MRI Demonstrates that Tissue-Type Plasminogen Activator Increases Stroke Volume if Cerebral Arteries Are not Successfully Recanalized

[1]  C. Beaulieu,et al.  Polynitroxyl Albumin Reduces Infarct Size in Transient Focal Cerebral Ischemia in the Rat: Potential Mechanisms Studied by Magnetic Resonance Imaging , 1998, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[2]  Christian M. Kerskens,et al.  Reperfusion after Thrombolytic Therapy of Embolic Stroke in the Rat: Magnetic Resonance and Biochemical Imaging , 1998, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[3]  S. Lipton,et al.  Tissue plasminogen activator (tPA) increase neuronal damage after focal cerebral ischemia in wild-type and tPA-deficient mice , 1998, Nature Medicine.

[4]  S. Strickland,et al.  Neuronal Death in the Hippocampus Is Promoted by Plasmin-Catalyzed Degradation of Laminin , 1997, Cell.

[5]  K. Hossmann,et al.  Improved model of thromboembolic stroke and rt-PA induced reperfusion in the rat , 1997, Brain Research.

[6]  S. Tsirka.  Clinical implications of the involvement of tPA in neuronal cell death , 1997, Journal of Molecular Medicine.

[7]  S. Tsirka,et al.  Neuronal cell death and tPA , 1996, Nature.

[8]  Sidney Strickland,et al.  Excitotoxin-induced neuronal degeneration and seizure are mediated by tissue plasminogen activator , 1995, Nature.

[9]  K. Hossmann,et al.  Diffusion nuclear magnetic resonance imaging in experimental stroke. Correlation with cerebral metabolites. , 1994, Stroke.

[10]  C. Korninger,et al.  Studies on the Specific Fibrinolytic Effect of Human Extrinsic (Tissue-Type) Plasminogen Activator in Human Blood and in Various Animal Species in Vitro , 1981, Thrombosis and Haemostasis.