Native incretins prevent the development of atherosclerotic lesions in apolipoprotein E knockout mice
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A. Miyazaki | T. Hirano | M. Terasaki | K. Nohtomi | Takuya Watanabe | M. Tomoyasu | M. Nagashima | J. Kim‐Kaneyama | T. Watanabe | J. Kim‑Kaneyama | Michishige Terasaki | Masako Tomoyasu | Masaharu Nagashima | Joo-Ri Kim-Kaneyama | Joo-ri Kim-Kaneyama
[1] S. Yamagishi,et al. Vildagliptin blocks vascular injury in thoracic aorta of diabetic rats by suppressing advanced glycation end product-receptor axis. , 2011, Pharmacological research.
[2] W. L. Jin,et al. Exendin-4, a glucagon-like peptide-1 receptor agonist, reduces intimal thickening after vascular injury. , 2011, Biochemical and biophysical research communications.
[3] S. Miyamoto,et al. Glucagon-like peptide-1 receptor agonist ameliorates renal injury through its anti-inflammatory action without lowering blood glucose level in a rat model of type 1 diabetes , 2011, Diabetologia.
[4] M. Shichiri,et al. Chronic infusion of salusin-alpha and -beta exerts opposite effects on atherosclerotic lesion development in apolipoprotein E-deficient mice. , 2010, Atherosclerosis.
[5] R. D. de Boer,et al. Glucagon-Like Peptide 1 Prevents Reactive Oxygen Species–Induced Endothelial Cell Senescence Through the Activation of Protein Kinase A , 2010, Arteriosclerosis, thrombosis, and vascular biology.
[6] S. N. Murthy,et al. The synthetic GLP-I receptor agonist, exenatide, reduces intimal hyperplasia in insulin resistant rats , 2010, Diabetes & vascular disease research.
[7] Yutaka Seino,et al. Intact glucagon-like peptide-1 levels are not decreased in Japanese patients with type 2 diabetes. , 2010, Endocrine journal.
[8] R. Kawamori,et al. Inhibition of Monocyte Adhesion to Endothelial Cells and Attenuation of Atherosclerotic Lesion by a Glucagon-like Peptide-1 Receptor Agonist, Exendin-4 , 2010, Diabetes.
[9] Youichi Kobayashi,et al. Preventive Effects of Heregulin-&bgr;1 on Macrophage Foam Cell Formation and Atherosclerosis , 2009, Circulation research.
[10] S. Shioda,et al. Leptin modulates ACAT1 expression and cholesterol efflux from human macrophages. , 2009, American journal of physiology. Endocrinology and metabolism.
[11] M. Nagasawa,et al. Cilostazol inhibits modified low-density lipoprotein uptake and foam cell formation in mouse peritoneal macrophages. , 2009, Atherosclerosis.
[12] Alberto Smith,et al. The monocyte/macrophage as a therapeutic target in atherosclerosis. , 2009, Current opinion in pharmacology.
[13] A. Dear,et al. A long-acting glucagon-like peptide-1 analogue attenuates induction of plasminogen activator inhibitor type-1 and vascular adhesion molecules. , 2009, The Journal of endocrinology.
[14] J. Egan,et al. The Role of Incretins in Glucose Homeostasis and Diabetes Treatment , 2008, Pharmacological Reviews.
[15] N. Irwin,et al. (Pro3)GIP[mPEG]: novel, long‐acting, mPEGylated antagonist of gastric inhibitory polypeptide for obesity‐diabetes (diabesity) therapy , 2008, British journal of pharmacology.
[16] M. Adachi,et al. Chronic urotensin II infusion enhances macrophage foam cell formation and atherosclerosis in apolipoprotein E-knockout mice , 2008, Journal of hypertension.
[17] M. Shichiri,et al. Impact of Salusin-α and -β on Human Macrophage Foam Cell Formation and Coronary Atherosclerosis , 2008, Circulation.
[18] I. Shimomura,et al. Continuous stimulation of human glucagon-like peptide-1 (7–36) amide in a mouse model (NOD) delays onset of autoimmune type 1 diabetes , 2007, Diabetologia.
[19] D. Drucker. The biology of incretin hormones. , 2006, Cell metabolism.
[20] M. Adachi,et al. Human Urotensin II Accelerates Foam Cell Formation in Human Monocyte-Derived Macrophages , 2005, Hypertension.
[21] Tanya Hansotia,et al. GIP and GLP-1 as incretin hormones: lessons from single and double incretin receptor knockout mice , 2005, Regulatory Peptides.
[22] J. Holst,et al. Effects of glucagon-like peptide-1 on endothelial function in type 2 diabetes patients with stable coronary artery disease. , 2004, American journal of physiology. Endocrinology and metabolism.
[23] A. Gotto,et al. Induction of CD36 expression by oxidized LDL and IL-4 by a common signaling pathway dependent on protein kinase C and PPAR-gamma. , 2000, Journal of lipid research.
[24] K. Botham. Cyclic AMP and the regulation of cholesterol metabolism. , 1992, Biochemical Society transactions.
[25] F. Palmieri,et al. Dipeptidyl(amino)peptidase IV and post proline cleaving enzyme in cultured endothelial and smooth muscle cells. , 1989, Advances in experimental medicine and biology.