Siglec-7 as a Novel Biomarker to Predict Mortality in Decompensated Cirrhosis and Acute Kidney Injury
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R. Chung | R. Thadhani | S. Karumanchi | Sahir Kalim | A. Allegretti | Dihua Xu | Joshua Wibecan | S. A. Karumanchi | Guillermo Ortiz | H. Shan | Ravi I. Thadhani | Dongsheng Zhang | S. Karumanchi
[1] R. Chung,et al. Changes in Kidney Function After Transjugular Intrahepatic Portosystemic Shunts Versus Large-Volume Paracentesis in Cirrhosis: A Matched Cohort Analysis. , 2016, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[2] G. Ferraioli,et al. Lack of Siglec-7 expression identifies a dysfunctional natural killer cell subset associated with liver inflammation and fibrosis in chronic HCV infection , 2015, Gut.
[3] M. Ronsoni,et al. Single‐centre validation of the EASL‐CLIF Consortium definition of acute‐on‐chronic liver failure and CLIF‐SOFA for prediction of mortality in cirrhosis , 2015, Liver international : official journal of the International Association for the Study of the Liver.
[4] W. Kim,et al. Diagnosis and management of acute kidney injury in patients with cirrhosis: revised consensus recommendations of the International Club of Ascites , 2015, Gut.
[5] Young Youn Cho,et al. CLIF‐SOFA scoring system accurately predicts short‐term mortality in acutely decompensated patients with alcoholic cirrhosis: a retrospective analysis , 2015, Liver international : official journal of the International Association for the Study of the Liver.
[6] A. Davenport,et al. Renal dysfunction in cirrhosis is not just a vasomotor nephropathy , 2014, Kidney international.
[7] P. Romero,et al. Interactions between Siglec-7/9 receptors and ligands influence NK cell-dependent tumor immunosurveillance. , 2014, The Journal of clinical investigation.
[8] R. Maserati,et al. Sialic acid-binding Ig-like lectin-7 interacts with HIV-1 gp120 and facilitates infection of CD4pos T cells and macrophages , 2013, Retrovirology.
[9] Stephen M. Canham,et al. Glycocalyx Engineering Reveals a Siglec-Based Mechanism for NK Cell Immunoevasion , 2013, Nature chemical biology.
[10] Foreman,et al. The state of US health, 1990-2010: burden of diseases, injuries, and risk factors. , 2013, JAMA.
[11] Terry M. Therneau,et al. Underestimation of liver-related mortality in the United States. , 2013, Gastroenterology.
[12] R. Moreau,et al. Acute-on-chronic liver failure is a distinct syndrome that develops in patients with acute decompensation of cirrhosis. , 2013, Gastroenterology.
[13] Dominique Thabut,et al. Liver fibrosis evaluation using real-time shear wave elastography: applicability and diagnostic performance using methods without a gold standard. , 2013, Journal of hepatology.
[14] C. Sprung,et al. Surviving Sepsis Campaign: International Guidelines for Management of Severe Sepsis and Septic Shock 2012 , 2013, Critical care medicine.
[15] M. Mondelli,et al. Engagement of Siglec-7 Receptor Induces a Pro-Inflammatory Response Selectively in Monocytes , 2012, PloS one.
[16] F. Wong,et al. Acute kidney injury in decompensated cirrhosis , 2012, Gut.
[17] Keiichiro Sakuma,et al. Colonic Epithelial Cells Express Specific Ligands for Mucosal Macrophage Immunosuppressive Receptors Siglec-7 and -9 , 2012, The Journal of Immunology.
[18] S. Pillai,et al. Siglecs and immune regulation. , 2012, Annual review of immunology.
[19] P. Sun,et al. Siglecs Facilitate HIV-1 Infection of Macrophages through Adhesion with Viral Sialic Acids , 2011, PloS one.
[20] A. Varki. Letter to the Glyco-Forum: Since there are PAMPs and DAMPs, there must be SAMPs? Glycan “self-associated molecular patterns” dampen innate immunity, but pathogens can mimic them , 2011 .
[21] Maurizio Biselli,et al. The MELD score in patients awaiting liver transplant: strengths and weaknesses. , 2011, Journal of hepatology.
[22] M. Kuijf,et al. Campylobacter jejuni Lipooligosaccharides Modulate Dendritic Cell-Mediated T Cell Polarization in a Sialic Acid Linkage-Dependent Manner , 2011, Infection and Immunity.
[23] M. Pavesi,et al. Prognostic importance of the cause of renal failure in patients with cirrhosis. , 2011, Gastroenterology.
[24] Michael J. Pencina,et al. Statistical methods for assessment of added usefulness of new biomarkers , 2010, Clinical chemistry and laboratory medicine.
