Time‐varying magnetic fields of 60 Hz at 7 mT induce DNA double‐strand breaks and activate DNA damage checkpoints without apoptosis
暂无分享,去创建一个
Kiwon Song | Hae June Lee | K. Song | Seung-Ho Yun | Jiyeon Kim | Yeojun Yoon | Seungho Yun | Gwan Soo Park | Jiyeon Kim | Y. Yoon | Hae-June Lee
[1] P. Storz. Reactive oxygen species in tumor progression. , 2005, Frontiers in bioscience : a journal and virtual library.
[2] K. Jung,et al. Effects of 60 Hz 14 µT magnetic field on the apoptosis of testicular germ cell in mice , 2009, Bioelectromagnetics.
[3] R. Bonner,et al. Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks , 2003, Nature Cell Biology.
[4] M Feychting,et al. Magnetic Fields and Breast Cancer in Swedish Adults Residing near High‐Voltage Power Lines , 1998, Epidemiology.
[5] J. Bastos,et al. Electromagnetic Modeling by Finite Element Methods , 2003 .
[6] V. Bohr,et al. Base excision repair of oxidative DNA damage and association with cancer and aging. , 2008, Carcinogenesis.
[7] V. Yamazaki,et al. A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage , 2000, Current Biology.
[8] K. Yoshitomi,et al. Apartment electrical wiring: A cause of extremely low frequency magnetic field exposure in residential areas , 2005, Bioelectromagnetics.
[9] B. Kaina,et al. DNA damage-induced cell death by apoptosis. , 2006, Trends in molecular medicine.
[10] R. Abraham. Cell cycle checkpoint signaling through the ATM and ATR kinases. , 2001, Genes & development.
[11] N. Parinandi,et al. Src-mediated Tyrosine Phosphorylation of p47phox in Hyperoxia-induced Activation of NADPH Oxidase and Generation of Reactive Oxygen Species in Lung Endothelial Cells* , 2005, Journal of Biological Chemistry.
[12] J. Hoeijmakers,et al. Chromosomal stability and the DNA double-stranded break connection , 2001, Nature Reviews Genetics.
[13] Jiri Bartek,et al. Chk1 and Chk2 kinases in checkpoint control and cancer. , 2003, Cancer cell.
[14] K. Shiota,et al. Methyl-CpG-binding Protein, MeCP2, Is a Target Molecule for Maintenance DNA Methyltransferase, Dnmt1* , 2003, The Journal of Biological Chemistry.
[15] R. Adair. A physical analysis of the ion parametric resonance model. , 1998, Bioelectromagnetics.
[16] Thomas Helleday,et al. DNA repair pathways as targets for cancer therapy , 2008, Nature Reviews Cancer.
[17] Satoshi Kawata,et al. Photogeneration of membrane potential hyperpolarization and depolarization in non-excitable cells , 2009, European Biophysics Journal.
[18] J. Hoeijmakers. Genome maintenance mechanisms for preventing cancer , 2001, Nature.
[19] P. Chan,et al. Overexpression of copper/zinc superoxide dismutase does not prevent neonatal lethality in mutant mice that lack manganese superoxide dismutase. , 2000, Free radical biology & medicine.
[20] Steven M. Holland,et al. Mechanical Stretch Enhances mRNA Expression and Proenzyme Release of Matrix Metalloproteinase‐2 (MMP‐2) via NAD(P)H Oxidase‐Derived Reactive Oxygen Species , 2003, Circulation research.
[21] D. Xiao,et al. Effects of Sinusoidal Magnetic Field Observed on Cell Proliferation, Ion Concentration, and Osmolarity in Two Human Cancer Cell Lines , 2006, Electromagnetic biology and medicine.
[22] Dimitris J. Panagopoulos,et al. Mechanism for action of electromagnetic fields on cells. , 2002, Biochemical and biophysical research communications.
[23] S. Fesik,et al. Controlling the Caspases , 2001, Science.
[24] D. Scudiero,et al. Tetrazolium-based assays for cellular viability: a critical examination of selected parameters affecting formazan production. , 1991, Cancer research.
[25] S. Elledge,et al. Ataxia telangiectasia-mutated phosphorylates Chk2 in vivo and in vitro. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[26] E. Appella,et al. UV-induced histone H2AX phosphorylation and DNA damage related proteins accumulate and persist in nucleotide excision repair-deficient XP-B cells. , 2011, DNA repair.
