Objective—Low high-density lipoprotein cholesterol (HDL-C) is associated with cardiometabolic pathologies. In this study, we investigate the biological pathways and individual genes behind low HDL-C by integrating results from 3 high- throughput data sources: adipose tissue transcriptomics, HDL lipi
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T. Lehtimäki | L. Peltonen | V. Salomaa | O. Raitakari | A. Palotie | S. Ripatti | M. Taskinen | I. Surakka | Antti-Pekka Sarin | J. Kettunen | J. Eriksson | A. Jula | M. Järvelin | M. Orešič | A. Ruokonen | D. Mirel | M. Jauhiainen | C. Ehnholm | L. Yetukuri | T. Hyötyläinen | Jing Tang | J. Naukkarinen | J. Soronen | Pirkka-Pekka Laurila | S. Söderlund