Left ventricular flow propagation during early filling is related to wall relaxation: a color M-mode Doppler analysis.

[1]  A. Beckett,et al.  AKUFO AND IBARAPA. , 1965, Lancet.

[2]  J. Weiss,et al.  Hemodynamic determinants of the time-course of fall in canine left ventricular pressure. , 1976, The Journal of clinical investigation.

[3]  Anderson,et al.  Viscoelastic properties of the diastolic left ventricle in the conscious dog. , 1977, Circulation research.

[4]  J. Weiss,et al.  Quantification of incomplete left ventricular relaxation: relationship to the time constant for isovolumic pressure fall. , 1980, European heart journal.

[5]  R. W. Brower,et al.  Interaction of left ventricular relaxation and filling during early diastole in human subjects. , 1980, The American journal of cardiology.

[6]  D. Brutsaert,et al.  Dual control of relaxation. Its role in the ventricular function in the mammalian heart. , 1980, Circulation research.

[7]  Y. Hirota,et al.  A Clinical Study of Left Ventricular Relaxation , 1980, Circulation.

[8]  L. Brasseur,et al.  Relationship Between Changes in Left Ventricular Inotropic State and Relaxation in Normal Subjects and in Patients with Coronary Artery Disease , 1981, Circulation.

[9]  W. Paulus,et al.  Modification of Abnormal Left Ventricular Diastolic Properties by Nifedipine in Patients with Hypertrophic Cardiomyopathy , 1982, Circulation.

[10]  J. S. Cole,et al.  Negative U wave: a highly specific but poorly understood sign of heart disease. , 1982, The American journal of cardiology.

[11]  C. B. Waldron,et al.  Analysis of Left Ventricular Pressure During Isovolumic Relaxation in Coronary Artery Disease , 1982, Circulation.

[12]  J. Carroll,et al.  Left ventricular relaxation in patients with left ventricular hypertrophy secondary to aortic valve disease. , 1982, Circulation.

[13]  P. Serruys,et al.  Effects of short-term administration of verapamil on left ventricular relaxation and filling dynamics measured by a combined hemodynamic-ultrasonic technique in patients with hypertrophic cardiomyopathy. , 1983, Circulation.

[14]  W. Paulus,et al.  Increased Regional Myocardial Stiffness of the Left Ventricle During Pacing‐induced Angina in Man , 1983, Circulation.

[15]  W. E. Craig,et al.  Comparison of the effects of nitroprusside and nifedipine on diastolic properties in patients with hypertrophic cardiomyopathy: altered left ventricular loading or improved muscle inactivation? , 1983, Journal of the American College of Cardiology.

[16]  W. Lew,et al.  Regional circumferential lengthening patterns in canine left ventricle. , 1983, The American journal of physiology.

[17]  J. Carroll,et al.  Exercise‐induced Ischemia: The Influence of Altered Relaxation on Early Diastolic Pressures , 1983, Circulation.

[18]  I. Mirsky.  Assessment of diastolic function: suggested methods and future considerations. , 1984, Circulation.

[19]  D. Wieting,et al.  3 – Dynamics and fluid dynamics of the mitral valve , 1984 .

[20]  M. Matsuzaki,et al.  Asynchronous left ventricular diastolic filling in patients with isolated disease of the left anterior descending coronary artery: assessment with radionuclide ventriculography. , 1984, Circulation.

[21]  F. Rademakers,et al.  Triple control of relaxation: implications in cardiac disease. , 1984, Circulation.

[22]  M. Quiñones,et al.  Determination of parameters of left ventricular diastolic filling with pulsed Doppler echocardiography: comparison with cineangiography. , 1985, Circulation.

[23]  J. Gwathmey,et al.  Altered calcium handling in experimental pressure-overload hypertrophy in the ferret. , 1985, Circulation research.

[24]  R. Gill.  Measurement of blood flow by ultrasound: accuracy and sources of error. , 1985, Ultrasound in medicine & biology.

[25]  Y Ishida,et al.  Left ventricular filling dynamics: influence of left ventricular relaxation and left atrial pressure. , 1986, Circulation.

[26]  J. Carroll,et al.  Myocardial relaxation: effects of preload on the time course of isovolumetric relaxation. , 1986, Circulation.

[27]  B. Lorell,et al.  Altered left ventricular diastolic properties during pacing-induced angina in patients with aortic stenosis. , 1986, Circulation.

[28]  J. Carroll,et al.  The differential effects of positive inotropic and vasodilator therapy on diastolic properties in patients with congestive cardiomyopathy. , 1986, Circulation.

[29]  O. Hess,et al.  Does verapamil improve left ventricular relaxation in patients with myocardial hypertrophy? , 1986, Circulation.

[30]  D. Altman,et al.  STATISTICAL METHODS FOR ASSESSING AGREEMENT BETWEEN TWO METHODS OF CLINICAL MEASUREMENT , 1986, The Lancet.

[31]  O. Hess,et al.  Myocardial relaxation and passive diastolic properties in man. , 1986, Circulation.

[32]  O. Hess,et al.  Diastolic filling dynamics in patients with aortic stenosis. , 1986, Circulation.