[25] Y. Yamaguchi,et al. Siglec-7 mediates nonapoptotic cell death independently of its immunoreceptor tyrosine-based inhibitory motifs in monocytic cell line U937. , 2010, Glycobiology.
[26] R. Schrier,et al. Renal failure in cirrhosis. , 2009, The New England journal of medicine.
[27] A. Moretta,et al. The decreased expression of Siglec-7 represents an early marker of dysfunctional natural killer-cell subsets associated with high levels of HIV-1 viremia. , 2009, Blood.
[28] W. Stremmel,et al. Limitations of the MELD score in predicting mortality or need for removal from waiting list in patients awaiting liver transplantation , 2009, BMC gastroenterology.
[29] G. Garcia‐Tsao,et al. Management and Treatment of Patients With Cirrhosis and Portal Hypertension: Recommendations From the Department of Veterans Affairs Hepatitis C Resource Center Program and the National Hepatitis C Program , 2009, The American Journal of Gastroenterology.
[30] James C Paulson,et al. Siglecs as targets for therapy in immune-cell-mediated disease. , 2009, Trends in pharmacological sciences.
[31] C. Parikh,et al. Acute kidney injury in cirrhosis , 2008, Hepatology.
[32] L. Pulliam,et al. Sialoadhesin Expressed on IFN-Induced Monocytes Binds HIV-1 and Enhances Infectivity , 2008, PloS one.
[33] M. Levy,et al. Surviving Sepsis Campaign: International guidelines for management of severe sepsis and septic shock: 2008 , 2007, Intensive Care Medicine.
[34] R. Kannagi. Carbohydrate antigen sialyl Lewis a--its pathophysiological significance and induction mechanism in cancer progression. , 2007, Chang Gung medical journal.
[35] Ajit Varki,et al. Siglecs and their roles in the immune system , 2007, Nature Reviews Immunology.
[36] Vicente Arroyo,et al. Diagnosis, prevention and treatment of hepatorenal syndrome in cirrhosis , 2007, Postgraduate Medical Journal.
[37] T. Gonwa,et al. Hepatorenal syndrome: pathophysiology and management. , 2006, Clinical journal of the American Society of Nephrology : CJASN.
[38] S. Friedman,et al. Anti-fibrotic activity of NK cells in experimental liver injury through killing of activated HSC. , 2006, Journal of Hepatology.
[39] R. Sun,et al. Natural killer cells ameliorate liver fibrosis by killing activated stellate cells in NKG2D-dependent and tumor necrosis factor-related apoptosis-inducing ligand-dependent manners. , 2006, Gastroenterology.
[40] H. Nauwynck,et al. Porcine Arterivirus Infection of Alveolar Macrophages Is Mediated by Sialic Acid on the Virus , 2004, Journal of Virology.
[41] K. Kumamoto,et al. Loss of Disialyl Lewisa, the Ligand for Lymphocyte Inhibitory Receptor Sialic Acid-Binding Immunoglobulin-Like Lectin-7 (Siglec-7) Associated with Increased Sialyl Lewisa Expression on Human Colon Cancers , 2004, Cancer Research.
[42] T. Yamaji,et al. A Small Region of the Natural Killer Cell Receptor, Siglec-7, Is Responsible for Its Preferred Binding to α2,8-Disialyl and Branched α2,6-Sialyl Residues , 2002, The Journal of Biological Chemistry.
[43] P. Klenerman,et al. Identification and Characterization of a Novel Siglec, Siglec-7, Expressed by Human Natural Killer Cells and Monocytes* , 1999, The Journal of Biological Chemistry.
[44] R. Biassoni,et al. Identification and Molecular Cloning of P75/Airm1, a Novel Member of the Sialoadhesin Family That Functions as an Inhibitory Receptor in Human Natural Killer Cells , 1999, The Journal of experimental medicine.
[45] R Core Team,et al. R: A language and environment for statistical computing. , 2014 .
[46] K. Hajian‐Tilaki,et al. Receiver Operating Characteristic (ROC) Curve Analysis for Medical Diagnostic Test Evaluation. , 2013, Caspian journal of internal medicine.
[47] C. Murray,et al. The state of US health, : burden of diseases, injuries, and risk factors. , 2013 .
[48] Norbert Lameire,et al. Notice , 2012, Kidney International Supplements.
[49] Yeong-Seok Im,et al. Hyponatremia and Mortality among Patients on the Liver-Transplant Waiting List , 2009 .
[50] R. Wiesner,et al. Model for end-stage liver disease (MELD) and allocation of donor livers. , 2003, Gastroenterology.
[51] T. Yamaji,et al. A small region of the natural killer cell receptor, Siglec-7, is responsible for its preferred binding to alpha 2,8-disialyl and branched alpha 2,6-sialyl residues. A comparison with Siglec-9. , 2002, The Journal of biological chemistry.
[52] M. F. Parry,et al. A Retrospective Analysis , 1990 .