Dietary restriction attenuates the neuronal loss, induction of heme oxygenase-1 and blood–brain barrier breakdown induced by impaired oxidative metabolism
暂无分享,去创建一个
[1] George Paxinos,et al. The Mouse Brain in Stereotaxic Coordinates , 2001 .
[2] Paul L Huang,et al. Vascular Factors Are Critical in Selective Neuronal Loss in an Animal Model of Impaired Oxidative Metabolism , 2000, Journal of neuropathology and experimental neurology.
[3] G. Desideri,et al. Early upregulation of endothelial adhesion molecules in obese hypertensive men. , 1999, Hypertension.
[4] M. Mattson,et al. Dietary restriction protects hippocampal neurons against the death-promoting action of a presenilin-1 mutation , 1999, Brain Research.
[5] G. Gibson,et al. Oxidative stress is associated with region-specific neuronal death during thiamine deficiency. , 1999, Journal of neuropathology and experimental neurology.
[6] M. Mattson,et al. Dietary restriction and 2‐deoxyglucose administration improve behavioral outcome and reduce degeneration of dopaminergic neurons in models of Parkinson's disease , 1999, Journal of neuroscience research.
[7] M. Mattson,et al. No benefit of dietary restriction on disease onset or progression in amyotrophic lateral sclerosis Cu/Zn-superoxide dismutase mutant mice , 1999, Brain Research.
[8] M. Mattson,et al. Food restriction reduces brain damage and improves behavioral outcome following excitotoxic and metabolic insults , 1999, Annals of neurology.
[9] S. Gandy,et al. Induction of nitric oxide synthase and microglial responses precede selective cell death induced by chronic impairment of oxidative metabolism. , 1998, The American journal of pathology.
[10] B. Yu,et al. Iron accumulation in aging: modulation by dietary restriction , 1998, Mechanisms of Ageing and Development.
[11] B. Brüne,et al. Nitrosative and oxidative stress induced heme oxygenase-1 accumulation in rat mesangial cells. , 1998, European journal of pharmacology.
[12] J. Buxbaum,et al. Novel neuritic clusters with accumulations of amyloid precursor protein and amyloid precursor-like protein 2 immunoreactivity in brain regions damaged by thiamine deficiency. , 1996, The American journal of pathology.
[13] Karen Marder,et al. Dietary lipids and antioxidants in Parkinson's disease: A population‐based, case‐control study , 1996, Annals of neurology.
[14] D. Latchman,et al. The degree of protection provided to neuronal cells by a pre-conditioning stress correlates with the amount of heat shock protein 70 it induces and not with the similarity of the subsequent stress , 1995, Neuroscience Letters.
[15] A. Turturro,et al. Longevity, Body Weight, and Neoplasia in Ad Libitum-Fed and Diet-Restricted C57BL6 Mice Fed NIH-31 Open Formula Diet , 1995, Toxicologic pathology.
[16] G. Gibson,et al. Blood-Brain Barrier Abnormalities in Vulnerable Brain Regions during Thiamine Deficiency , 1995, Experimental Neurology.
[17] J. Olney,et al. Excitotoxic Cytopathology, Progression, and Reversibility of Thiamine Deficiency‐induced Diencephalic Lesions , 1995, Journal of neuropathology and experimental neurology.
[18] J. Pipkin,et al. Chronic caloric restriction induces stress proteins in the hypothalamus of rats , 1994, Mechanisms of Ageing and Development.
[19] N. Hattori,et al. An immunohistochemical study on α‐ketoglutarate dehydrogenase complex in Parkinson's disease , 1994 .
[20] J. Kril,et al. Thiamine-dependent enzyme changes in the brains of alcoholics: relationship to the Wernicke-Korsakoff syndrome. , 1993, Alcoholism, clinical and experimental research.
[21] M. Ingvar,et al. A one-step immunohistochemical method for detection of blood—brain barrier disturbances for immunoglobulins in lesioned rat brain with special reference to false-positive labelling in immunohistochemistry , 1993, Journal of Neuroscience Methods.
[22] R. J. Mullen,et al. NeuN, a neuronal specific nuclear protein in vertebrates. , 1992, Development.
[23] G. Brotzman. Thiamine Deficiency , 1992, The Journal of the American Board of Family Medicine.
[24] D. Lowenstein,et al. The stress protein response in cultured neurons: Characterization and evidence for a protective role in excitotoxicity , 1991, Neuron.
[25] B. Yu,et al. Modulation of free radicals and superoxide dismutases by age and dietary restriction , 1990, Aging.
[26] P. Langlais,et al. Protective effects of the glutamate antagonist MK-801 on pyrithiamine- induced lesions and amino acid changes in rat brain , 1990, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[27] B. Yu,et al. Effect of chronic food restriction in aging rats II. Liver cytosolic antioxidants and related enzymes , 1989, Mechanisms of Ageing and Development.
[28] Richard Weindruch,et al. The Retardation of Aging and Disease by Dietary Restriction , 1988 .
[29] J. Blass,et al. Reduced activities of thiamine-dependent enzymes in the brains and peripheral tissues of patients with Alzheimer's disease. , 1988, Archives of neurology.
[30] R. Weindruch,et al. Influences of dietary restriction and age on liver enzyme activities and lipid peroxidation in mice. , 1987, The Journal of nutrition.
[31] S. Hsu,et al. Use of avidin-biotin-peroxidase complex (ABC) in immunoperoxidase techniques: a comparison between ABC and unlabeled antibody (PAP) procedures. , 1981, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[32] M. R. Mickey,et al. Survival and disease patterns in C57BL/6J mice subjected to undernutrition , 1980, Experimental Gerontology.
[33] Manjit,et al. Neurology , 1912, NeuroImage.
[34] R. Coggeshall,et al. Methods for determining numbers of cells and synapses: A case for more uniform standards of review , 1996, The Journal of comparative neurology.
[35] B. Yu. Modulation of aging processes by dietary restriction , 1994 .