Multifaceted roles of miR-1s in repressing the fetal gene program in the heart

[1]  P. D. de Jong,et al.  microRNA-1 regulates sarcomere formation and suppresses smooth muscle gene expression in the mammalian heart , 2013, eLife.

[2]  T. Boettger,et al.  miR-1/133a Clusters Cooperatively Specify the Cardiomyogenic Lineage by Adjustment of Myocardin Levels during Embryonic Heart Development , 2013, PLoS genetics.

[3]  Rick B. Vega,et al.  Nuclear receptor/microRNA circuitry links muscle fiber type to energy metabolism. , 2013, The Journal of clinical investigation.

[4]  Carlos Torroja,et al.  Mutations in the NOTCH pathway regulator MIB1 cause left ventricular noncompaction cardiomyopathy , 2013, Nature Medicine.

[5]  E. Olson,et al.  MicroRNAs in cardiovascular disease: from pathogenesis to prevention and treatment. , 2013, The Journal of clinical investigation.

[6]  Richard T. Lee,et al.  Mammalian Heart Renewal by Preexisting Cardiomyocytes , 2012, Nature.

[7]  Jian Wang,et al.  The function of miRNA in cardiac hypertrophy , 2012, Cellular and Molecular Life Sciences.

[8]  杜昕,et al.  Inherited cardiomyopathies , 2012 .

[9]  Chaoqian Xu,et al.  The muscle-specific microRNA miR-1 regulates cardiac arrhythmogenic potential by targeting GJA1 and KCNJ2 , 2011, Nature Medicine.

[10]  C. Thummel,et al.  The Drosophila estrogen-related receptor directs a metabolic switch that supports developmental growth. , 2011, Cell metabolism.

[11]  V. Vedantham,et al.  Direct Reprogramming of Fibroblasts into Functional Cardiomyocytes by Defined Factors , 2010, Cell.

[12]  G. Lopaschuk,et al.  Energy Metabolic Phenotype of the Cardiomyocyte During Development, Differentiation, and Postnatal Maturation , 2010, Journal of cardiovascular pharmacology.

[13]  K. Chien,et al.  Pregenerative medicine: developmental paradigms in the biology of cardiovascular regeneration. , 2010, The Journal of clinical investigation.

[14]  A. Goette,et al.  Changes in microRNA-1 expression and IK1 up-regulation in human atrial fibrillation. , 2009, Heart rhythm.

[15]  Chaoqian Xu,et al.  Tanshinone IIA protects against sudden cardiac death induced by lethal arrhythmias via repression of microRNA‐1 , 2009, British journal of pharmacology.

[16]  D. Roberts,et al.  Human ISL1 heart progenitors generate diverse multipotent cardiovascular cell lineages , 2009, Nature.

[17]  T. Golub,et al.  MicroRNA-1 Negatively Regulates Expression of the Hypertrophy-Associated Calmodulin and Mef2a Genes , 2009, Molecular and Cellular Biology.

[18]  V. Narry Kim,et al.  Functional links between clustered microRNAs: suppression of cell-cycle inhibitors by microRNA clusters in gastric cancer , 2009, Nucleic acids research.

[19]  E. Olson,et al.  microRNA-133a regulates cardiomyocyte proliferation and suppresses smooth muscle gene expression in the heart. , 2008, Genes & development.

[20]  Samuel Bernard,et al.  Evidence for Cardiomyocyte Renewal in Humans , 2008, Science.

[21]  C. Zechner,et al.  A Role for the Transcriptional Coactivator PGC-1α in Muscle Refueling* , 2007, Journal of Biological Chemistry.

[22]  R. Naviaux,et al.  ERRgamma directs and maintains the transition to oxidative metabolism in the postnatal heart. , 2007, Cell metabolism.

[23]  Michael T. McManus,et al.  Dysregulation of Cardiogenesis, Cardiac Conduction, and Cell Cycle in Mice Lacking miRNA-1-2 , 2007, Cell.

[24]  Yong Zhao,et al.  A developmental view of microRNA function. , 2007, Trends in biochemical sciences.

[25]  Jian-Fu Chen,et al.  The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation , 2006, Nature Genetics.

[26]  Christine E Seidman,et al.  The genetic basis for cardiac remodeling. , 2005, Annual review of genomics and human genetics.

[27]  Yong Zhao,et al.  Serum response factor regulates a muscle-specific microRNA that targets Hand2 during cardiogenesis , 2005, Nature.

[28]  William C Stanley,et al.  Myocardial substrate metabolism in the normal and failing heart. , 2005, Physiological reviews.

[29]  J. Seidman,et al.  Glycogen storage diseases presenting as hypertrophic cardiomyopathy. , 2005, The New England journal of medicine.

[30]  A. Depaoli-Roach,et al.  Abnormal Cardiac Development in the Absence of Heart Glycogen , 2004, Molecular and Cellular Biology.

[31]  J. Rychik,et al.  Fetal Cardiovascular Physiology , 2004, Pediatric Cardiology.

