Serum soluble TWEAK levels in severe traumatic brain injury and its prognostic significance.
暂无分享,去创建一个
Ze Zhong | Bei Tang | Hui-Ping Wu | Jianping Ma | Zheng Qiu | Jiayuan Hu | Jin-Ping Wu
[1] V. Felipo,et al. Peripheral inflammation induces neuroinflammation that alters neurotransmission and cognitive and motor function in hepatic encephalopathy: Underlying mechanisms and therapeutic implications , 2019, Acta physiologica.
[2] Guo-yi Gao,et al. Traumatic brain injury in China , 2019, The Lancet Neurology.
[3] S. S. Fatima,et al. Evaluation of soluble TNF-like weak inducer of apoptosis (sTWEAK) levels to predict preeclampsia in early weeks of pregnancy. , 2019, European journal of obstetrics, gynecology, and reproductive biology.
[4] J. Ranjeva,et al. High levels of serum soluble TWEAK are associated with neuroinflammation during multiple sclerosis , 2019, Journal of Translational Medicine.
[5] A. Nagel,et al. Potential of Sodium MRI as a Biomarker for Neurodegeneration and Neuroinflammation in Multiple Sclerosis , 2019, Front. Neurol..
[6] K. Nash,et al. Neuroinflammation and fractalkine signaling in Alzheimer’s disease , 2019, Journal of Neuroinflammation.
[7] Seyed A Safavynia,et al. The Role of Neuroinflammation in Postoperative Cognitive Dysfunction: Moving From Hypothesis to Treatment , 2019, Front. Psychiatry.
[8] Jianwei Pan,et al. Serum ST2 as a potential prognostic biomarker for traumatic brain injury. , 2018, Clinica chimica acta; international journal of clinical chemistry.
[9] C. Ryalino,et al. Modified Revised Trauma–Marshall score as a proposed tool in predicting the outcome of moderate and severe traumatic brain injury , 2018, Open access emergency medicine : OAEM.
[10] M. V. van Es,et al. Outcome in Patients with Isolated Moderate to Severe Traumatic Brain Injury , 2018, Critical care research and practice.
[11] M. Drutskaya,et al. Cytokines as Mediators of Neuroinflammation in Experimental Autoimmune Encephalomyelitis , 2018, Biochemistry (Moscow).
[12] Raghavendra Nayak,et al. Serum Magnesium as a Marker of Neurological Outcome in Severe Traumatic Brain Injury Patients , 2018, Asian journal of neurosurgery.
[13] M. Chopp,et al. Current understanding of neuroinflammation after traumatic brain injury and cell-based therapeutic opportunities , 2018, Chinese journal of traumatology = Zhonghua chuang shang za zhi.
[14] T. V. van Essen,et al. Decision making in very severe traumatic brain injury (Glasgow Coma Scale 3-5): a literature review of acute neurosurgical management. , 2018, Journal of neurosurgical sciences.
[15] Shu-Yan Liu,et al. Clinical significance of serum interleukin‐8 and soluble tumor necrosis factor‐like weak inducer of apoptosis levels in patients with diabetic nephropathy , 2018, Journal of diabetes investigation.
[16] Hester F. Lingsma,et al. Differential effects of the Glasgow Coma Scale Score and its Components: An analysis of 54,069 patients with traumatic brain injury. , 2017, Injury.
[17] S. Acosta,et al. Neuroinflammation in traumatic brain injury: A chronic response to an acute injury , 2017, Brain circulation.
[18] Saurabh Sharma,et al. Recent Advances in Pathophysiology of Traumatic Brain Injury , 2017, Current neuropharmacology.
[19] N. Greig,et al. Neuroinflammation in animal models of traumatic brain injury , 2016, Journal of Neuroscience Methods.
[20] M. Schwartz,et al. TNF-like weak inducer of apoptosis promotes blood brain barrier disruption and increases neuronal cell death in MRL/lpr mice. , 2015, Journal of autoimmunity.
[21] Dingbo Yang,et al. Plasma copeptin level predicts acute traumatic coagulopathy and progressive hemorrhagic injury after traumatic brain injury , 2014, Peptides.
[22] J. Rysä,et al. Thrombospondin-4, tumour necrosis factor-like weak inducer of apoptosis (TWEAK) and its receptor Fn14: Novel extracellular matrix modulating factors in cardiac remodelling , 2012, Annals of medicine.
[23] M. Ren,et al. The role of TWEAK/Fn14 in cardiac remodeling , 2012, Molecular Biology Reports.
[24] Qiao-ling Zhou,et al. Tumor necrosis factor-like weak inducer of apoptosis and its potential roles in lupus nephritis , 2012, Inflammation Research.
[25] M. Sánchez-Niño,et al. TWEAK, a multifunctional cytokine in kidney injury. , 2011, Kidney international.
[26] C. Sommer,et al. Induction of the cytokine TWEAK and its receptor Fn14 in ischemic stroke , 2008, Journal of the Neurological Sciences.
[27] M. Yepes. TWEAK and the Central Nervous System , 2007, Molecular Neurobiology.
[28] M. Yepes. Tweak and FN14 in central nervous system health and disease. , 2007, Frontiers in bioscience : a journal and virtual library.
[29] L. Burkly,et al. TWEAK—Fn14 Pathway Inhibition Protects the Integrity of the Neurovascular Unit during Cerebral Ischemia , 2007, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[30] M. Yepes,et al. Inhibition of TWEAK activity as a new treatment for inflammatory and degenerative diseases. , 2006, Drug news & perspectives.
[31] M. Berens,et al. TWEAK and Fn14: new molecular targets for cancer therapy? , 2006, Cancer letters.
[32] M. T. Hasan,et al. IKK mediates ischemia-induced neuronal death , 2005, Nature Medicine.
[33] D. Lawrence,et al. A soluble Fn14-Fc decoy receptor reduces infarct volume in a murine model of cerebral ischemia. , 2005, The American journal of pathology.
[34] J. Winkles,et al. TWEAK, a member of the TNF superfamily, is a multifunctional cytokine that binds the TweakR/Fn14 receptor. , 2003, Cytokine & growth factor reviews.
[35] Dingbo Yang,et al. Prognostic significance of serum translocator protein in patients with traumatic brain injury. , 2019, Clinica chimica acta; international journal of clinical chemistry.
[36] Ş. Arslan,et al. Soluble Tumor Necrosis Factor (TNF)-Like Weak Inducer of Apoptosis (Tweak) Independently Predicts Subclinical Atherosclerosis in Behcet's Disease. , 2018, Acta medica.