METTL16 drives leukemogenesis and leukemia stem cell self-renewal by reprogramming BCAA metabolism.
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Jingling Xue | M. Wei | G. Marcucci | M. Wunderlich | J. Mulloy | Lei Dong | Bin Zhang | Chao Shen | R. Su | Min Gao | Xiaolan Deng | Y. Qing | Xi Qin | Zhicong Zhao | Wei Li | C. Chen | Lu Yang | Lei Gao | Zhenglai Zhang | Li Han | S. Pokharel | B. Tan | K. Wang | Zhenhua Chen | Andrew Small | Yangchan Li | Jianjun Chen | Keith Leung | Meiling Chen
[1] M. Wei,et al. The m6A reader IGF2BP2 regulates glutamine metabolism and represents a therapeutic target in acute myeloid leukemia. , 2022, Cancer cell.
[2] Wei Liu,et al. METTL16 exerts an m6A-independent function to facilitate translation and tumorigenesis , 2022, Nature Cell Biology.
[3] J. Eirín-López,et al. Evolution of Methyltransferase-Like (METTL) Proteins in Metazoa: A Complex Gene Family Involved in Epitranscriptomic Regulation and Other Epigenetic Processes , 2021, Molecular biology and evolution.
[4] N. Chandel,et al. Cancer metabolism: looking forward , 2021, Nature Reviews Cancer.
[5] Chuan He,et al. A Critical Role of Nuclear m6A Reader YTHDC1 in Leukemogenesis by Regulating MCM Complex-Mediated DNA Replication. , 2021, Blood.
[6] Jianjun Chen,et al. RNA modifications in hematopoietic malignancies: A new research frontier. , 2021, Blood.
[7] D. Patel,et al. N6-Methyladenosine on mRNA facilitates a phase-separated nuclear body that suppresses myeloid leukemic differentiation. , 2021, Cancer cell.
[8] Andrew J. Bannister,et al. Small-molecule inhibition of METTL3 as a strategy against myeloid leukaemia , 2021, Nature.
[9] Amanda E. Jones,et al. Silencing of LINE-1 retrotransposons is a selective dependency of myeloid leukemia , 2021, Nature Genetics.
[10] Lei Jiang,et al. R-2-hydroxyglutarate attenuates aerobic glycolysis in leukemia by targeting the FTO/m6A/PFKP/LDHB axis. , 2021, Molecular cell.
[11] Yue Huang,et al. Targeting FTO Suppresses Cancer Stem Cell Maintenance and Immune Evasion. , 2020, Cancer cell.
[12] Chuan He,et al. RNA Demethylase ALKBH5 Selectively Promotes Tumorigenesis and Cancer Stem Cell Self-Renewal in Acute Myeloid Leukemia. , 2020, Cell stem cell.
[13] Shaoguang Li,et al. Leukemogenic Chromatin Alterations Promote AML Leukemia Stem Cells via a KDM4C-ALKBH5-AXL Signaling Axis. , 2020, Cell stem cell.
[14] S. Yamashita,et al. Mechanistic insights into m6A modification of U6 snRNA by human METTL16 , 2020, Nucleic acids research.
[15] Jianjun Chen,et al. RNA Modifications in Cancer: Functions, Mechanisms, and Therapeutic Implications , 2020, Annual Review of Cancer Biology.
[16] Jianjun Chen,et al. m6A Modification in Coding and Non-coding RNAs: Roles and Therapeutic Implications in Cancer. , 2020, Cancer cell.
[17] Ji Ae Song,et al. Glucose-dependent control of leucine metabolism by leucyl-tRNA synthetase 1 , 2019, Science.
[18] Michael G. Kharas,et al. m6A RNA Methylation Maintains Hematopoietic Stem Cell Identity and Symmetric Commitment , 2019, Cell reports.
[19] D. O’Carroll,et al. Targeting the RNA m6A Reader YTHDF2 Selectively Compromises Cancer Stem Cells in Acute Myeloid Leukemia , 2019, Cell stem cell.
