Free radicals as triggers of brain edema formation after stroke.
暂无分享,去创建一个
Ji Hoe Heo | Seung Koo Lee | J. Heo | Sang Won Han | S. Han
[1] D. Corbett,et al. Efficacy of disodium 4-[(tert-butylimino)methyl]benzene-1,3-disulfonate N-oxide (NXY-059), a free radical trapping agent, in a rat model of hemorrhagic stroke , 2001, Neuropharmacology.
[2] S. Ibayashi,et al. Free radical scavenger, edaravone, in stroke with internal carotid artery occlusion , 2004, Journal of the Neurological Sciences.
[3] J. Koziol,et al. Matrix Metalloproteinases Increase Very Early during Experimental Focal Cerebral Ischemia , 1999, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[4] Carol Cass,et al. Nitric Oxide and Cyclic GMP Increase the Expression of Matrix Metalloproteinase-9 in Vascular Smooth Muscle , 2003, Journal of Pharmacology and Experimental Therapeutics.
[5] F. Barone,et al. Matrix metalloproteinase expression increases after cerebral focal ischemia in rats: inhibition of matrix metalloproteinase-9 reduces infarct size. , 1998, Stroke.
[6] Kyung-Yul Lee,et al. Increase in Plasma Matrix Metalloproteinase-9 in Acute Stroke Patients With Thrombolysis Failure , 2003, Stroke.
[7] D. Lawrence,et al. Tissue-type plasminogen activator induces opening of the blood-brain barrier via the LDL receptor-related protein. , 2003, The Journal of clinical investigation.
[8] P. Bracci,et al. Matrix Metalloproteinase-9 and Myeloperoxidase Expression: Quantitative Analysis by Antigen Immunohistochemistry in a Model of Transient Focal Cerebral Ischemia , 2004, Stroke.
[9] V. Vallyathan,et al. Hydroxyl radical formation is greater in striatal core than in penumbra in a rat model of ischemic stroke , 2003, Journal of neuroscience research.
[10] R. Borchardt,et al. VEGF increases BMEC monolayer permeability by affecting occludin expression and tight junction assembly. , 2001, American journal of physiology. Heart and circulatory physiology.
[11] M. Schroeter,et al. Astrocytes enhance radical defence in capillary endothelial cells constituting the blood‐brain barrier , 1999, FEBS letters.
[12] N. Plesnila,et al. Contribution of Anion Transporters to the Acidosis‐Induced Swelling and Intracellular Acidification of Glial Cells , 2000, Journal of neurochemistry.
[13] E. Lengyel,et al. Stimulation of 92-kDa Gelatinase B Promoter Activity by ras Is Mitogen-activated Protein Kinase Kinase 1-independent and Requires Multiple Transcription Factor Binding Sites Including Closely Spaced PEA3/ets and AP-1 Sequences (*) , 1996, The Journal of Biological Chemistry.
[14] N. van Bruggen,et al. VEGF antagonism reduces edema formation and tissue damage after ischemia/reperfusion injury in the mouse brain. , 1999, The Journal of clinical investigation.
[15] C. Cierniewski,et al. Dual regulatory effects of nitric oxide on plasminogen activator inhibitor type 1 expression in endothelial cells. , 2000, European journal of biochemistry.
[16] K. Scharffetter-Kochanek,et al. Stable Overexpression of Manganese Superoxide Dismutase in Mitochondria Identifies Hydrogen Peroxide as a Major Oxidant in the AP-1-mediated Induction of Matrix-degrading Metalloprotease-1* , 1999, The Journal of Biological Chemistry.
[17] J. Bodmer,et al. Histamine and thrombin modulate endothelial focal adhesion through centripetal and centrifugal forces. , 1996, The Journal of clinical investigation.
[18] M. Goldberg,et al. AMPA/Kainate Receptor Activation Mediates Hypoxic Oligodendrocyte Death and Axonal Injury in Cerebral White Matter , 2001, The Journal of Neuroscience.
[19] K. Welch,et al. Acute tissue response to cerebral ischemia in the gerbil An ultrastructural study , 1977, Journal of the Neurological Sciences.
[20] M. Kornfeld,et al. Collagenase-induced intracerebral hemorrhage in rats. , 1990, Stroke.
[21] P. Chan,et al. The cytosolic antioxidant, copper/zinc superoxide dismutase, attenuates blood–brain barrier disruption and oxidative cellular injury after photothrombotic cortical ischemia in mice , 2001, Neuroscience.
