HLA‐DR3 mediated CD4 T cell response against GAD65 in type 1 diabetes patients
暂无分享,去创建一个
N. Mehra | N. Mishra | N. Tandon | U. Kanga | G. Mishra | Neeraj Kumar | Neihenuo Chuzho | Akanksha Sharma
[1] Xia Li,et al. GAD65 Antibody Epitopes and Genetic Background in Latent Autoimmune Diabetes in Youth (LADY) , 2022, Frontiers in Immunology.
[2] N. Mehra,et al. Differential HLA Association of GAD65 and IA2 Autoantibodies in North Indian Type 1 Diabetes Patients , 2021, Journal of diabetes research.
[3] Hakeem G. Almabrazi,et al. Clinical features, epidemiology, autoantibody status, HLA haplotypes and genetic mechanisms of type 1 diabetes mellitus among children in Qatar , 2021, Scientific Reports.
[4] Wei-Wei Zhang,et al. IL-17A is involved in diabetic inflammatory pathogenesis by its receptor IL-17RA , 2020, Experimental biology and medicine.
[5] H. Siljander,et al. Dynamics of Islet Autoantibodies During Prospective Follow-Up From Birth to Age 15 Years , 2020, The Journal of clinical endocrinology and metabolism.
[6] N. Tandon,et al. Diverse human leukocyte antigen association of type 1 diabetes in north India , 2019, Journal of diabetes.
[7] Zihan Zheng,et al. A complex auxiliary: IL‐17/Th17 signaling during type 1 diabetes progression , 2019, Molecular immunology.
[8] Jason S. Mitchell,et al. Interferon-gamma drives programmed death-ligand 1 expression on islet β cells to limit T cell function during autoimmune diabetes , 2018, Scientific Reports.
[9] G. Allam,et al. The potential pathogenic role of IL-17/Th17 cells in both type 1 and type 2 diabetes mellitus. , 2018, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.
[10] F. Lo,et al. Autoantibodies against islet cell antigens in children with type 1 diabetes mellitus , 2018, Oncotarget.
[11] Adam L. Burrack,et al. T Cell-Mediated Beta Cell Destruction: Autoimmunity and Alloimmunity in the Context of Type 1 Diabetes , 2017, Front. Endocrinol..
[12] C. Mathews,et al. Type I Interferon Is a Catastrophic Feature of the Diabetic Islet Microenvironment , 2017, Front. Endocrinol..
[13] A. Pugliese,et al. Autoreactive T cells in type 1 diabetes. , 2017, The Journal of clinical investigation.
[14] A. Steck,et al. T1D Autoantibodies: room for improvement? , 2017, Current opinion in endocrinology, diabetes, and obesity.
[15] V. Mohan,et al. Registry of Youth Onset Diabetes in India (YDR) , 2016, Journal of diabetes science and technology.
[16] E. Bonifacio,et al. The 6 year incidence of diabetes-associated autoantibodies in genetically at-risk children: the TEDDY study , 2015, Diabetologia.
[17] P. Santamaria,et al. MHC Class II Polymorphisms, Autoreactive T-Cells, and Autoimmunity , 2013, Front. Immunol..
[18] N. Tandon,et al. Genomic evaluation of HLA‐DR3+ haplotypes associated with type 1 diabetes , 2013, Annals of the New York Academy of Sciences.
[19] Junbao Yang,et al. CD4+ T cells recognize diverse epitopes within GAD65: implications for repertoire development and diabetes monitoring , 2013, Immunology.
[20] M. Peakman,et al. Antigen targets of type 1 diabetes autoimmunity. , 2012, Cold Spring Harbor perspectives in medicine.
[21] M. Rewers,et al. The interplay of autoimmunity and insulin resistance in type 1 diabetes. , 2012, Discovery medicine.
[22] M. Atkinson,et al. Demonstration of islet-autoreactive CD8 T cells in insulitic lesions from recent onset and long-term type 1 diabetes patients , 2012, The Journal of experimental medicine.
[23] N. Mehra,et al. Utility of saliva and hair follicles in donor selection for hematopoietic stem cell transplantation and chimerism monitoring , 2012, Chimerism.
[24] M. Pietropaolo,et al. GAD65 autoantibodies and its role as biomarker of Type 1 diabetes and Latent Autoimmune Diabetes in Adults (LADA). , 2011, Drugs of the future.
[25] P. Marrack,et al. Specificity and detection of insulin-reactive CD4+ T cells in type 1 diabetes in the nonobese diabetic (NOD) mouse , 2011, Proceedings of the National Academy of Sciences.
[26] P. Bingley,et al. Evidence That HLA Class I and II Associations With Type 1 Diabetes, Autoantibodies to GAD and Autoantibodies to IA-2, Are Distinct , 2011, Diabetes.
