EGCG, GCG, TFDG, or TSA Inhibiting Melanin Synthesis by Downregulating MC1R Expression
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[1] Huaping Li,et al. Effects of tea polyphenols on UVA-induced melanogenesis via inhibition of α-MSH-MC1R signalling pathway , 2022, Postepy dermatologii i alergologii.
[2] U. Engelhardt,et al. Effect of Active Groups and Oxidative Dimerization on the Antimelanogenic Activity of Catechins and Their Dimeric Oxidation Products. , 2022, Journal of Agricultural and Food Chemistry.
[3] S. R. Kopalli,et al. Antimelanogenesis Effects of Theasinensin A , 2021, International journal of molecular sciences.
[4] H. Haenssle,et al. From Melanocytes to Melanoma Cells: Characterization of the Malignant Transformation by Four Distinctly Different Melanin Fluorescence Spectra (Review) † , 2021, International journal of molecular sciences.
[5] Zhigang Wang,et al. Visualized podocyte-targeting and focused ultrasound responsive glucocorticoid nano-delivery system against immune-associated nephropathy without glucocorticoid side effect , 2021, Theranostics.
[6] D. Fisher,et al. Skin pigmentation and its control: From ultraviolet radiation to stem cells , 2020, Experimental dermatology.
[7] Xiaowei Xu,et al. NB-UVB Induces Melanocytic Differentiation of Human Hair Follicle Neural Crest Stem Cells , 2020, Annals of dermatology.
[8] Juan Li,et al. Anti-melanogenic effects of epigallocatechin-3-gallate (EGCG), epicatechin-3-gallate (ECG) and gallocatechin-3-gallate (GCG) via down-regulation of cAMP/CREB /MITF signaling pathway in B16F10 melanoma cells. , 2020, Fitoterapia.
[9] W. Pavan,et al. The Genetics of Human Skin and Hair Pigmentation. , 2019, Annual review of genomics and human genetics.
[10] K. Gibson-Corley,et al. Enhancing the efficacy of melanocortin 1 receptor-targeted radiotherapy by pharmacologically upregulating the receptor in metastatic melanoma. , 2019, Molecular pharmaceutics.
[11] C. Goding,et al. MITF—the first 25 years , 2019, Genes & development.
[12] Zhongwen Xie,et al. Using Caffeine and Free Amino Acids To Enhance the Transepithelial Transport of Catechins in Caco-2 Cells. , 2019, Journal of agricultural and food chemistry.
[13] Song-Hee Kim,et al. α-Viniferin Improves Facial Hyperpigmentation via Accelerating Feedback Termination of cAMP/PKA-Signaled Phosphorylation Circuit in Facultative Melanogenesis , 2018, Theranostics.
[14] Jongsung Lee,et al. Skin Protective Effect of Epigallocatechin Gallate , 2018, International journal of molecular sciences.
[15] Donghwa Kim,et al. Anti-melanogenic activity of phytosphingosine via the modulation of the microphthalmia-associated transcription factor signaling pathway. , 2017, Journal of dermatological science.
[16] Young Chul Kim,et al. Anti-melanogenic effects of black, green, and white tea extracts on immortalized melanocytes , 2015, Journal of veterinary science.
[17] Te-Sheng Chang,et al. Natural Melanogenesis Inhibitors Acting Through the Down-Regulation of Tyrosinase Activity , 2012, Materials.
[18] Young Hun Kim,et al. Aromatic-turmerone inhibits α-MSH and IBMX-induced melanogenesis by inactivating CREB and MITF signaling pathways , 2011, Archives of Dermatological Research.
[19] C. Bertolotto,et al. Fifteen‐year quest for microphthalmia‐associated transcription factor target genes , 2010, Pigment cell & melanoma research.
[20] Yuehua Wu,et al. Protective effects of green tea extracts on photoaging and photommunosuppression , 2009, Skin research and technology : official journal of International Society for Bioengineering and the Skin (ISBS) [and] International Society for Digital Imaging of Skin (ISDIS) [and] International Society for Skin Imaging.
[21] Y. Akao,et al. Effects of Theaflavins on Melanin Biosynthesis in Mouse B16 Melanoma Cells , 2009, Bioscience, biotechnology, and biochemistry.
