Tumorigenesis Suppressor Pdcd4 Down-Regulates Mitogen-Activated Protein Kinase Kinase Kinase Kinase 1 Expression To Suppress Colon Carcinoma Cell Invasion
暂无分享,去创建一个
N. Colburn | T. Tan | Qing Wang | Nancy H. Colburn | Hsin-Sheng Yang | Connie P. Matthews | Timothy Clair | Alyson R. Baker | Chou-Chi H. Li | Tse-Hua Tan | Hsin-Sheng Yang | C. P. Matthews | T. Clair | Qing Wang | Chou-chi Li | H. Yang
[1] C. Proud. Turned on by insulin , 1994, Nature.
[2] C. Proud. Translation. Turned on by insulin. , 1994, Nature.
[3] B. Ozanne,et al. Fos-transformation activates genes associated with invasion. , 1994, Oncogene.
[4] T. Honjo,et al. Isolation of a novel mouse gene MA-3 that is induced upon programmed cell death. , 1995, Gene.
[5] L. Matrisian,et al. Mechanisms controlling the transcription of matrix metalloproteinase genes in normal and neoplastic cells. , 1996, Enzyme & protein.
[6] T. Pawson,et al. HPK1, a hematopoietic protein kinase activating the SAPK/JNK pathway. , 1996, The EMBO journal.
[7] M. C. Hu,et al. Human HPK1, a novel human hematopoietic progenitor kinase that activates the JNK/SAPK kinase cascade. , 1996, Genes & development.
[8] H. Kizaki,et al. Molecular cloning of the genes suppressed in RVC lymphoma cells by topoisomerase inhibitors. , 1996, Biochemical and biophysical research communications.
[9] S. Schreiber,et al. A Signaling Pathway to Translational Control , 1996, Cell.
[10] B. Ozanne,et al. AP-1-mediated invasion requires increased expression of the hyaluronan receptor CD44 , 1997, Molecular and cellular biology.
[11] G. Stamp,et al. Imbalance of expression of matrix metalloproteinases (MMPs) and tissue inhibitors of the matrix metalloproteinases (TIMPs) in human pancreatic carcinoma , 1997, The Journal of pathology.
[12] William C. Parks,et al. The Activity of Collagenase-1 Is Required for Keratinocyte Migration on a Type I Collagen Matrix , 1997, The Journal of cell biology.
[13] C. Brinckerhoff,et al. The AP-1 site and MMP gene regulation: what is all the fuss about? , 1997, Matrix biology : journal of the International Society for Matrix Biology.
[14] D. Taub,et al. A dominant negative mutant of jun blocking 12‐O‐tetradecanoylphorbol‐13‐acetate–induced invasion in mouse keratinocytes , 1997, Molecular carcinogenesis.
[15] E. Goldsmith,et al. A constitutively active and nuclear form of the MAP kinase ERK2 is sufficient for neurite outgrowth and cell transformation , 1998, Current Biology.
[16] S. Yuspa,et al. The pathogenesis of squamous cell cancer: lessons learned from studies of skin carcinogenesis. , 1998, Journal of dermatological science.
[17] L. Azzoni,et al. Differential transcriptional regulation of CD161 and a novel gene, 197/15a, by IL-2, IL-15, and IL-12 in NK and T cells. , 1998, Journal of immunology.
[18] N. Colburn,et al. Differentially expressed protein Pdcd4 inhibits tumor promoter-induced neoplastic transformation. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[19] Hiroyuki Ogata,et al. KEGG: Kyoto Encyclopedia of Genes and Genomes , 1999, Nucleic Acids Res..
[20] T. Tan,et al. Hematopoietic Progenitor Kinase 1 Is a Component of Transforming Growth Factor β-induced c-Jun N-terminal Kinase Signaling Cascade* , 1999, The Journal of Biological Chemistry.
[21] A. Albini,et al. c-fos-induced growth factor/vascular endothelial growth factor D induces angiogenesis in vivo and in vitro. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[22] A. De Benedetti,et al. eIF4E expression in tumors: its possible role in progression of malignancies. , 1999, The international journal of biochemistry & cell biology.
[23] S. Feller,et al. Interaction of Hematopoietic Progenitor Kinase 1 with Adapter Proteins Crk and CrkL Leads to Synergistic Activation of c-Jun N-Terminal Kinase , 1999, Molecular and Cellular Biology.
[24] J. Winnie,et al. Transcriptional regulation of cell invasion: AP-1 regulation of a multigenic invasion programme. , 2000, European journal of cancer.
[25] T. Kislinger,et al. Blockade of RAGE–amphoterin signalling suppresses tumour growth and metastases , 2000, Nature.
[26] Susumu Goto,et al. KEGG: Kyoto Encyclopedia of Genes and Genomes , 2000, Nucleic Acids Res..
[27] S. Curran,et al. Matrix metalloproteinases: molecular aspects of their roles in tumour invasion and metastasis. , 2000, European journal of cancer.
[28] M. Cobb,et al. HPK1 is activated by lymphocyte antigen receptors and negatively regulates AP-1. , 2000, Immunity.