[27] F. Tseng,et al. Design and fabrication of a microplatform for the proximity effect study of localized ELF-EMF on the growth of in vitro HeLa and PC-12 cells , 2010 .
[28] P. Jeggo,et al. An overview of three new disorders associated with genetic instability: LIG4 syndrome, RS-SCID and ATR-Seckel syndrome. , 2004, DNA repair.
[29] Wen-Hwa Lee,et al. The DNA double-strand break response pathway: becoming more BRCAish than ever. , 2004, DNA repair.
[30] K J Johanson,et al. Microelectrophoretic study of radiation-induced DNA damages in individual mammalian cells. , 1984, Biochemical and biophysical research communications.
[31] H. Lai,et al. Acute exposure to a 60 Hz magnetic field increases DNA strand breaks in rat brain cells. , 1997, Bioelectromagnetics.
[32] C. Stevens,et al. Chk2 activates E2F-1 in response to DNA damage , 2003, Nature Cell Biology.
[33] J. Zweier,et al. Viswanathan Natarajan Cells Regulation by Map Kinases in Human Lung Endothelial Hyperoxia-induced Nad(p)h Oxidase Activation And , 2002 .
[34] H. Niida,et al. DNA damage checkpoints in mammals. , 2006, Mutagenesis.
[35] A. Boninsegna,et al. 50-Hz extremely low frequency electromagnetic fields enhance cell proliferation and DNA damage: possible involvement of a redox mechanism. , 2005, Biochimica et biophysica acta.
[36] J. Y. Kim,et al. Anti-inflammatory effect of a human prothrombin fragment-2-derived peptide, NSA9, in EOC2 microglia. , 2008, Biochemical and biophysical research communications.
[37] F. Grasso,et al. Cell Proliferation/Cell Death Balance in Renal Cell Cultures after Exposure to a Static Magnetic Field , 2001, Nephron.
[38] M. Cathcart,et al. Regulation of superoxide anion production by NADPH oxidase in monocytes/macrophages: contributions to atherosclerosis. , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[39] T. Dawson,et al. Mediation of Poly(ADP-Ribose) Polymerase-1-Dependent Cell Death by Apoptosis-Inducing Factor , 2002, Science.
[40] E. Rogakou,et al. DNA Double-stranded Breaks Induce Histone H2AX Phosphorylation on Serine 139* , 1998, The Journal of Biological Chemistry.
[41] Junjie Chen,et al. Tumor Suppressor P53 Binding Protein 1 (53bp1) Is Involved in DNA Damage–Signaling Pathways , 2001, The Journal of cell biology.
[42] P. Raskmark,et al. Changes in cell proliferation due to environmental non-ionizing radiation 1. ELF electromagnetic fields , 1995 .
[43] B. Halliwell,et al. Damage to DNA by reactive oxygen and nitrogen species: role in inflammatory disease and progression to cancer. , 1996, The Biochemical journal.
[44] J. Bartek,et al. Ataxia-telangiectasia-mutated (ATM) and NBS1-dependent Phosphorylation of Chk1 on Ser-317 in Response to Ionizing Radiation* , 2003, The Journal of Biological Chemistry.
[45] R. Tice,et al. A simple technique for quantitation of low levels of DNA damage in individual cells. , 1988, Experimental cell research.
[46] K. Song,et al. Repetitive exposure to a 60-Hz time-varying magnetic field induces DNA double-strand breaks and apoptosis in human cells. , 2010, Biochemical and biophysical research communications.
[47] M. Moskowitz,et al. PARP-1--a Perpetrator of Apoptotic Cell Death? , 2002, Science.
[48] F Sturmans,et al. Cancer mortality and residence near electricity transmission equipment: a retrospective cohort study. , 1993, International journal of epidemiology.
[49] J Wade Harper,et al. The DNA damage response: ten years after. , 2007, Molecular cell.
[50] G. Gajda,et al. Evaluating DNA Damage in Rodent Brain after Acute 60 Hz Magnetic-Field Exposure , 2005, Radiation research.
[51] John Swanson,et al. Childhood cancer in relation to distance from high voltage power lines in England and Wales: a case-control study , 2005, BMJ : British Medical Journal.