[27] J. Todd,et al. Genetic Analysis of Adult-Onset Autoimmune Diabetes , 2011, Diabetes.
[28] W. Almawi,et al. Glutamic Acid Decarboxylase 65 and Islet Cell Antigen 512/IA-2 Autoantibodies in Relation to Human Leukocyte Antigen Class II DR and DQ Alleles and Haplotypes in Type 1 Diabetes Mellitus , 2011, Clinical and Vaccine Immunology.
[29] J. Ilonen,et al. IL-17 Immunity in Human Type 1 Diabetes , 2010, The Journal of Immunology.
[30] Johnny Ludvigsson,et al. GAD treatment and insulin secretion in recent-onset type 1 diabetes. , 2008, The New England journal of medicine.
[31] V. Kuchroo,et al. Interplay Between Effector Th17 and Regulatory T Cells , 2008, Journal of Clinical Immunology.
[32] J. Barker,et al. Autoantibodies in type 1 diabetes , 2008, Autoimmunity.
[33] E. Bonifacio,et al. GAD Autoantibody Affinity and Epitope Specificity Identify Distinct Immunization Profiles in Children at Risk for Type 1 Diabetes , 2007, Diabetes.
[34] M. Peakman,et al. Translational Mini‐Review Series on Type 1 Diabetes: Systematic analysis of T cell epitopes in autoimmune diabetes , 2007, Clinical and experimental immunology.
[35] N. Tandon,et al. Biomarkers of susceptibility to type 1 diabetes with special reference to the Indian population. , 2007, The Indian journal of medical research.
[36] H. Mcdevitt,et al. The role of TNF- (cid:1) in the pathogenesis of type 1 diabetes in the nonobese diabetic mouse: Analysis of dendritic cell maturation , 2005 .
[37] H. Mcdevitt,et al. Prevention of type I diabetes transfer by glutamic acid decarboxylase 65 peptide 206-220-specific T cells. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[38] Mark Peakman,et al. Autoreactive T cell responses show proinflammatory polarization in diabetes but a regulatory phenotype in health. , 2004, The Journal of clinical investigation.
[39] P. Snow,et al. Induction of Autoantigen-Specific Th2 and Tr1 Regulatory T Cells and Modulation of Autoimmune Diabetes1 , 2003, The Journal of Immunology.
[40] H. Mcdevitt,et al. CD4+ T Cells from Glutamic Acid Decarboxylase (GAD)65-specific T Cell Receptor Transgenic Mice Are Not Diabetogenic and Can Delay Diabetes Transfer , 2002, The Journal of experimental medicine.
[41] R. Flavell,et al. A Dual Role for TNF-α in Type 1 Diabetes: Islet-Specific Expression Abrogates the Ongoing Autoimmune Process When Induced Late but Not Early During Pathogenesis1 , 2001, The Journal of Immunology.
[42] E. Sercarz,et al. Regulatory and Effector CD4 T Cells in Nonobese Diabetic Mice Recognize Overlapping Determinants on Glutamic Acid Decarboxylase and Use Distinct Vβ Genes1 , 2001, The Journal of Immunology.
[43] R. Tisch,et al. Induction of glutamic acid decarboxylase 65-specific Th2 cells and suppression of autoimmune diabetes at late stages of disease is epitope dependent. , 1999, Journal of immunology.
[44] G. Benichou,et al. Differential activation of T cells by natural antigen peptide analogues: influence on autoimmune and alloimmune in vivo T cell responses. , 1998, Journal of immunology.
[45] H. Erlich,et al. Genetics of type 1 diabetes. , 2012, Cold Spring Harbor perspectives in medicine.
[46] P. Bingley. Clinical applications of diabetes antibody testing. , 2010, The Journal of clinical endocrinology and metabolism.
[47] H. Mugishima,et al. Differences in prevalence of antibodies to GAD and IA-2 and their titers at diagnosis in children with slowly and rapidly progressive forms of type 1 diabetes. , 2009, Diabetes research and clinical practice.
[48] H. Mcdevitt,et al. The role of TNF-alpha in the pathogenesis of type 1 diabetes in the nonobese diabetic mouse: analysis of dendritic cell maturation. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[49] N. Mehra,et al. HLA haplotypes associated with type 1 diabetes mellitus in North Indian children. , 2004, Human immunology.
[50] G. Soltész,et al. Prevalence and HLA association of GAD65 antibodies in Hungarian schoolchildren. , 2003, Human immunology.
[51] F. Wong,et al. GAD-reactive CD4+ Th1 cells induce diabetes in NOD/SCID mice. , 1998, The Journal of clinical investigation.
[52] W. Scherbaum,et al. GAD65 is recognized by T-cells, but not by antibodies from NOD-mice. , 1994, Autoimmunity.