The F286Y mutation of PrlA4 tempers the signal sequence suppressor phenotype by reducing the SecA binding affinity
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[1] A. Driessen,et al. Mapping the Sites of Interaction between SecY and SecE by Cysteine Scanning Mutagenesis* , 2001, The Journal of Biological Chemistry.
[2] A. Driessen,et al. The structural basis of protein targeting and translocation in bacteria , 2001, Nature Structural Biology.
[3] T. Rapoport,et al. Projection structure and oligomeric properties of a bacterial core protein translocase , 2001, The EMBO journal.
[4] A. Engel,et al. SecYEG assembles into a tetramer to form the active protein translocation channel , 2000, The EMBO journal.
[5] A. Driessen,et al. Preprotein Translocation by a Hybrid Translocase Composed of Escherichia coli and Bacillus subtilis Subunits , 1999, Journal of bacteriology.
[6] A. Driessen,et al. Cysteine-directed cross-linking demonstrates that helix 3 of SecE is close to helix 2 of SecY and helix 3 of a neighboring SecE. , 1999, Biochemistry.
[7] W. Wickner,et al. The PrlA and PrlG phenotypes are caused by a loosened association among the translocase SecYEG subunits , 1999, The EMBO journal.
[8] K. Nishiyama,et al. Membrane deinsertion of SecA underlying proton motive force‐dependent stimulation of protein translocation , 1999, The EMBO journal.
[9] T. Silhavy,et al. PrlA4 prevents the rejection of signal sequence defective preproteins by stabilizing the SecA–SecY interaction during the initiation of translocation , 1998, The EMBO journal.
[10] A. Driessen,et al. Interaction between SecA and SecYEG in micellar solution and formation of the membrane-inserted state. , 1998, Biochemistry.
[11] A. Driessen,et al. The catalytic cycle of the Escherichia coli SecA ATPase comprises two distinct preprotein translocation events , 1997, The EMBO journal.
[12] D. Belin,et al. prl Mutations in the Escherichia coli secG Gene* , 1997, The Journal of Biological Chemistry.
[13] J. Beckwith,et al. Targeting of signal sequenceless proteins for export in Escherichia coli with altered protein translocase. , 1996, The EMBO journal.
[14] J. Barbero,et al. Different PrlA proteins increase the efficiency of periplasmic production of human interleukin-6 in Escherichia coli. , 1996, Journal of biotechnology.
[15] J. Tommassen,et al. prlA suppressors in Escherichia coli relieve the proton electrochemical gradient dependency of translocation of wild-type precursors. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[16] T. Silhavy,et al. Suppression of signal sequence defects and azide resistance in Escherichia coli commonly result from the same mutations in secA , 1995, Journal of bacteriology.
[17] W. Wickner,et al. SecA promotes preprotein translocation by undergoing ATP-driven cycles of membrane insertion and deinsertion , 1994, Cell.
[18] R. Doebele,et al. PrlA and PrlG suppressors reduce the requirement for signal sequence recognition , 1994, Journal of bacteriology.
[19] K. Nishiyama,et al. A novel membrane protein involved in protein translocation across the cytoplasmic membrane of Escherichia coli. , 1993, The EMBO journal.
[20] R. S. Osborne,et al. PrlA suppressor mutations cluster in regions corresponding to three distinct topological domains. , 1993, The EMBO journal.
[21] J. Beckwith,et al. A signal sequence is not required for protein export in prlA mutants of Escherichia coli. , 1993, The EMBO journal.
[22] A. Driessen,et al. Precursor protein translocation by the Escherichia coli translocase is directed by the protonmotive force. , 1992, The EMBO journal.
[23] W. Wickner,et al. SecY, SecE, and band 1 form the membrane-embedded domain of Escherichia coli preprotein translocase. , 1992, The Journal of biological chemistry.
[24] J. Hendrick,et al. SecA protein needs both acidic phospholipids and SecY/E protein for functional high-affinity binding to the Escherichia coli plasma membrane. , 1991, The Journal of biological chemistry.
[25] S. Mizushima,et al. Reconstitution of a protein translocation system containing purified SecY, SecE, and SecA from Escherichia coli. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[26] Arnold J. M. Driessen,et al. Δμ H+ and ATP function at different steps of the catalytic cycle of preprotein translocase , 1991, Cell.
[27] M. Inouye,et al. Effect of OmpA signal peptide mutations on OmpA secretion, synthesis, and assembly , 1991, Journal of bacteriology.
[28] F. Hartl,et al. The binding cascade of SecB to SecA to SecY E mediates preprotein targeting to the E. coli plasma membrane , 1990, Cell.
[29] K. Bieker,et al. Thesec andprl genes ofEscherichia coli , 1990, Journal of bioenergetics and biomembranes.
[30] W. Wickner,et al. The ATPase activity of secA is regulated by acidic phospholipids, secY, and the leader and mature domains of precursor proteins , 1990, Cell.
[31] W. Wickner,et al. Specific recognition of the leader region of precursor proteins is required for the activation of translocation ATPase of Escherichia coli. , 1989, Proceedings of the National Academy of Sciences of the United States of America.
[32] L. Gansheroff,et al. New suppressors of signal-sequence mutations, prlG, are linked tightly to the secE gene of Escherichia coli. , 1989, Genes & development.
[33] W. Wickner,et al. SecA protein, a peripheral protein of the Escherichia coli plasma membrane, is essential for the functional binding and translocation of proOmpA. , 1989, The EMBO journal.
[34] Koreaki Ito,et al. SecA protein hydrolyzes ATP and is an essential component of the protein translocation ATPase of Escherichia coli. , 1989, The EMBO journal.
[35] J. Fikes,et al. Novel secA alleles improve export of maltose-binding protein synthesized with a defective signal peptide , 1989, Journal of bacteriology.
[36] T. Sako,et al. Distinct mutation sites in prlA suppressor mutant strains of Escherichia coli respond either to suppression of signal peptide mutations or to blockage of staphylokinase processing , 1988, Journal of bacteriology.
[37] S. Emr,et al. Suppressor mutations that restore export of a protein with a defective signal sequence , 1981, Cell.
[38] A. Driessen,et al. University of Groningen A single amino acid substitution in SecY stabilizes the interaction with SecA , 1999 .