Decreased rate of sodium conductance inactivation in the node of Ranvier induced by a polypeptide toxin from sea anemone.
暂无分享，去创建一个
[1] R. Keynes,et al. The temporal and steady‐state relationships between activation of the sodium conductance and movement of the gating particles in the squid giant axon. , 1976, The Journal of physiology.
[2] L. Goldman. Letter: pronase and models for the sodium conductance , 1975, The Journal of general physiology.
[3] La´szlo Be´ress,et al. Isolation and characterisation of three polypeptides with neurotoxic activity fromAnemonia sulcata , 1975 .
[4] F. Bezanilla,et al. Destruction of Sodium Conductance Inactivation in Squid Axons Perfused with Pronase , 1973, The Journal of general physiology.
[5] W. Ulbricht,et al. The rate of action of tetrodotoxin on myelinated nerve fibres of Xenopus laevis and Rana esculenta , 1973, The Journal of physiology.
[6] L. Mullins. A Single Channel or a Dual Channel Mechanism for Nerve Excitation , 1968, The Journal of general physiology.
[7] C. Tanford. Contribution of Hydrophobic Interactions to the Stability of the Globular Conformation of Proteins , 1962 .
[8] B. Frankenhaeuser,et al. Quantitative description of sodium currents in myelinated nerve fibres of Xenopus laevis , 1960, The Journal of physiology.
[9] F. Dodge,et al. Membrane currents in isolated frog nerve fibre under voltage clamp conditions , 1958, The Journal of physiology.
[10] B. Frankenhaeuser,et al. Potassium permeability in myelinated nerve fibres of Xenopus laevis , 1962, The Journal of physiology.