[22] M. Marikovsky,et al. Cu/Zn Superoxide Dismutase Plays Important Role in Immune Response1 , 2003, The Journal of Immunology.
[23] T. Omae,et al. Separating changes in the intra‐ and extracellular water apparent diffusion coefficient following focal cerebral ischemia in the rat brain , 2002, Magnetic resonance in medicine.
[24] G. Schmid-Schönbein,et al. Polymorphonuclear Leukocytes Occlude Capillaries Following Middle Cerebral Artery Occlusion and Reperfusion in Baboons , 1991, Stroke.
[25] C. Granziera,et al. Astrocyte-Specific Expression of Aquaporin-9 in Mouse Brain is Increased after Transient Focal Cerebral Ischemia , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[26] J. Arenillas,et al. Matrix Metalloproteinase-9 Pretreatment Level Predicts Intracranial Hemorrhagic Complications After Thrombolysis in Human Stroke , 2003, Circulation.
[27] T. Kent,et al. Superoxide anion production during reperfusion is reduced by an antineutrophil antibody after prolonged cerebral ischemia. , 1999, Free radical biology & medicine.
[28] P. Chan,et al. Brain injury, edema, and vascular permeability changes induced by oxygen‐derived free radicals , 1984, Neurology.
[29] P. Sandercock,et al. Is Breakdown of the Blood-Brain Barrier Responsible for Lacunar Stroke, Leukoaraiosis, and Dementia? , 2003, Stroke.
[30] G. Rosenberg,et al. Closure of the Blood-Brain Barrier by Matrix Metalloproteinase Inhibition Reduces rtPA-Mediated Mortality in Cerebral Ischemia With Delayed Reperfusion , 2003, Stroke.
[31] T. Hinds,et al. Inhibition of Ca2+-pump ATPase and the Na+/K+-pump ATPase by iron-generated free radicals. Protection by 6,7-dimethyl-2,4-DI-1- pyrrolidinyl-7H-pyrrolo[2,3-d] pyrimidine sulfate (U-89843D), a potent, novel, antioxidant/free radical scavenger. , 1996, Biochemical pharmacology.
[32] G. Rosenberg,et al. Matrix metalloproteinases and TIMPs are associated with blood-brain barrier opening after reperfusion in rat brain. , 1998, Stroke.
[33] W. Hacke,et al. 'Malignant' middle cerebral artery territory infarction : Clinical course and prognostic signs , 1996 .
[34] H. Weiss,et al. Effects of cyclic GMP on microvascular permeability of the cerebral cortex. , 1999, Microvascular research.
[35] G. Hamann,et al. Microvascular basal lamina antigens disappear during cerebral ischemia and reperfusion. , 1995, Stroke.
[36] J. Garcìa,et al. Neuronal necrosis after middle cerebral artery occlusion in Wistar rats progresses at different time intervals in the caudoputamen and the cortex. , 1995, Stroke.
[37] I. Romero,et al. Transendothelial permeability changes induced by free radicals in an in vitro model of the blood-brain barrier. , 1999, Free radical biology & medicine.
[38] M Chopp,et al. VEGF enhances angiogenesis and promotes blood-brain barrier leakage in the ischemic brain. , 2000, The Journal of clinical investigation.
[39] Jiankun Cui,et al. S-Nitrosylation of Matrix Metalloproteinases: Signaling Pathway to Neuronal Cell Death , 2002, Science.
[40] T. Kietzmann,et al. Enhanced plasminogen activator inhibitor-1 expression in transgenic mice with hepatocyte-specific overexpression of superoxide dismutase or glutathione peroxidase. , 2004, Antioxidants & redox signaling.
[41] Ching‐Jen Wang,et al. Ras Induction of Superoxide Activates ERK-dependent Angiogenic Transcription Factor HIF-1α and VEGF-A Expression in Shock Wave-stimulated Osteoblasts* , 2004, Journal of Biological Chemistry.
[42] J. Garcìa,et al. Cerebral white matter is highly vulnerable to ischemia. , 1996, Stroke.
[43] T. Nagafuji,et al. Blockade of nitric oxide formation by Nω-nitro-l-arginine mitigates ischemic brain edema and subsequent cerebral infarction in rats , 1992, Neuroscience Letters.
[44] D. DeLong,et al. Effect of a Novel Free Radical Scavenger, Edaravone (MCI-186), on Acute Brain Infarction , 2003, Cerebrovascular Diseases.
