Clinical utility of next generation sequencing to detect IGH/IL3 rearrangements [t(5;14)(q31.1;q32.1)] in B-lymphoblastic leukemia/lymphoma.
暂无分享,去创建一个
J. Smadbeck | G. Vasmatzis | R. Ketterling | N. Hoppman | H. Olteanu | P. Greipp | L. Baughn | Jess F Peterson | Xinjie Xu | Kathryn E. Pearce | A. Guenzel | Crystal L Golden | C. Williamson | Jonna C Benevides Demasi | Jess F. Peterson
[1] Sarah H. Johnson,et al. Detection of cryptic CCND1 rearrangements in mantle cell lymphoma by next generation sequencing. , 2020, Annals of diagnostic pathology.
[2] E. Clappier,et al. B-ALL With t(5;14)(q31;q32); IGH-IL3 Rearrangement and Eosinophilia: A Comprehensive Analysis of a Peculiar IGH-Rearranged B-ALL , 2019, Front. Oncol..
[3] P. L. Bergsagel,et al. Mate pair sequencing outperforms fluorescence in situ hybridization in the genomic characterization of multiple myeloma , 2019, Blood Cancer Journal.
[4] Y. Ning,et al. Characterization of TCF3 rearrangements in pediatric B-lymphoblastic leukemia/lymphoma by mate-pair sequencing (MPseq) identifies complex genomic rearrangements and a novel TCF3/TEF gene fusion , 2019, Blood Cancer Journal.
[5] J. Smadbeck,et al. Elucidating a false-negative MYC break-apart fluorescence in situ hybridization probe study by next-generation sequencing in a patient with high-grade B-cell lymphoma with IGH/MYC and IGH/BCL2 rearrangements , 2019, Cold Spring Harbor molecular case studies.
[6] A. Nordgren,et al. High-resolution detection of chromosomal rearrangements in leukemias through mate pair whole genome sequencing , 2018, PloS one.
[7] A. Reiter,et al. Myeloid neoplasms with eosinophilia. , 2017, Blood.
[8] G. Wertheim,et al. Marked eosinophilia masking B lymphoblastic leukemia , 2016, American journal of hematology.
[9] K. Wood,et al. Cutaneous B‐lymphoblastic lymphoma with IL3/IgH translocation presenting with hypereosinophilia and acute endocarditis , 2015, Pediatric blood & cancer.
[10] R. Wade,et al. IGH@ translocations are prevalent in teenagers and young adults with acute lymphoblastic leukemia and are associated with a poor outcome. , 2014, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[11] N. Douet-Guilbert,et al. Molecular cytogenetics of IGH rearrangements in non-Hodgkin B-cell lymphoma , 2007, Cytogenetic and Genome Research.
[12] Takashi Akasaka,et al. Five members of the CEBP transcription factor family are targeted by recurrent IGH translocations in B-cell precursor acute lymphoblastic leukemia (BCP-ALL). , 2007, Blood.
[13] C. Willman,et al. Activation of the interleukin-3 gene by chromosome translocation in acute lymphocytic leukemia with eosinophilia. , 1990, Blood.
[14] T. Meeker,et al. The t(5;14) chromosomal translocation in a case of acute lymphocytic leukemia joins the interleukin-3 gene to the immunoglobulin heavy chain gene. , 1989, Blood.
[15] T. Hogan,et al. Acute lymphoblastic leukemia with chromosomal 5;14 translocation and hypereosinophilia: case report and literature review. , 1987, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[16] E. A. Kelly,et al. Essential Mechanisms of Differential Activation of Eosinophils by IL-3 Compared to GM-CSF and IL-5. , 2016, Critical reviews in immunology.
[17] Y. Sato,et al. Hypereosinophilic syndrome in acute lymphoblastic leukemia with a chromosome translocation [t(5q;14q)]. , 1984, Medical and pediatric oncology.