology & visual science.
[28] Cole Trapnell,et al. Improving RNA-Seq expression estimates by correcting for fragment bias , 2011, Genome Biology.
[29] Cole Trapnell,et al. Role of Rodent Secondary Motor Cortex in Value-based Action Selection Nih Public Access Author Manuscript , 2006 .
[30] P. Kaufman,et al. Regulation of cross-linked actin network (CLAN) formation in human trabecular meshwork (HTM) cells by convergence of distinct beta1 and beta3 integrin pathways. , 2009, Investigative ophthalmology & visual science.
[31] Yoshihiro Yamanishi,et al. KEGG for linking genomes to life and the environment , 2007, Nucleic Acids Res..
[32] H. Uusitalo,et al. Matrix metalloproteinases and their inhibitors in the chamber angle of normal eyes and patients with primary open-angle glaucoma and exfoliation glaucoma , 2007, Graefe's Archive for Clinical and Experimental Ophthalmology.
[33] P. Kaufman,et al. Changes in aqueous humor dynamics with age and glaucoma , 2005, Progress in Retinal and Eye Research.
[34] G. Naumann,et al. Matrix metalloproteinases and their inhibitors in aqueous humor of patients with pseudoexfoliation syndrome/glaucoma and primary open-angle glaucoma. , 2003, Investigative ophthalmology & visual science.
[35] Q. Ma,et al. TCDD-inducible poly(ADP-ribose) polymerase: a novel response to 2,3,7,8-tetrachlorodibenzo-p-dioxin. , 2001, Biochemical and biophysical research communications.
[36] E. Tamm,et al. Transforming Growth Factor-β1 Induces α-Smooth Muscle-Actin Expression in Cultured Human and Monkey Trabecular Meshwork , 1996 .