Degradation of p53 can be targeted by HPV E6 sequences distinct from those required for p53 binding and trans-activation
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[1] T. Crook,et al. Status of c-myc, p53 and retinoblastoma genes in human papillomavirus positive and negative squamous cell carcinomas of the anus. , 1991, Oncogene.
[2] K. Münger,et al. The state of the p53 and retinoblastoma genes in human cervical carcinoma cell lines. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[3] T. Crook,et al. p53 point mutation in HPV negative human cervical carcinoma cell lines. , 1991, Oncogene.
[4] A. Murray,et al. Cyclin is degraded by the ubiquitin pathway , 1991, Nature.
[5] D. Lowy,et al. In vitro biological activities of the E6 and E7 genes vary among human papillomaviruses of different oncogenic potential , 1991, Journal of virology.
[6] A Ciechanover,et al. Degradation of nuclear oncoproteins by the ubiquitin system in vitro. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[7] Arnold J. Levine,et al. The E6 oncoprotein encoded by human papillomavirus types 16 and 18 promotes the degradation of p53 , 1990, Cell.
[8] M. Green,et al. Chemical synthesis of human papillomavirus type 16 E7 oncoprotein: autonomous protein domains for induction of cellular DNA synthesis and for trans activation , 1990, Journal of virology.
[9] K. Vousden. Human papillomavirus oncoproteins. , 1990, Seminars in cancer biology.
[10] B. Vogelstein,et al. p53 functions as a cell cycle control protein in osteosarcomas , 1990, Molecular and cellular biology.
[11] S. Fields,et al. Presence of a potent transcription activating sequence in the p53 protein. , 1990, Science.
[12] G. Lozano,et al. Transcriptional activation by wild-type but not transforming mutants of the p53 anti-oncogene. , 1990, Science.
[13] B. Vogelstein,et al. Suppression of human colorectal carcinoma cell growth by wild-type p53. , 1990, Science.
[14] E. Appella,et al. Negative growth regulation in a glioblastoma tumor cell line that conditionally expresses human wild-type p53. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[15] E. Androphy,et al. Transcriptional activation by the papillomavirus E6 zinc finger oncoprotein. , 1990, The EMBO journal.
[16] A. Levine,et al. Association of human papillomavirus types 16 and 18 E6 proteins with p53. , 1990, Science.
[17] D. Lowy,et al. The region of the HPV E7 oncoprotein homologous to adenovirus E1a and Sv40 large T antigen contains separate domains for Rb binding and casein kinase II phosphorylation. , 1990, The EMBO journal.
[18] D. Lowy,et al. HPV16 E6 and E7 proteins cooperate to immortalize human foreskin keratinocytes. , 1989, The EMBO journal.
[19] K. Münger,et al. Complex formation of human papillomavirus E7 proteins with the retinoblastoma tumor suppressor gene product. , 1989, The EMBO journal.
[20] E. Villiers. Heterogeneity of the human papillomavirus group. , 1989 .
[21] V. Kh,et al. Human papillomaviruses and cervical carcinoma. , 1989 .
[22] R. Schlegel,et al. The E6 and E7 genes of the human papillomavirus type 16 together are necessary and sufficient for transformation of primary human keratinocytes , 1989, Journal of virology.
[23] A. Levine,et al. The p53 proto-oncogene can act as a suppressor of transformation , 1989, Cell.
[24] K. Vousden,et al. A point mutational analysis of human papillomavirus type 16 E7 protein , 1989, Journal of virology.
[25] D. Lowy,et al. Papillomavirus polypeptides E6 and E7 are zinc-binding proteins , 1989, Journal of virology.
[26] S. Grossman,et al. Identification of human papillomavirus type 18 transforming genes in immortalized and primary cells , 1989, Journal of virology.
[27] K. Münger,et al. The human papilloma virus-16 E7 oncoprotein is able to bind to the retinoblastoma gene product. , 1989, Science.
[28] C. Woodworth,et al. Immortalization of human foreskin keratinocytes by various human papillomavirus DNAs corresponds to their association with cervical carcinoma , 1989, Journal of virology.
[29] P. L. Chen,et al. Suppression of the neoplastic phenotype by replacement of the RB gene in human cancer cells. , 1988, Science.
[30] D. Lowy,et al. The E7 open reading frame of human papillomavirus type 16 encodes a transforming gene. , 1988, Oncogene research.
[31] Wen-Hwa Lee,et al. SV40 large tumor antigen forms a specific complex with the product of the retinoblastoma susceptibility gene , 1988, Cell.
[32] Stephen H. Friend,et al. Association between an oncogene and an anti-oncogene: the adenovirus E1A proteins bind to the retinoblastoma gene product , 1988, Nature.
[33] D. Pim,et al. Comparison of the in vitro transforming activities of human papillomavirus types. , 1988, The EMBO journal.
[34] K. Münger,et al. The human papillomavirus type 16 E7 gene encodes transactivation and transformation functions similar to those of adenovirus E1A , 1988, Cell.
[35] J. Trowsdale,et al. Analysis of HLA-DR glycoproteins by DNA-mediated gene transfer. Definition of DR2 beta gene products and antigen presentation to T cell clones from leprosy patients , 1988, The Journal of experimental medicine.
[36] S. Grossman,et al. Inducible and constitutive enhancer domains in the noncoding region of human papillomavirus type 18 , 1988, Journal of virology.
[37] T. Kanda,et al. Human papillomavirus type 16 open reading frame E7 encodes a transforming gene for rat 3Y1 cells , 1988, Journal of virology.
[38] D. Pim,et al. Primary structure polymorphism at amino acid residue 72 of human p53 , 1987, Molecular and cellular biology.
[39] L. Banks,et al. Isolation of human-p53-specific monoclonal antibodies and their use in the studies of human p53 expression. , 1986, European journal of biochemistry.
[40] A. Schneider-Gädicke,et al. Different human cervical carcinoma cell lines show similar transcription patterns of human papillomavirus type 18 early genes. , 1986, The EMBO journal.
[41] J. Yewdell,et al. Monoclonal antibody analysis of p53 expression in normal and transformed cells , 1986, Journal of virology.
[42] F. Wettstein,et al. Transcription of human papillomavirus type 16 early genes in a cervical cancer and a cancer-derived cell line and identification of the E7 protein. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[43] A. Faras,et al. The nucleotide sequence and genome organization of human papilloma virus type 11. , 1986 .
[44] L. Gissmann,et al. The nucleotide sequence and genome organization of human papilloma virus type 11. , 1986, Virology.
[45] D. Hazuda,et al. Xenopus transcription factor A requires zinc for binding to the 5 S RNA gene. , 1983, The Journal of biological chemistry.
[46] M. Dürst,et al. DNA sequence and genome organization of genital human papillomavirus type 6b. , 1983, The EMBO journal.
[47] G. Ringold,et al. Expression and regulation of Escherichia coli lacZ gene fusions in mammalian cells. , 1983, Journal of molecular and applied genetics.
[48] B. Howard,et al. Recombinant genomes which express chloramphenicol acetyltransferase in mammalian cells , 1982, Molecular and cellular biology.
[49] A. Levine,et al. Adenovirus E1b-58kd tumor antigen and SV40 large tumor antigen are physically associated with the same 54 kd cellular protein in transformed cells , 1982, Cell.
[50] D. Pim,et al. Monoclonal antibodies specific for simian virus 40 tumor antigens , 1981, Journal of virology.
[51] D. Lane,et al. T antigen is bound to a host protein in SY40-transformed cells , 1979, Nature.
[52] Jeffrey H. Miller. Experiments in molecular genetics , 1972 .