[45] Cornelius Weiller,et al. Prediction of Malignant Middle Cerebral Artery Infarction by Early Perfusion- and Diffusion-Weighted Magnetic Resonance Imaging , 2003, Stroke.
[46] I. Klatzo. Evolution of brain edema concepts. , 1994, Acta neurochirurgica. Supplementum.
[47] K. Arai,et al. Lipoprotein receptor–mediated induction of matrix metalloproteinase by tissue plasminogen activator , 2003, Nature Medicine.
[48] D. Lumenta,et al. Effects of LF 16-0687 Ms, a bradykinin B2 receptor antagonist, on brain edema formation and tissue damage in a rat model of temporary focal cerebral ischemia , 2002, Brain Research.
[49] H. Oh,et al. Lithospermic acid B isolated from Salvia miltiorrhiza ameliorates ischemia/reperfusion-induced renal injury in rats. , 2004, Life sciences.
[50] Hall Ed,et al. Involvement of lipid peroxidation in CNS injury. , 1992 .
[51] Turgay Dalkara,et al. Reperfusion-Induced Oxidative/Nitrative Injury to Neurovascular Unit After Focal Cerebral Ischemia , 2004, Stroke.
[52] C. Iadecola,et al. Time Dependence of Effect of Nitric Oxide Synthase Inhibition on Cerebral Ischemic Damage , 1995, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[53] K. Kugiyama,et al. Lysophosphatidylcholine induces urokinase-type plasminogen activator and its receptor in human macrophages partly through redox-sensitive pathway. , 2000, Arteriosclerosis, thrombosis, and vascular biology.
[54] L. Iversen,et al. Heparin-binding protein (HBP/CAP37): A missing link in neutrophil-evoked alteration of vascular permeability , 2001, Nature Medicine.
[55] D. Choi,et al. Brain tissue responses to ischemia. , 2000, The Journal of clinical investigation.
[56] M. Fini,et al. Effects of Matrix Metalloproteinase-9 Gene Knock-Out on the Proteolysis of Blood–Brain Barrier and White Matter Components after Cerebral Ischemia , 2001, The Journal of Neuroscience.
[57] Ole P. Ottersen,et al. The molecular basis of water transport in the brain , 2003, Nature Reviews Neuroscience.
[58] G. Manley,et al. Aquaporin-4 deletion in mice reduces brain edema after acute water intoxication and ischemic stroke , 2000, Nature Medicine.
[59] S. Holland,et al. The vascular NADPH oxidase subunit p47phox is involved in redox-mediated gene expression. , 2002, Free radical biology & medicine.
[60] U. Ito,et al. CT enhancement after prolonged high-dose contrast infusion in the early stage of cerebral infarction. , 1986, Stroke.
[61] L. Hertz,et al. Peroxide‐scavenging deficit underlies oligodendrocyte susceptibility to oxidative stress , 1998, Glia.
[62] B. Risberg,et al. Reactive oxygen intermediates and ischemia-reperfusion injury release tissue plasminogen activator from isolated rat hearts. , 1993, Thrombosis research.
[63] A. Mazar,et al. Activation Systems for Latent Matrix Metalloproteinase-2 are Upregulated Immediately after Focal Cerebral Ischemia , 2003, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[64] W. Heiss,et al. Extracellular Concentrations of Non–Transmitter Amino Acids in Peri-Infarct Tissue of Patients Predict Malignant Middle Cerebral Artery Infarction , 2003, Stroke.
[65] E. Lo,et al. Reduction of Tissue Plasminogen Activator-Induced Hemorrhage and Brain Injury by Free Radical Spin Trapping after Embolic Focal Cerebral Ischemia in Rats , 2000, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[66] A. Betz. Identification of Hypoxanthine Transport and Xanthine Oxidase Activity in Brain Capillaries , 1985, Journal of neurochemistry.
[67] R. V. Sharma,et al. Role of reactive oxygen species in IL-1 beta-stimulated sustained ERK activation and MMP-9 induction. , 2001, American journal of physiology. Heart and circulatory physiology.
[68] H. Duan,et al. v-Ha-RaS oncogene upregulates the 92-kDa type IV collagenase (MMP-9) gene by increasing cellular superoxide production and activating NF-kappaB. , 2001, Free radical biology & medicine.
[69] M. Rice,et al. Differential compartmentalization of brain ascorbate and glutathione between neurons and glia , 1997, Neuroscience.
[70] U. Förstermann,et al. Nitric Oxide Increases the Decay of Matrix Metalloproteinase 9 mRNA by Inhibiting the Expression of mRNA-Stabilizing Factor HuR , 2003, Molecular and Cellular Biology.