[22] Y. Lou,et al. Effect of Caffeine on UVB‐induced Carcinogenesis, Apoptosis, and the Elimination of UVB‐induced Patches of p53 Mutant Epidermal Cells in SKH‐1 Mice † , 2008, Photochemistry and photobiology.
[23] Y. Lou,et al. Stimulatory effect of oral administration of tea, coffee or caffeine on UVB-induced apoptosis in the epidermis of SKH-1 mice. , 2007, Toxicology and applied pharmacology.
[24] Graça Raposo,et al. Melanosomes — dark organelles enlighten endosomal membrane transport , 2007, Nature Reviews Molecular Cell Biology.
[25] E. Steingrímsson,et al. The Microphthalmia-Associated Transcription Factor Mitf Interacts with β-Catenin To Determine Target Gene Expression , 2006, Molecular and Cellular Biology.
[26] D. Fisher,et al. Topical drug rescue strategy and skin protection based on the role of Mc1r in UV-induced tanning , 2006, Nature.
[27] H. Uyama,et al. Tyrosinase inhibitors from natural and synthetic sources: structure, inhibition mechanism and perspective for the future , 2005, Cellular and Molecular Life Sciences CMLS.
[28] G. Babcock,et al. alpha-Melanocortin and endothelin-1 activate antiapoptotic pathways and reduce DNA damage in human melanocytes. , 2005, Cancer research.
[29] N. Copeland,et al. Melanocytes and the microphthalmia transcription factor network. , 2004, Annual review of genetics.
[30] P. Riley. Melanogenesis and melanoma. , 2003, Pigment cell research.
[31] Y. Tsuji,et al. Selective down-regulation of tyrosinase family gene TYRP1 by inhibition of the activity of melanocyte transcription factor, MITF. , 2002, Nucleic acids research.
[32] Y. P. Lu,et al. Inhibitory effects of orally administered green tea, black tea, and caffeine on skin carcinogenesis in mice previously treated with ultraviolet B light (high-risk mice): relationship to decreased tissue fat. , 2001, Cancer research.
[33] C. Elmets,et al. Cutaneous photoprotection from ultraviolet injury by green tea polyphenols. , 2001, Journal of the American Academy of Dermatology.
[34] A. Chakraborty,et al. UV Light and MSH Receptors , 1999, Annals of the New York Academy of Sciences.
[35] Chung S. Yang. Inhibition of carcinogenesis by tea , 1997, Nature.
[36] Y. Lou,et al. Effects of tea, decaffeinated tea, and caffeine on UVB light-induced complete carcinogenesis in SKH-1 mice: demonstration of caffeine as a biologically important constituent of tea. , 1997, Cancer research.
[37] R. Cone,et al. Binding of melanotropic hormones to the melanocortin receptor MC1R on human melanocytes stimulates proliferation and melanogenesis. , 1996, Endocrinology.
[38] Y. Lou,et al. Inhibitory effects of black tea, green tea, decaffeinated black tea, and decaffeinated green tea on ultraviolet B light-induced skin carcinogenesis in 7,12-dimethylbenz[a]anthracene-initiated SKH-1 mice. , 1994, Cancer research.
[39] M. Lee,et al. Role of gene expression and protein synthesis of tyrosinase, TRP-1, lamp-1, and CD63 in UVB-induced melanogenesis in human melanomas. , 1994, The Journal of investigative dermatology.
[40] J. Wikberg,et al. Molecular cloning and expression of the human melanocyte stimulating hormone receptor cDNA , 1992, FEBS letters.
[41] M. Mortrud,et al. The cloning of a family of genes that encode the melanocortin receptors. , 1992, Science.
[42] D. McGee,et al. Induction of skin tanning by subcutaneous administration of a potent synthetic melanotropin. , 1991, JAMA.
[43] P. Riley,et al. Melanogenesis: a realistic target for antimelanoma therapy? , 1991, European journal of cancer.
[44] T. Fitzpatrick,et al. Some aspects of melanin biology: 1950-1975. , 1976, The Journal of investigative dermatology.
[45] A. Lerner,et al. MELANOCYTE-STIMULATING HORMONE AND ADRENOCORTICOTROPHIC HORMONE. THEIR RELATION TO PIGMENTATION. , 1964, The New England journal of medicine.
[46] Ju Xiu-lian. Research Progress of Tyrosinase Inhibitors , 2013 .