[20] T. Pulinilkunnil,et al. Role of branched‐chain amino acid–catabolizing enzymes in intertissue signaling, metabolic remodeling, and energy homeostasis , 2019, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[21] Lei Ding,et al. Stage-specific requirement for Mettl3-dependent m6A mRNA methylation during haematopoietic stem cell differentiation , 2019, Nature Cell Biology.
[22] C. Winter,et al. A reference collection of patient-derived cell line and xenograft models of proneural, classical and mesenchymal glioblastoma , 2019, Scientific Reports.
[23] R. Deberardinis,et al. Loss of EZH2 Reprograms BCAA Metabolism to Drive Leukemic Transformation. , 2019, Cancer discovery.
[24] Ping-yuan Wang,et al. Structural Basis for Regulation of METTL16, an S-Adenosylmethionine Homeostasis Factor. , 2018, Molecular cell.
[25] A. McCarthy,et al. Methylation of Structured RNA by the m6A Writer METTL16 Is Essential for Mouse Embryonic Development , 2018, Molecular cell.
[26] Wengong Wang,et al. Loss of YTHDF2-mediated m6A-dependent mRNA clearance facilitates hematopoietic stem cell regeneration , 2018, Cell Research.
[27] Xi C. He,et al. Suppression of m6A reader Ythdf2 promotes hematopoietic stem cell expansion , 2018, Cell Research.
[28] Wen-jie Dong,et al. Mettl3–Mettl14 methyltransferase complex regulates the quiescence of adult hematopoietic stem cells , 2018, Cell Research.
[29] Jianjun Chen,et al. RNA N6-methyladenosine modification in cancers: current status and perspectives , 2018, Cell Research.
[30] Stefan Hüttelmaier,et al. Recognition of RNA N6-methyladenosine by IGF2BP Proteins Enhances mRNA Stability and Translation , 2018, Nature Cell Biology.
[31] James E. Bradner,et al. R-2HG Exhibits Anti-tumor Activity by Targeting FTO/m6A/MYC/CEBPA Signaling , 2018, Cell.
[32] Yue Sheng,et al. METTL14 Inhibits Hematopoietic Stem/Progenitor Differentiation and Promotes Leukemogenesis via mRNA m6A Modification. , 2017, Cell stem cell.
[33] Tsutomu Suzuki,et al. S-Adenosylmethionine Synthesis Is Regulated by Selective N6-Adenosine Methylation and mRNA Degradation Involving METTL16 and YTHDC1. , 2017, Cell reports.
[34] Junwei Shi,et al. Promoter-bound METTL3 maintains myeloid leukaemia by m6A-dependent translation control , 2017, Nature.
[35] S. Fröhling,et al. BCAT1 restricts αKG levels in AML stem cells leading to IDHmut-like DNA hypermethylation , 2017, Nature.
[36] Henning Urlaub,et al. Human METTL16 is a N6‐methyladenosine (m6A) methyltransferase that targets pre‐mRNAs and various non‐coding RNAs , 2017, EMBO reports.
[37] F. Liu,et al. m6A modulates haematopoietic stem and progenitor cell specification , 2017, Nature.
[38] Francine E. Garrett-Bakelman,et al. The N6-methyladenosine (m6A)-forming enzyme METTL3 controls myeloid differentiation of normal and leukemia cells , 2017, Nature Medicine.
[39] Phillip G. Montgomery,et al. Defining a Cancer Dependency Map , 2017, Cell.
[40] Yang Xie,et al. The U6 snRNA m6A Methyltransferase METTL16 Regulates SAM Synthetase Intron Retention , 2017, Cell.
[41] N. Kannan,et al. Cancer progression by reprogrammed BCAA metabolism in myeloid leukemia , 2017, Nature.
[42] D. Pollyea,et al. Therapeutic targeting of acute myeloid leukemia stem cells. , 2017, Blood.
[43] Jie Jin,et al. FTO Plays an Oncogenic Role in Acute Myeloid Leukemia as a N6-Methyladenosine RNA Demethylase. , 2017, Cancer cell.
