Irisin and ALCAT1 mediated aerobic exercise-alleviated oxidative stress and apoptosis in skeletal muscle of mice with myocardial infarction.
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Zhenjun Tian | Zujie Xu | Fan-Ni Wu | Wenyan Bo | Mengxin Cai | W. Ren | Yixuan Ma | Hangzhuo Li | Shou Pan
[1] Yuguang Shi,et al. Pharmacological inhibition of ALCAT1 mitigates amyotrophic lateral sclerosis by attenuating SOD1 protein aggregation , 2022, Molecular metabolism.
[2] Feng Li-Li,et al. Aerobic exercise and resistance exercise alleviate skeletal muscle atrophy through IGF-1/IGF-1R-PI3K/Akt pathway in mice with myocardial infarction. , 2021, American journal of physiology. Cell physiology.
[3] Yixuan Ma,et al. The Roles of FGF21 and ALCAT1 in Aerobic Exercise-Induced Cardioprotection of Postmyocardial Infarction Mice , 2021, Oxidative medicine and cellular longevity.
[4] Zhenjun Tian,et al. Exercise Training Enhances Myocardial Mitophagy and Improves Cardiac Function via Irisin/FNDC5-PINK1/Parkin Pathway in MI Mice , 2021, Biomedicines.
[5] H. Tsutsui,et al. Systemic oxidative stress is associated with lower aerobic capacity and impaired skeletal muscle energy metabolism in heart failure patients , 2021, Scientific reports.
[6] M. Sandri,et al. Mechanisms of muscle atrophy and hypertrophy: implications in health and disease , 2021, Nature communications.
[7] Ju-Hee Kang,et al. Exercise as a Therapeutic Strategy for Sarcopenia in Heart Failure: Insights into Underlying Mechanisms , 2020, Cells.
[8] J. Springer,et al. Muscle Wasting and Sarcopenia in Heart Failure—The Current State of Science , 2020, International journal of molecular sciences.
[9] Zhuo Li,et al. Aerobic exercise alleviates oxidative stress-induced apoptosis in kidneys of myocardial infarction mice by inhibiting ALCAT1 and activating FNDC5/Irisin signaling pathway. , 2020, Free radical biology & medicine.
[10] S. Uslu,et al. The effect of nutrition and exercise training on irisin and semaphorin-3E levels in obese patients , 2020, Archives of physiology and biochemistry.
[11] T. Nakajima,et al. Inhibition of xanthine oxidase in the acute phase of myocardial infarction prevents skeletal muscle abnormalities and exercise intolerance. , 2020, Cardiovascular research.
[12] H. Crossland,et al. Impacts of rat hind-limb Fndc5/Irisin overexpression upon muscle and adipose tissue metabolism. , 2020, American journal of physiology. Endocrinology and metabolism.
[13] J. Ge,et al. Exercise improves cardiac function and glucose metabolism in mice with experimental myocardial infarction through inhibiting HDAC4 and upregulating GLUT1 expression , 2020, Basic Research in Cardiology.
[14] P. Abete,et al. Sarcopenia and Heart Failure , 2020, Nutrients.
[15] K. Busch,et al. ALCAT1 Overexpression Affects Supercomplex Formation and Increases ROS in Respiring Mitochondria , 2019, Oxidative medicine and cellular longevity.
[16] Min Zhang,et al. Irisin attenuates oxidized low‐density lipoprotein impaired angiogenesis through AKT/mTOR/S6K1/Nrf2 pathway , 2019, Journal of cellular physiology.
[17] J. Nie,et al. Cardiolipin remodeling by ALCAT1 links mitochondrial dysfunction to Parkinson’s diseases , 2019, Aging cell.
[18] Zhiwei Liu,et al. Effects of different types of exercise on skeletal muscle atrophy, antioxidant capacity and growth factors expression following myocardial infarction , 2018, Life sciences.
[19] T. Brzozowski,et al. Myokine irisin-induced protection against oxidative stress in vitro. Involvement of heme oxygenase-1 and antioxidazing enzymes superoxide dismutase-2 and glutathione peroxidase. , 2018, Journal of physiology and pharmacology : an official journal of the Polish Physiological Society.
[20] S. Du,et al. Interval exercise training increases LIF expression and prevents myocardial infarction‐induced skeletal muscle atrophy in rats , 2018, Life sciences.
[21] Musarrat Maisha Reza,et al. Irisin is a pro-myogenic factor that induces skeletal muscle hypertrophy and rescues denervation-induced atrophy , 2017, Nature Communications.