[71] R. Regan,et al. Hemin induces an iron‐dependent, oxidative injury to human neuron‐like cells , 2003, Journal of neuroscience research.
[72] J. Koziol,et al. Rapid Differential Endogenous Plasminogen Activator Expression After Acute Middle Cerebral Artery Occlusion , 2001, Stroke.
[73] O. Kempski,et al. Neuron-glial interaction during injury and edema of the CNS. , 1994, Acta neurochirurgica. Supplementum.
[74] M. Fujimura,et al. Early appearance of activated matrix metalloproteinase-9 and blood–brain barrier disruption in mice after focal cerebral ischemia and reperfusion , 1999, Brain Research.
[75] G. D. del Zoppo,et al. Rapid Loss of Microvascular Integrin Expression during Focal Brain Ischemia Reflects Neuron Injury , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[76] J. Tsuruda,et al. Diffusion-weighted MR imaging of anisotropic water diffusion in cat central nervous system. , 1990, Radiology.
[77] J. Cervós-Navarro,et al. Morphological changes in acute cerebral ischemia after occlusion and reperfusion in the rat. , 1990, Advances in neurology.
[78] S. Warach,et al. Impact of Establishing a Primary Stroke Center at a Community Hospital on the Use of Thrombolytic Therapy: The NINDS Suburban Hospital Stroke Center Experience , 2003, Stroke.
[79] D. Collen,et al. Oxygen radicals generated during anoxia followed by reoxygenation reduce the synthesis of tissue-type plasminogen activator and plasminogen activator inhibitor-1 in human endothelial cell culture. , 1990, The Journal of biological chemistry.
[80] J. Garcìa,et al. Brain microvessels: factors altering their patency after the occlusion of a middle cerebral artery (Wistar rat). , 1994, The American journal of pathology.
[81] J. Koziol,et al. Activated Microvessels Express Vascular Endothelial Growth Factor and Integrin αvβ3 During Focal Cerebral Ischemia , 1999 .
[82] B. Siesjö,et al. Molecular mechanisms of acidosis-mediated damage. , 1996, Acta neurochirurgica. Supplement.
[83] E. Haley. High-dose tirilazad for acute stroke (RANTTAS II). RANTTAS II Investigators. , 1998, Stroke.
[84] T. Davis,et al. Calcium Modulation of Adherens and Tight Junction Function: A Potential Mechanism for Blood-Brain Barrier Disruption After Stroke , 2002, Stroke.
[85] D. Ray,et al. Reversible disruption of tight junction complexes in the rat blood‐brain barrier, following transitory focal astrocyte loss , 2004, Glia.
[86] O. Wu,et al. Delayed rt-PA Treatment in a Rat Embolic Stroke Model: Diagnosis and Prognosis of Ischemic Injury and Hemorrhagic Transformation with Magnetic Resonance Imaging , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[87] R. Caldwell,et al. Experimental diabetes causes breakdown of the blood-retina barrier by a mechanism involving tyrosine nitration and increases in expression of vascular endothelial growth factor and urokinase plasminogen activator receptor. , 2003, The American journal of pathology.
[88] Jieli Chen,et al. Nitric Oxide Enhances Angiogenesis via the Synthesis of Vascular Endothelial Growth Factor and cGMP After Stroke in the Rat , 2003, Circulation research.
[89] K. Takakura,et al. Ebselen in acute ischemic stroke: a placebo-controlled, double-blind clinical trial. Ebselen Study Group. , 1998, Stroke.
[90] W. Armstead,et al. Polyethylene Glycol Superoxide Dismutase and Catalase Attenuate Increased Blood–Brain Barrier Permeability After Ischemia in Piglets , 1992, Stroke.
[91] J. Kourie,et al. Interaction of reactive oxygen species with ion transport mechanisms. , 1998, American journal of physiology. Cell physiology.
[92] G. Rosenberg,et al. Xanthine oxidase activates pro-matrix metalloproteinase-2 in cultured rat vascular smooth muscle cells through non-free radical mechanisms. , 2004, Archives of biochemistry and biophysics.
[93] H. Harper,et al. Clustering of Urokinase Receptors (uPAR; CD87) Induces Proinflammatory Signaling in Human Polymorphonuclear Neutrophils1 , 2000, The Journal of Immunology.
[94] Elisabetta Dejana,et al. Endothelial cell–cell junctions: happy together , 2004, Nature Reviews Molecular Cell Biology.