[44] J. Steitz,et al. Methyltransferase-like protein 16 binds the 3′-terminal triple helix of MALAT1 long noncoding RNA , 2016, Proceedings of the National Academy of Sciences.
[45] Jiwang Zhang,et al. miR-22 has a potent anti-tumour role with therapeutic potential in acute myeloid leukaemia , 2016, Nature Communications.
[46] Meagan E. Sullender,et al. Optimized sgRNA design to maximize activity and minimize off-target effects of CRISPR-Cas9 , 2015, Nature Biotechnology.
[47] R. Deberardinis,et al. Metabolic pathways promoting cancer cell survival and growth , 2015, Nature Cell Biology.
[48] Daniel J Weisdorf,et al. Acute Myeloid Leukemia. , 2015, The New England journal of medicine.
[49] Neville E. Sanjana,et al. Improved vectors and genome-wide libraries for CRISPR screening , 2014, Nature Methods.
[50] G E Tomlinson,et al. WTAP is a novel oncogenic protein in acute myeloid leukemia , 2014, Leukemia.
[51] Hunter N. B. Moseley,et al. A Computational Framework for High-Throughput Isotopic Natural Abundance Correction of Omics-Level Ultra-High Resolution FT-MS Datasets , 2013, Metabolites.
[52] Rainer König,et al. BCAT1 promotes cell proliferation through amino acid catabolism in gliomas carrying wild-type IDH1 , 2013, Nature Medicine.
[53] Y. Saunthararajah,et al. AML cells are differentially sensitive to chemotherapy treatment in a human xenograft model. , 2013, Blood.
[54] Thomas R. Gingeras,et al. STAR: ultrafast universal RNA-seq aligner , 2013, Bioinform..
[55] P. Ward,et al. Metabolic reprogramming: a cancer hallmark even warburg did not anticipate. , 2012, Cancer cell.
[56] Colin N. Dewey,et al. RSEM: accurate transcript quantification from RNA-Seq data with or without a reference genome , 2011, BMC Bioinformatics.
[57] S. Clarke,et al. Uncovering the Human Methyltransferasome* , 2010, Molecular & Cellular Proteomics.
[58] J. Rowley,et al. Leukaemogenesis: more than mutant genes , 2010, Nature Reviews Cancer.
[59] Hunter N. B. Moseley,et al. Correcting for the effects of natural abundance in stable isotope resolved metabolomics experiments involving ultra-high resolution mass spectrometry , 2010, BMC Bioinformatics.
[60] P. Yaswen,et al. A Versatile Viral System for Expression and Depletion of Proteins in Mammalian Cells , 2009, PloS one.
[61] S. Armstrong,et al. Therapeutic Implications of Leukemia Stem Cell Development , 2007, Clinical Cancer Research.
[62] M. Cleary,et al. Identification and characterization of leukemia stem cells in murine MLL-AF9 acute myeloid leukemia. , 2006, Cancer cell.
[63] T. Golub,et al. Transformation from committed progenitor to leukaemia stem cell initiated by MLL–AF9 , 2006, Nature.
[64] Ming Yan,et al. A previously unidentified alternatively spliced isoform of t(8;21) transcript promotes leukemogenesis , 2006, Nature Medicine.
[65] M. Brosnan,et al. Branched-chain amino acids: enzyme and substrate regulation. , 2006, The Journal of nutrition.
[66] Pablo Tamayo,et al. Gene set enrichment analysis: A knowledge-based approach for interpreting genome-wide expression profiles , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[67] J. Dick,et al. Acute myeloid leukemia originates from a hierarchy of leukemic stem cell classes that differ in self-renewal capacity , 2004, Nature Immunology.
[68] William C Hahn,et al. Lentivirus-delivered stable gene silencing by RNAi in primary cells. , 2003, RNA.
[69] R. Naviaux,et al. The pCL vector system: rapid production of helper-free, high-titer, recombinant retroviruses , 1996, Journal of virology.
[70] R. Miller,et al. Branched-chain amino acid metabolism. , 1984, Annual review of nutrition.