The role of TGF-β and myofibroblasts in the arteritis of Kawasaki disease.

[1]  Michael Levin,et al.  Genome-wide linkage and association mapping identify susceptibility alleles in ABCC4 for Kawasaki disease , 2011, Journal of Medical Genetics.

[2]  Samarjit Patnaik,et al.  Noncanonical TGFβ Signaling Contributes to Aortic Aneurysm Progression in Marfan Syndrome Mice , 2011, Science.

[3]  Dean Y. Li,et al.  Inhibition of transforming growth factor β worsens elastin degradation in a murine model of Kawasaki disease. , 2011, The American journal of pathology.

[4]  Scott Mellis,et al.  Transforming Growth Factor-&bgr; Signaling Pathway in Patients With Kawasaki Disease , 2011, Circulation. Cardiovascular genetics.

[5]  A. Desmoulière,et al.  The Myofibroblast: one function, multiple origins , 2010 .

[6]  A. Cavani,et al.  IL-17 and IL-22: siblings, not twins. , 2010, Trends in immunology.

[7]  K. Nishigaki,et al.  Guidelines for Diagnosis and Management of Cardiovascular Sequelae in Kawasaki Disease (JCS 2008) : Digest Version , 2010 .

[8]  Xiao-Fan Wang,et al.  Smad3 Signaling Critically Regulates Fibroblast Phenotype and Function in Healing Myocardial Infarction , 2010, Circulation research.

[9]  J. Burns,et al.  Memory T-cells and characterization of peripheral T-cell clones in acute Kawasaki disease , 2010, Autoimmunity.

[10]  T. Huizinga,et al.  De Novo Generation and Enhanced Suppression of Human CD4+CD25+ Regulatory T Cells by Retinoic Acid1 , 2009, The Journal of Immunology.

[11]  Raghu Kalluri,et al.  The basics of epithelial-mesenchymal transition. , 2009, The Journal of clinical investigation.

[12]  Liwu Li,et al.  Differential Regulation of Foxp3 and IL-17 Expression in CD4 T Helper Cells by IRAK-11 , 2009, The Journal of Immunology.

[13]  J. Hulot,et al.  Multidrug resistance-associated protein 4 regulates cAMP-dependent signaling pathways and controls human and rat SMC proliferation. , 2008, The Journal of clinical investigation.

[14]  J. Newburger,et al.  Resistance to intravenous immunoglobulin in children with Kawasaki disease. , 2008, The Journal of pediatrics.

[15]  W. Daniel,et al.  Activated myeloid dendritic cells accumulate and co-localize with CD3+ T cells in coronary artery lesions in patients with Kawasaki disease. , 2007, Experimental and molecular pathology.

[16]  P. Adegboyega,et al.  Subepithelial Myofibroblasts are Novel Nonprofessional APCs in the Human Colonic Mucosa1 , 2006, The Journal of Immunology.

[17]  R. J. Hocking,et al.  TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. , 2006, Immunity.

[18]  Kei Takahashi,et al.  Neutrophilic involvement in the damage to coronary arteries in acute stage of Kawasaki disease , 2005, Pediatrics international : official journal of the Japan Pediatric Society.

[19]  Walter R Wilson,et al.  Diagnosis, Treatment, and Long-Term Management of Kawasaki Disease: A Statement for Health Professionals From the Committee on Rheumatic Fever, Endocarditis and Kawasaki Disease, Council on Cardiovascular Disease in the Young, American Heart Association , 2004, Pediatrics.

[20]  C. Yutani,et al.  Immunohistochemical study of apparently intact coronary artery in a child after Kawasaki disease , 2004, Pediatrics international : official journal of the Japan Pediatric Society.

[21]  J. Burns,et al.  Kawasaki syndrome , 2004, The Lancet.

[22]  T. Oharaseki,et al.  Histopathological features of murine systemic vasculitis caused by Candida albicans extract – an animal model of Kawasaki Disease , 2004, Inflammation Research.

[23]  S. Crawford,et al.  CTLA-4 (CD152) Expression in T Cells during the Acute Stage of Kawasaki Disease , 2003, Pediatric Research.

[24]  E. Camenzind,et al.  Mechanisms of Neointima Formation and Remodeling in the Porcine Coronary Artery , 2001, Circulation.

[25]  D. Powell Water transport revisited , 1999, The Journal of physiology.

[26]  E. Ebert,et al.  Mesenchymal cells stimulate human intestinal intraepithelial lymphocytes. , 1997, Gastroenterology.

[27]  G. Gabbiani,et al.  Differentiation of smooth muscle cells in human blood vessels as defined by smoothelin, a novel marker for the contractile phenotype. , 1997, Arteriosclerosis, thrombosis, and vascular biology.

[28]  M Takahashi,et al.  A single intravenous infusion of gamma globulin as compared with four infusions in the treatment of acute Kawasaki syndrome. , 1991, The New England journal of medicine.

[29]  Y. Kohno,et al.  Class II major histocompatibility antigen expression on coronary arterial endothelium in a patient with Kawasaki disease. , 1990, Human pathology.

[30]  W. Schürch,et al.  Myofibroblasts from diverse pathologic settings are heterogeneous in their content of actin isoforms and intermediate filament proteins. , 1989, Laboratory investigation; a journal of technical methods and pathology.

[31]  J. Stockman,et al.  Noncanonical TGFβ Signaling Contributes to Aortic Aneurysm Progression in Marfan Syndrome Mice , 2012 .

[32]  J. Stockman,et al.  Angiotensin II Blockade and Aortic-Root Dilation in Marfan's Syndrome , 2010 .

[33]  Ying E Zhang,et al.  Non-Smad pathways in TGF-β signaling , 2009, Cell Research.

[34]  Yusuke Nakamura,et al.  ITPKC functional polymorphism associated with Kawasaki disease susceptibility and formation of coronary artery aneurysms , 2008, Nature Genetics.

[35]  E. Silverman,et al.  Superantigenic activity is responsible for induction of coronary arteritis in mice: an animal model of Kawasaki disease. , 2003, International immunology.

[36]  A B West,et al.  Myofibroblasts. I. Paracrine cells important in health and disease. , 1999, The American journal of physiology.

[37]  D. Powell,et al.  Powell, D. W. et al. Myofibroblasts. I. Paracrine cells important in health and disease. Am. J. Physiol. 277, C1-C9 , 1999 .