[32]  Yuan-Ping Pang,et al.  ABCC9 mutations identified in human dilated cardiomyopathy disrupt catalytic KATP channel gating , 2004, Nature Genetics.

[33]  J. Seidman,et al.  Constitutively active AMP kinase mutations cause glycogen storage disease mimicking hypertrophic cardiomyopathy. , 2002, The Journal of clinical investigation.

[34]  Da-Zhi Wang,et al.  Activation of Cardiac Gene Expression by Myocardin, a Transcriptional Cofactor for Serum Response Factor , 2001, Cell.

[35]  R J Schwartz,et al.  Impaired vascular contractility and blood pressure homeostasis in the smooth muscle α‐actin null mouse , 2000, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[36]  E. Morkin.  Control of cardiac myosin heavy chain gene expression , 2000, Microscopy research and technique.

[37]  W. Wahli,et al.  Peroxisome proliferator-activated receptors: nuclear control of metabolism. , 1999, Endocrine reviews.

[38]  J. Vanoverschelde,et al.  Glucose for the heart. , 1999, Circulation.

[39]  R. Maas,et al.  Embryonic expression suggests an important role for CRP2/SmLIM in the developing cardiovascular system. , 1998, Circulation research.

[40]  K. Birukov,et al.  Organization of the genetic locus for chicken myosin light chain kinase is complex: Multiple proteins are encoded and exhibit differential expression and localization , 1998, Journal of cellular biochemistry.

[41]  G. Boivin,et al.  β-Tropomyosin Overexpression Induces Severe Cardiac Abnormalities , 1998 .

[42]  C. Allis,et al.  Phosphorylation of histone H3 at serine 10 is correlated with chromosome condensation during mitosis and meiosis in Tetrahymena. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[43]  B. A. Harder,et al.  Various hypertrophic stimuli induce distinct phenotypes in cardiomyocytes , 1997, Journal of Molecular Medicine.

[44]  R. Virmani,et al.  Apoptosis in myocytes in end-stage heart failure. , 1996, The New England journal of medicine.

[45]  K. Tanaka,et al.  Molecular cloning and characterization of human non-smooth muscle calponin. , 1996, Journal of biochemistry.

[46]  R. Solaro,et al.  Exchange of - for -Tropomyosin in Hearts of Transgenic Mice Induces Changes in Thin Filament Response to Ca, Strong Cross-bridge Binding, and Protein Phosphorylation (*) , 1996, The Journal of Biological Chemistry.

[47]  E. Olson,et al.  SM22 alpha, a marker of adult smooth muscle, is expressed in multiple myogenic lineages during embryogenesis. , 1996, Circulation research.

[48]  E. Morrisey,et al.  Developmental Pattern of Expression and Genomic Organization of the Calponin-h1 Gene , 1996, The Journal of Biological Chemistry.

[49]  C. Pothoulakis,et al.  Clostridium difficile colitis. , 1994, The New England journal of medicine.

[50]  P. Howles,et al.  Developmental analysis of tropomyosin gene expression in embryonic stem cells and mouse embryos , 1993, Molecular and cellular biology.

[51]  A. Ciechanover,et al.  Stress-induced alterations in autophagic pathway: relationship to ubiquitin system. , 1992, The American journal of physiology.

[52]  K. Boheler,et al.  Switches in cardiac muscle gene expression as a result of pressure and volume overload. , 1992, The American journal of physiology.

[53]  H. Arnold,et al.  A novel human myosin alkali light chain is developmentally regulated. Expression in fetal cardiac and skeletal muscle and in adult atria. , 1988, European journal of biochemistry.

[54]  R. Schwartz,et al.  Sequential activation of alpha-actin genes during avian cardiogenesis: vascular smooth muscle alpha-actin gene transcripts mark the onset of cardiomyocyte differentiation , 1988, The Journal of cell biology.

[55]  J. Weiss,et al.  Glycolysis preferentially inhibits ATP-sensitive K+ channels in isolated guinea pig cardiac myocytes. , 1987, Science.

[56]  W. Friedman,et al.  The intrinsic physiologic properties of the developing heart. , 1972, Progress in cardiovascular diseases.

[57]  P. J. Randle,et al.  The pentose cycle and insulin release in mouse pancreatic islets. , 1972, The Biochemical journal.

[58]  Da-Zhi Wang,et al.  MicroRNAs in heart development. , 2012, Current topics in developmental biology.

[59]  M. Parmacek,et al.  Cardiomyocyte renewal. , 2009, The New England journal of medicine.

[60]  C. Zechner,et al.  A role for the transcriptional coactivator PGC-1alpha in muscle refueling. , 2007, The Journal of biological chemistry.

[61]  J. Rossant,et al.  Deletion of the selection cassette, but not cis-acting elements, in targeted Flk1-lacZ allele reveals Flk1 expression in multipotent mesodermal progenitors. , 2006, Blood.

[62]  F. Epstein,et al.  Inherited cardiomyopathies. , 1994, The New England journal of medicine.

[63]  T. Parker,et al.  Growth factors, proto-oncogenes, and plasticity of the cardiac phenotype. , 1991, Annual review of physiology.