[22] M. Puszczewicz,et al. A review on irisin, a new protagonist that mediates muscle-adipose-bone-neuron connectivity. , 2017, European review for medical and pharmacological sciences.
[23] Jan Dudek. Role of Cardiolipin in Mitochondrial Signaling Pathways , 2017, Front. Cell Dev. Biol..
[24] T. Eijsvogels,et al. Benefits of lifelong exercise training on left ventricular function after myocardial infarction , 2017, European journal of preventive cardiology.
[25] S. Colucci,et al. Irisin and musculoskeletal health , 2017, Annals of the New York Academy of Sciences.
[26] C. Scavone,et al. Exercise training decreases NADPH oxidase activity and restores skeletal muscle mass in heart failure rats. , 2017, Journal of applied physiology.
[27] M. Irigoyen,et al. Myocardial Infarction and Exercise Training: Evidence from Basic Science. , 2017, Advances in experimental medicine and biology.
[28] S. Powers,et al. Redox control of skeletal muscle atrophy. , 2016, Free radical biology & medicine.
[29] Hai-Jian Sun,et al. Irisin inhibits hepatic gluconeogenesis and increases glycogen synthesis via the PI3K/Akt pathway in type 2 diabetic mice and hepatocytes. , 2015, Clinical science.
[30] Sungha Park,et al. Irisin, a novel myokine is an independent predictor for sarcopenia and carotid atherosclerosis in dialysis patients. , 2015, Atherosclerosis.
[31] S. Kim,et al. Irisin, a Novel Myokine, Regulates Glucose Uptake in Skeletal Muscle Cells via AMPK. , 2015, Molecular endocrinology.
[32] G. Schuler,et al. Fibronectin type III domain containing 5 expression in skeletal muscle in chronic heart failure—relevance of inflammatory cytokines , 2015, Journal of cachexia, sarcopenia and muscle.
[33] J. Nie,et al. ALCAT1 controls mitochondrial etiology of fatty liver diseases, linking defective mitophagy to steatosis , 2015, Hepatology.
[34] G. Frühbeck,et al. Leptin administration activates irisin-induced myogenesis via nitric oxide-dependent mechanisms, but reduces its effect on subcutaneous fat browning in mice , 2014, International Journal of Obesity.
[35] T. Egawa,et al. AICAR stimulation metabolome widely mimics electrical contraction in isolated rat epitrochlearis muscle. , 2013, American journal of physiology. Cell physiology.
[36] Michael F. N. O'Leary,et al. The effects of chronic muscle use and disuse on cardiolipin metabolism. , 2013, Journal of applied physiology.
[37] J. Nie,et al. Ablation of ALCAT1 Mitigates Hypertrophic Cardiomyopathy through Effects on Oxidative Stress and Mitophagy , 2012, Molecular and Cellular Biology.
[38] U. Wisløff,et al. Exercise Training Prevents Oxidative Stress and Ubiquitin-Proteasome System Overactivity and Reverse Skeletal Muscle Atrophy in Heart Failure , 2012, PloS one.
[39] D. Chan,et al. Lysocardiolipin acyltransferase 1 (ALCAT1) controls mitochondrial DNA fidelity and biogenesis through modulation of MFN2 expression , 2012, Proceedings of the National Academy of Sciences.
[40] S. Powers,et al. Reactive oxygen species: impact on skeletal muscle. , 2011, Comprehensive Physiology.
[41] Christopher J Lynch,et al. Cardiolipin remodeling by ALCAT1 links oxidative stress and mitochondrial dysfunction to obesity. , 2010, Cell metabolism.
[42] C. Padovani,et al. Chronic heart failure-induced skeletal muscle atrophy, necrosis, and changes in myogenic regulatory factors. , 2010, Medical science monitor : international medical journal of experimental and clinical research.
[43] S. Li,et al. The microsomal cardiolipin remodeling enzyme acyl-CoA lysocardiolipin acyltransferase is an acyltransferase of multiple anionic lysophospholipids This work was supported by Lilly Research Laboratories. Published, JLR Papers in Press, December 15, 2008. , 2009, Journal of Lipid Research.
[44] E. Dupont-Versteegden. Apoptosis in skeletal muscle and its relevance to atrophy. , 2006, World journal of gastroenterology.
[45] D. Hardie,et al. AMP-activated protein kinase: ancient energy gauge provides clues to modern understanding of metabolism. , 2005, Cell metabolism.