[33]  A. Labovitz,et al.  Evaluation of left ventricular diastolic function: clinical relevance and recent Doppler echocardiographic insights. , 1987, American heart journal.

[34]  Robert A. Phillips,et al.  Doppler echocardiography analysis of left ventricular filling in treated hypertensive patients , 1987 .

[35]  R. Bonow,et al.  Regional left ventricular asynchrony and impaired global left ventricular filling in hypertrophic cardiomyopathy: effect of verapamil. , 1987, Journal of the American College of Cardiology.

[36]  George A. Williams,et al.  Effect of aging on left ventricular diastolic filling in normal subjects. , 1987, The American journal of cardiology.

[37]  R. Walsh,et al.  Load independence of the rate of isovolumic relaxation in man. , 1987, Circulation.

[38]  D. Brutsaert,et al.  Nonuniformity: a physiologic modulator of contraction and relaxation of the normal heart. , 1987, Journal of the American College of Cardiology.

[39]  L. Hatle,et al.  Intraventricular flow during isovolumic relaxation: description and characterization by Doppler echocardiography. , 1987, Journal of the American College of Cardiology.

[40]  W Grossman,et al.  Abnormal intracellular calcium handling in myocardium from patients with end-stage heart failure. , 1987, Circulation research.

[41]  P. Ludbrook,et al.  The transmitral pressure-flow velocity relation. Effect of abrupt preload reduction. , 1988, Circulation.

[42]  P. Ludmer,et al.  Intracoronary infusion of dobutamine to patients with and without severe congestive heart failure. Dose-response relationships, correlation with circulating catecholamines, and effect of phosphodiesterase inhibition. , 1988, The Journal of clinical investigation.

[43]  B. Lorell,et al.  Diastolic function in patients with aortic stenosis: influence of left ventricular load reduction. , 1988, Journal of the American College of Cardiology.

[44]  R L Popp,et al.  Relation of Transmitral Flow Velocity Patterns to Left Ventricle Diastolic Function: New Insights From a Combined Hemodynamic and Doppler Echocardiographic Study , 1988, Journal of the American College of Cardiology.

[45]  E. Pritchett,et al.  Prevention of symptomatic recurrences of paroxysmal atrial fibrillation in patients initially tolerating antiarrhythmic therapy. A multicenter, double-blind, crossover study of flecainide and placebo with transtelephonic monitoring. Flecainide Supraventricular Tachycardia Study Group. , 1989, Circulation.

[46]  Jean Charles Gilbert,et al.  Determinants of left ventricular filling and of the diastolic pressure-volume relation. , 1989, Circulation research.

[47]  R. Bonow,et al.  Beta-adrenergic stimulation with isoproterenol enhances left ventricular diastolic performance in hypertrophic cardiomyopathy despite potentiation of myocardial ischemia. Comparison to rapid atrial pacing. , 1989, Circulation.

[48]  A. Tajik,et al.  Assessment of diastolic function of the heart: background and current applications of Doppler echocardiography. Part I. Physiologic and pathophysiologic features. , 1989, Mayo Clinic proceedings.

[49]  M. Feldman,et al.  Is tau a preload-independent measure of isovolumetric relaxation? , 1989, Circulation.

[50]  P Rizzon,et al.  Functional and structural abnormalities in patients with dilated cardiomyopathy. , 1989, Journal of the American College of Cardiology.

[51]  Left ventricular diastolic function: comparison of pulsed Doppler echocardiographic and hemodynamic indexes in subjects with and without coronary artery disease. , 1989, Journal of the American College of Cardiology.

[52]  W. Lew,et al.  Nonuniformity and volume loading independently influence isovolumic relaxation rates. , 1989, The American journal of physiology.

[53]  A. Weyman,et al.  Analysis of the early transmitral Doppler velocity curve: effect of primary physiologic changes and compensatory preload adjustment. , 1990, Journal of the American College of Cardiology.

[54]  M. Kotler,et al.  Flow patterns in dilated cardiomyopathy: a pulsed-wave and color flow Doppler study. , 1990, Journal of the American Society of Echocardiography : official publication of the American Society of Echocardiography.

[55]  P. Brun,et al.  Relations between posterior wall kinetics during diastole and left ventricular filling. , 1990, Journal of the American College of Cardiology.

[56]  G. Heyndrickx,et al.  Wide-range load shift of combined aortic valvuloplasty-arterial vasodilation slows isovolumic relaxation of the hypertrophied left ventricle. , 1990, Circulation.

[57]  E. Yellin,et al.  Diastolic viscous properties of the intact canine left ventricle. , 1979, Circulation research.

[58]  O. Smiseth,et al.  Left ventricular filling at elevated diastolic pressures: relationship between transmitral Doppler flow velocities and atrial contribution. , 1990, American heart journal.

[59]  I. Mirsky,et al.  Effects of beta-adrenergic stimulation with dobutamine on isovolumic relaxation in the normal and failing human left ventricle. , 1991, Circulation.

[60]  R L Popp,et al.  Left ventricular diastolic dysfunction in end-stage dilated cardiomyopathy: simultaneous Doppler echocardiography and hemodynamic evaluation. , 1991, Journal of the American Society of Echocardiography : official publication of the American Society of Echocardiography.