[29] Arthur Weiss,et al. Hematopoietic Progenitor Kinase 1 Associates Physically and Functionally with the Adaptor Proteins B Cell Linker Protein and SLP-76 in Lymphocytes* , 2001, The Journal of Biological Chemistry.
[30] P. Ling,et al. Leukocyte-specific adaptor protein Grap2 interacts with hematopoietic progenitor kinase 1 (HPK1) to activate JNK signaling pathway in T lymphocytes , 2001, Oncogene.
[31] R. Reeves,et al. Architectural Transcription Factor HMGI(Y) Promotes Tumor Progression and Mesenchymal Transition of Human Epithelial Cells , 2001, Molecular and Cellular Biology.
[32] M. C. Hu,et al. Involvement of Hematopoietic Progenitor Kinase 1 in T Cell Receptor Signaling* , 2001, The Journal of Biological Chemistry.
[33] N. Colburn,et al. A novel transformation suppressor, Pdcd4, inhibits AP-1 transactivation but not NF-κB or ODC transactivation , 2001, Oncogene.
[34] E. Shin,et al. c-Jun N-terminal kinase is involved in motility of endothelial cell , 2001, Experimental & Molecular Medicine.
[35] R. DuBois,et al. Detection of differentially expressed genes in human colon carcinoma cells treated with a selective COX-2 inhibitor , 2001, Oncogene.
[36] D. Boyd,et al. An inhibitor of c-jun aminoterminal kinase (SP600125) represses c-Jun activation, DNA-binding and PMA-inducible 92-kDa type IV collagenase expression. , 2002, Biochimica et biophysica acta.
[37] R. Carmody,et al. DUG is a novel homologue of translation initiation factor 4G that binds eIF4A. , 2002, Biochemical and biophysical research communications.
[38] W. Park,et al. Up-regulation of PDCD4 in senescent human diploid fibroblasts. , 2002, Biochemical and biophysical research communications.
[39] K. Anderson,et al. Identification of genes regulated by Dexamethasone in multiple myeloma cells using oligonucleotide arrays , 2002, Oncogene.
[40] N. Sonenberg,et al. The Transformation Suppressor Pdcd4 Is a Novel Eukaryotic Translation Initiation Factor 4A Binding Protein That Inhibits Translation , 2003, Molecular and Cellular Biology.
[41] Jennifer L. Knies,et al. Pdcd4 suppresses tumor phenotype in JB6 cells by inhibiting AP-1 transactivation , 2003, Oncogene.
[42] P. O'dwyer,et al. Role of Jun and Jun kinase in resistance of cancer cells to therapy. , 2003, Drug resistance updates : reviews and commentaries in antimicrobial and anticancer chemotherapy.
[43] Iver Petersen,et al. Loss of PDCD4 expression in human lung cancer correlates with tumour progression and prognosis , 2003, The Journal of pathology.
[44] N. Colburn,et al. Expression of dominant negative c-jun inhibits ultraviolet B-induced squamous cell carcinoma number and size in an SKH-1 hairless mouse model. , 2003, Molecular cancer research : MCR.
[45] Nahum Sonenberg,et al. A Novel Function of the MA-3 Domains in Transformation and Translation Suppressor Pdcd4 Is Essential for Its Binding to Eukaryotic Translation Initiation Factor 4A , 2004, Molecular and Cellular Biology.
[46] H. Samuels,et al. Induction of PDCD4 tumor suppressor gene expression by RAR agonists, antiestrogen and HER-2/neu antagonist in breast cancer cells. Evidence for a role in apoptosis , 2004, Oncogene.
[47] N. Colburn,et al. Characterization of programmed cell death 4 in multiple human cancers reveals a novel enhancer of drug sensitivity. , 2004, Molecular cancer therapeutics.
[48] K. Klempnauer,et al. Transformation suppressor protein Pdcd4 interferes with JNK-mediated phosphorylation of c-Jun and recruitment of the coactivator p300 by c-Jun , 2004, Oncogene.
[49] A. Hasilik,et al. Pdcd4 inhibits growth of tumor cells by suppression of carbonic anhydrase type II , 2004, Molecular and Cellular Endocrinology.
[50] R. Arnold,et al. Programmed Cell Death Protein 4 (PDCD4) Acts As a Tumor Suppressor in Neuroendocrine Tumor Cells , 2004, Annals of the New York Academy of Sciences.
[51] N. Colburn,et al. Dominant-negative c-Jun (TAM67) target genes: HMGA1 is required for tumor promoter-induced transformation , 2004, Oncogene.
[52] N. Colburn,et al. Epidermal expression of the translation inhibitor programmed cell death 4 suppresses tumorigenesis. , 2005, Cancer research.
[53] T. Tan,et al. Functional interactions of HPK1 with adaptor proteins , 2005, Journal of cellular biochemistry.
[54] A. Wlodawer,et al. Mutational analysis of the DEAD-box RNA helicase eIF4AII characterizes its interaction with transformation suppressor Pdcd4 and eIF4GI. , 2005, RNA.