Studying schizophrenia in the post-genomic era: perspectives from the 2016 summer Banbury Workshop at Cold Spring Harbor Laboratory
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[1] Giulio Genovese,et al. Schizophrenia risk from complex variation of complement component 4 , 2016, Nature.
[2] Tyrone D. Cannon,et al. Association of Thalamic Dysconnectivity and Conversion to Psychosis in Youth and Young Adults at Elevated Clinical Risk. , 2015, JAMA psychiatry.
[3] A. Malhotra,et al. Baseline Striatal Functional Connectivity as a Predictor of Response to Antipsychotic Drug Treatment. , 2015, The American journal of psychiatry.
[4] M. Owen,et al. Genetic Risk for Schizophrenia: Convergence on Synaptic Pathways Involved in Plasticity , 2015, Biological Psychiatry.
[5] C. Spencer,et al. Biological Insights From 108 Schizophrenia-Associated Genetic Loci , 2014, Nature.
[6] D. Butler. Translational research: Crossing the valley of death , 2008, Nature.
[7] R. V. Van Heertum,et al. Increased baseline occupancy of D2 receptors by dopamine in schizophrenia. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[8] Tyrone D. Cannon,et al. Prediction of psychosis in youth at high clinical risk: a multisite longitudinal study in North America. , 2008, Archives of general psychiatry.