Case 1: A neonate with vesicular scalp lesions.
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A baby was born via vaginal delivery at 41 weeks’ gestation to a 25-year-old primigravid with no known history of genital herpes simplex virus (HSV). Both parents reported previous oral HSV but no active lesions. Maternal history revealed a childhood varicella zoster virus (VZV) infection. Antenatal VZV titres were consistent with immunity. Integrated prenatal screen testing was declined. Membranes ruptured 8 h before delivery, with one documented maternal fever of 38.5°C. No genital HSV lesions were observed at delivery.
[1] M. García-Romero,et al. BULLOUS APLASIA CUTIS CONGENITA: CASE REPORT AND REVIEW OF THE LITERATURE , 2011, Indian journal of dermatology.
[2] L. Eichenfield,et al. Bullous aplasia cutis congenita. , 2003, Journal of the American Academy of Dermatology.
[3] J. F. Klein,et al. Aplasia Cutis Congenita in a Setting of Fetus Papyraceus Associated with Small Fetal Abdominal Circumference and High Alpha‐Fetoprotein and Amniotic Acetylcholinesterase , 2015, Pediatric dermatology.