Cooperative effect of ribosomal protein s19 and Pim-1 kinase on murine c-Myc expression and myeloid/erythroid cellularity
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[1] N. Dahl,et al. Ribosomal protein S19 and S24 insufficiency cause distinct cell cycle defects in Diamond-Blackfan anemia. , 2009, Biochimica et biophysica acta.
[2] I. Bernstein,et al. Hematopoietic stem cell function and survival depend on c-Myc and N-Myc activity. , 2008, Cell stem cell.
[3] I. Dianzani,et al. A new database for ribosomal protein genes which are mutated in Diamond‐Blackfan Anemia , 2008, Human mutation.
[4] M. Dai,et al. Crosstalk between c‐Myc and ribosome in ribosomal biogenesis and cancer , 2008, Journal of cellular biochemistry.
[5] Davide Ruggero,et al. Suppression of Myc oncogenic activity by ribosomal protein haploinsufficiency , 2008, Nature.
[6] Z. Wang,et al. Pim kinase-dependent inhibition of c-Myc degradation , 2008, Oncogene.
[7] S. Karlsson,et al. Ribosomal Protein S19 Deficiency Leads to Reduced Proliferation and Increased Apoptosis but Does Not Affect Terminal Erythroid Differentiation in a Cell Line Model of Diamond‐Blackfan Anemia , 2008, Stem cells.
[8] T. Golub,et al. Identification of RPS14 as a 5q- syndrome gene by RNA interference screen , 2007, Nature.
[9] M. Dai,et al. Feedback Regulation of c-Myc by Ribosomal Protein L11 , 2007, Cell cycle.
[10] J. Partanen,et al. c-Myc Blazing a Trail of Death: Coupling of the Mitochondrial and Death Receptor Apoptosis Pathways by c-Myc , 2007, Cell cycle.
[11] S. Oliviero,et al. PIM1-dependent phosphorylation of histone H3 at serine 10 is required for MYC-dependent transcriptional activation and oncogenic transformation , 2007, Nature Cell Biology.
[12] Claudio Santoro,et al. Analysis of the Ribosomal Protein S19 Interactome*S , 2007, Molecular & Cellular Proteomics.
[13] S. Karlsson,et al. Human RPS19, the gene mutated in Diamond-Blackfan anemia, encodes a ribosomal protein required for the maturation of 40S ribosomal subunits. , 2007, Blood.
[14] M. Minden,et al. Myelodysplastic syndromes: the complexity of stem-cell diseases , 2007, Nature Reviews Cancer.
[15] H. Tamary,et al. CURRENT DIAGNOSIS OF INHERITED BONE MARROW FAILURE SYNDROMES , 2007, Pediatric hematology and oncology.
[16] S. Karlsson,et al. Erythropoiesis in the Rps19 disrupted mouse: Analysis of erythropoietin response and biochemical markers for Diamond-Blackfan anemia. , 2006, Blood cells, molecules & diseases.
[17] C. Thompson,et al. The survival kinases Akt and Pim as potential pharmacological targets. , 2005, The Journal of clinical investigation.
[18] Koichi Miyake,et al. Deficiency of ribosomal protein S19 in CD34+ cells generated by siRNA blocks erythroid development and mimics defects seen in Diamond-Blackfan anemia. , 2005, Blood.
[19] S. Conrad,et al. c-Myc Suppresses p21WAF1/CIP1 Expression during Estrogen Signaling and Antiestrogen Resistance in Human Breast Cancer Cells* , 2005, Journal of Biological Chemistry.
[20] T. Möröy,et al. The serine/threonine kinase Pim-1. , 2005, The international journal of biochemistry & cell biology.
[21] Jos Jonkers,et al. Mice Deficient for All PIM Kinases Display Reduced Body Size and Impaired Responses to Hematopoietic Growth Factors , 2004, Molecular and Cellular Biology.
[22] A. P. Bolliger. Cytologic evaluation of bone marrow in rats: indications, methods, and normal morphology. , 2004, Veterinary clinical pathology.
[23] O. Silvennoinen,et al. Pim-1 kinase inhibits STAT5-dependent transcription via its interactions with SOCS1 and SOCS3. , 2004, Blood.
[24] S. Karlsson,et al. Targeted Disruption of the Ribosomal Protein S19 Gene Is Lethal Prior to Implantation , 2004, Molecular and Cellular Biology.
[25] P. Pandolfi,et al. Does the ribosome translate cancer? , 2003, Nature Reviews Cancer.
[26] R. Fodde,et al. A Matter of Dosage , 2002, Science.
[27] M Schwab,et al. N‐myc enhances the expression of a large set of genes functioning in ribosome biogenesis and protein synthesis , 2001, The EMBO journal.
[28] Steve D. M. Brown,et al. A systematic, genome-wide, phenotype-driven mutagenesis programme for gene function studies in the mouse , 2000, Nature Genetics.
[29] K. Noguchi,et al. Physical and Functional Interactions between Pim-1 Kinase and Cdc25A Phosphatase , 1999, The Journal of Biological Chemistry.
[30] Peter Gustavsson,et al. The gene encoding ribosomal protein S19 is mutated in Diamond-Blackfan anaemia , 1999, Nature Genetics.
[31] K. Noguchi,et al. Pim-1 kinase stimulates c-Myc-mediated death signaling upstream of caspase-3 (CPP32)-like protease activation , 1997, Oncogene.
[32] P. Laird,et al. In vivo analysis of Pim-1 deficiency. , 1993, Nucleic acids research.
[33] G. Evan,et al. Cooperative interaction between c-myc and bcl-2 proto-oncogenes , 1992, Nature.
[34] D. Green,et al. Apoptotic cell death induced by c-myc is inhibited by bcl-2 , 1992, Nature.
[35] S. Ellis,et al. Diamond Blackfan anemia: a disorder of red blood cell development. , 2008, Current topics in developmental biology.
[36] C. Grandori,et al. Activation by c-Myc of transcription by RNA polymerases I, II and III. , 2006, Biochemical Society symposium.
[37] Claudio Santoro,et al. Interactions between RPS19, mutated in Diamond-Blackfan anemia, and the PIM-1 oncoprotein. , 2005, Haematologica.
[38] B. Alter. Childhood red cell aplasia , 1980 .