Differential effects of N-methyl-D-aspartate (NMDA) and non-NMDA receptor antagonists on spinal release of amino acids after development of acute arthritis in rats
暂无分享,去创建一个
[1] K. Westlund,et al. Reduction in joint swelling and hyperalgesia following post-treatment with a non-NMDA glutamate receptor antagonist , 1994, Pain.
[2] K. Westlund,et al. Joint inflammation is reduced by dorsal rhizotomy and not by sympathectomy or spinal cord transection. , 1994, Annals of the rheumatic diseases.
[3] W. Willis,et al. Do dorsal root reflexes augment peripheral inflammation? , 1994, Neuroreport.
[4] K. Westlund,et al. Behavioral and immunohistochemical changes in an experimental arthritis model in rats , 1993, Pain.
[5] R. Dubner,et al. NMDA receptor antagonists attenuate mechanical hyperalgesia in rats with unilateral inflammation of the hindpaw , 1993, Neuroscience Letters.
[6] W. Willis,et al. Joint inflammation and hyperalgesia are reduced by spinal bicuculline. , 1993, Neuroreport.
[7] K. Westlund,et al. Spinal cord amino acid release and content in an arthritis model: the effects of pretreatment with non-NMDA, NMDA, and NK1 receptor antagonists , 1993, Brain Research.
[8] K. Westlund,et al. Centrally administered non-NMDA but not NMDA receptor antagonists block peripheral knee joint inflammation , 1993, Pain.
[9] H. Schaible,et al. N-methyl-D-aspartate (NMDA) and non-NMDA receptor antagonists block the hyperexcitability of dorsal horn neurons during development of acute arthritis in rat's knee joint. , 1993, Journal of neurophysiology.
[10] K. Westlund,et al. An experimental arthritis model in rats: The effects of NMDA and non-NMDA antagonists on aspartate and glutamate release in the dorsal horn , 1993, Neuroscience Letters.
[11] W. Willis,et al. The non-NMDA antagonist CNQX prevents release of amino acids into the rat spinal cord dorsal horn evoked by sciatic nerve stimulation , 1992, Neuroscience Letters.
[12] K. Westlund,et al. An experimental arthritis in rats: Dorsal horn aspartate and glutamate increases , 1992, Neuroscience Letters.
[13] R. Dubner,et al. The intrathecal administration of excitatory amino acid receptor antagonists selectively attenuated carrageenan-induced behavioral hyperalgesia in rats. , 1992, European journal of pharmacology.
[14] L. Sorkin,et al. The role of NMDA and non-NMDA excitatory amino acid receptors in the excitation of primate spinothalamic tract neurons by mechanical, chemical, thermal, and electrical stimuli , 1992, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[15] W. Willis,et al. Neural changes in acute arthritis in monkeys. I. Parallel enhancement of responses of spinothalamic tract neurons to mechanical stimulation and excitatory amino acids , 1992, Brain Research Reviews.
[16] W. Willis,et al. Neural changes in acute arthritis in monkeys. IV. Time-course of amino acid release into the lumbar dorsal horn , 1992, Brain Research Reviews.
[17] W. Willis,et al. Neural changes in acute arthritis in monkeys. III. Changes in substance P, calcitonin gene-related peptide and glutamate in the dorsal horn of the spinal cord , 1992, Brain Research Reviews.
[18] W. Willis,et al. Neural changes in acute arthritis in monkeys. II. Increased glutamate immunoreactivity in the medial articular nerve , 1992, Brain Research Reviews.
[19] D. Price,et al. Intrathecal MK-801 and local nerve anesthesia synergistically reduce nociceptive behaviors in rats with experimental peripheral mononeuropathy , 1992, Brain Research.
[20] T. Salt. The possible involvement of excitatory amino acids and NMDA receptors in thalamic pain mechanisms and central pain syndromes , 1992 .
[21] D. Budai,et al. Enhancement of NMDA-evoked neuronal activity by glycine in the rat spinal cord in vivo , 1992, Neuroscience Letters.
[22] A. Beitz,et al. Fluorogold administration via microdialysis labels neurons terminating within the dialysis region , 1992, Journal of Neuroscience Methods.
[23] H. Schaible,et al. The Effects of NMDA Antagonists on Neuronal Activity in Cat Spinal Cord Evoked by Acute Inflammation in the Knee Joint , 1991, The European journal of neuroscience.
[24] A. Hama,et al. MK-801 blocks the development of thermal hyperalgesia in a rat model of experimental painful neuropathy , 1991, Brain Research.
[25] F. F. Lam,et al. Inhibition of carrageenan induced inflammation in the rat knee joint by substance P antagonist. , 1989, Annals of the rheumatic diseases.
[26] M. Randić,et al. Excitatory and inhibitory amino acids and peptide-induced responses in acutely isolated rat spinal dorsal horn neurons , 1989, Neuroscience Letters.
[27] H. Schaible,et al. Time course of mechanosensitivity changes in articular afferents during a developing experimental arthritis. , 1988, Journal of neurophysiology.
[28] R. Dubner,et al. A new and sensitive method for measuring thermal nociception in cutaneous hyperalgesia , 1987, Pain.
[29] G. Wilcox,et al. Nociceptive action of excitatory amino acids in the mouse: effects of spinally administered opioids, phencyclidine and sigma agonists. , 1987, The Journal of pharmacology and experimental therapeutics.
[30] G. Urca,et al. IntrathecalN-methyl-d-aspartate (NMDA) activates both nociceptive and antinociceptive systems , 1987, Brain Research.
[31] W. Ferrell,et al. Extravasation in the knee induced by antidromic stimulation of articular C fibre afferents of the anaesthetized cat. , 1986, The Journal of physiology.
[32] T. Williams,et al. Inflammatory oedema induced by synergism between calcitonin gene‐related peptide (CGRP) and mediators of increased vascular permeability , 1985, British journal of pharmacology.
[33] H. Schaible,et al. Effects of an experimental arthritis on the sensory properties of fine articular afferent units. , 1985 .
[34] M. Moskowitz,et al. Intraneuronal substance P contributes to the severity of experimental arthritis. , 1984, Science.
[35] C. Woolf. Evidence for a central component of post-injury pain hypersensitivity , 1983, Nature.