altered smooth muscle response to isoprostane decreased pulmonary arterial nitric oxide release and exposure in the newborn rat results in

[1]  A. Tanswell,et al.  Effect of 8-isoprostaglandin F2alpha on the newborn rat pulmonary arterial muscle and endothelium. , 2003, Journal of applied physiology.

[2]  A. Tanswell,et al.  Macrophages as a major source of oxygen radicals in the hyperoxic newborn rat lung. , 2003, Free radical biology & medicine.

[3]  A. Tanswell,et al.  Chronic O2 exposure enhances vascular and airway smooth muscle contraction in the newborn but not adult rat. , 2003, Journal of applied physiology.

[4]  N. Vaziri,et al.  Nitric oxide synthase expression in hypertension induced by inhibition of glutathione synthase. , 2002, The Journal of pharmacology and experimental therapeutics.

[5]  J. Cracowski,et al.  Increased lipid peroxidation in patients with pulmonary hypertension. , 2001, American journal of respiratory and critical care medicine.

[6]  A. Tanswell,et al.  Gadolinium Chloride Inhibits Pulmonary Macrophage Influx and Prevents O2-Induced Pulmonary Hypertension in the Neonatal Rat , 2001, Pediatric Research.

[7]  L. Janssen Isoprostanes: an overview and putative roles in pulmonary pathophysiology. , 2001, American journal of physiology. Lung cellular and molecular physiology.

[8]  A. Tanswell,et al.  Endothelin-1 and O2-Mediated Pulmonary Hypertension in Neonatal Rats: A Role for Products of Lipid Peroxidation , 2000, Pediatric Research.

[9]  N. Vaziri,et al.  Induction of oxidative stress by glutathione depletion causes severe hypertension in normal rats. , 2000, Hypertension.

[10]  Massimo Corradi,et al.  Exhaled 8-isoprostane as a new non-invasive biomarker of oxidative stress in cystic fibrosis , 2000, Thorax.

[11]  E. Camporesi,et al.  Inducible nitric oxide synthase in the lung and exhaled nitric oxide after hyperoxia. , 1999, American journal of physiology. Lung cellular and molecular physiology.

[12]  K. Dikranian,et al.  Expression of nitric oxide synthase isoforms (NOS II and NOS III) in adult rat lung in hyperoxic pulmonary hypertension , 1999, Cell and Tissue Research.

[13]  M. Radomski,et al.  The role of nitric oxide and metalloproteinases in the pathogenesis of hyperoxia‐induced lung injury in newborn rats , 1998, British journal of pharmacology.

[14]  B. Christman,et al.  Exhaled breath condensate isoprostanes are elevated in patients with acute lung injury or ARDS. , 1998, Chest.

[15]  P. Montuschi,et al.  8-Isoprostane as a biomarker of oxidative stress in interstitial lung diseases. , 1998, American journal of respiratory and critical care medicine.

[16]  K. Suzuki,et al.  Impaired hypoxic vasoconstriction in intraacinar microvasculature in hyperoxia-exposed rat lungs. , 1998, American journal of respiratory and critical care medicine.

[17]  O. Saugstad,et al.  Bronchopulmonary dysplasia and oxidative stress: are we closer to an understanding of the pathogenesis of BPD? , 1997, Acta paediatrica.

[18]  H. Wong,et al.  DIFFERENTIAL EFFECTS OF HYPEROXIA ON THE INDUCIBLE AND CONSTITUTIVE ISOFORMS OF NITRIC OXIDE SYNTHASE IN THE LUNG , 1997, Shock.

[19]  T. Evans,et al.  Evidence for a dilator function of 8‐iso prostaglandin F2α in rat pulmonary artery , 1997 .

[20]  Jeffrey F. Young,et al.  Changes in Structure, Mechanics, and Insulin-Like Growth Factor-Related Gene Expression in the Lungs of Newborn Rats Exposed to Air or 60% Oxygen , 1996, Pediatric Research.

[21]  A. Quyyumi,et al.  Impaired endothelium-dependent vasodilation in patients with essential hypertension. Evidence that nitric oxide abnormality is not localized to a single signal transduction pathway. , 1995, Circulation.

[22]  A. Tanswell,et al.  α1-Antitrypsin Protects Neonatal Rats from Pulmonary Vascular and Parenchymal Effects of Oxygen Toxicity , 1994, Pediatric Research.

[23]  N. Flavahan,et al.  Loss of endothelial pertussis toxin-sensitive G protein function in atherosclerotic porcine coronary arteries. , 1991, Circulation.