Heavy metal localization and age related accumulation in the rat nervous system

[1]  G. Voigt,et al.  Histochemische Untersuchungen über den Verbleib des Quecksilbers bei der experimentellen Sublimatvergiftung , 2005, Klinische Wochenschrift.

[2]  K. Fleischhauer,et al.  Intravitale Dithizonfärbung homologer Felder der Ammonsformation von Säugern , 2004, Zeitschrift für Zellforschung und Mikroskopische Anatomie.

[3]  A. Brun,et al.  The effect of aging on lysosomal permeability in nerve cells of the central nervous system. An enzyme histochemical study in rat , 2004, Histochemie.

[4]  A. Brun,et al.  Histochemical evidence for lysosomal uptake of lead in tissue cultured fibroblasts , 2004, Histochemie.

[5]  F. Timm,et al.  Zur Histochemie der Schwermetalle Das Sulfid-Silberverfahren , 2004, Deutsche Zeitschrift für die gesamte gerichtliche Medizin.

[6]  T. Slater.  Free-radical mechanisms in tissue injury. , 1984, The Biochemical journal.

[7]  H. A. Johnson,et al.  Neuron survival in the aging mouse. , 1972, Experimental gerontology.

[8]  U. Brunk,et al.  Electron microscopical studies on rat brain neurons. Localization of acid phosphatase and mode of formation of lipofuscin bodies. , 1972, Journal of ultrastructure research.

[9]  A. Brun,et al.  THE JOURNAL OF HISTOCHEMISTRY AND CYTOCHEMISTRY , 2005 .

[10]  Lindquist Rr.  Studies on the pathogenesis of hepatolenticular degeneration. 3. The effect of copper on rat liver lysosomes. , 1968 .

[11]  S. E. Toth.  The origin of lipofuscin age pigments. , 1968, Experimental gerontology.

[12]  A. Brun,et al.  Histochemical demonstration of heavy metals with the sulfide-silver method. A methodological study. , 1968, Acta histochemica.

[13]  A. Brun,et al.  Histochemical studies on brain phosphatases in experimental lead poisoning. , 2009, Acta pathologica et microbiologica Scandinavica.

[14]  Lindquist Rr.  Studies on the pathogenesis of hepatolenticular degeneration. I. Acid phosphatase activity in copper-loaded rat livers. , 1967 .

[15]  W. J. Brown,et al.  Subcellular distribution and enzyme changes following subacute copper intoxication. , 1967, Laboratory investigation; a journal of technical methods and pathology.

[16]  R. Lindquist.  Studies on the pathogenesis of hepatolenticular degeneration. I. Acid phosphatase activity in copper-loaded rat livers. , 1967, The American journal of pathology.

[17]  N. Kaneta.  Histochemical studies on the diencephalon of senescent rats. , 1966, The Tohoku journal of experimental medicine.

[18]  M. Kraft,et al.  Zur histochemie und genese der chronischen quecksilbervergiftung , 1966 .

[19]  S. Goldfischer,et al.  Electron probe microanalysis of liver in Wilson's disease. Simultaneous assay for copper and for lead deposited by acid phosphatase activity in lysosomes. , 1966, The American journal of pathology.

[20]  S. Goldfischer.  THE LOCALIZATION OF COPPER IN THE PERICANALICULAR GRANULES (LYSOSOMES) OF LIVER IN WILSON'S DISEASE (HEPATOLENTICULAR DEGENERATION) . , 1965, The American journal of pathology.

[21]  J. Worm‐Petersen.  VITAMIN B12, HAEMOGLOBIN AND IRON CONCENTRATION IN HUMAN BRAIN TISSUE , 1964, Acta neurologica Scandinavica.

[22]  G. Voigt,et al.  [Histochemical studies on the distribution of mercury in experimental mercury poisoning]. , 1963, Klinische Wochenschrift.

[23]  H. Bertha,et al.  Untersuchungen über die regionale Kationenverteilung im menschlichen Gehirn , 1962 .

[24]  B. Hallgren,et al.  THE EFFECT OF AGE ON THE NON‐HAEMIN IRON IN THE HUMAN BRAIN , 1958, Journal of neurochemistry.

[25]  K. Fleischhauer,et al.  [Intravital dithizone staining of homologous fields of the Ammon's formation in mammals]. , 1957, Zeitschrift fur Zellforschung und mikroskopische Anatomie.

[26]  J. Cumings.  The copper and iron content of brain and liver in the normal and in hepato-lenticular degeneration. , 1948, Brain : a journal of neurology.

[27]  N. Lewis.  Suggestive research leads in contemporary neurochemistry. , 1948, Journal of the American Medical Association.