Cardiovascular Harmful Effects of Recommended Daily Doses (13 µg/kg/day), Tolerable Upper Intake Doses (0.14 mg/kg/day) and Twice the Tolerable Doses (0.28 mg/kg/day) of Copper
暂无分享,去创建一个
D. Vassallo | M. Simões | M. Miguel | G. Wiggers | Filipe Martinuzo Filetti | I. R. Schereider | Ingridy Reinholz Grafites Schereider
[1] F. Luo,et al. Angiotensin I-converting an enzyme inhibitory peptide: an emerging candidate for vascular dysfunction therapy. , 2021, Critical reviews in biotechnology.
[2] D. Vassallo,et al. Chronic mercury exposure induces oxidative stress in female rats by endothelial nitric oxide synthase uncoupling and cyclooxygenase‐2 activation, without affecting oestrogen receptor function , 2021, Basic & clinical pharmacology & toxicology.
[3] Rosiane Viana Zuza Diniz,et al. Biomarkers of Zinc and Copper Status and Associated Factors in Outpatients with Ischemic and Non-Ischemic Heart Failure. , 2021, Journal of the American College of Nutrition.
[4] A. Prystupa,et al. Assessment of Concentrations of Heavy Metals in Postmyocardial Infarction Patients and Patients Free from Cardiovascular Event , 2021, Cardiology research and practice.
[5] R. Burnett,et al. Long-term exposure to iron and copper in fine particulate air pollution and their combined impact on reactive oxygen species concentration in lung fluid: a population-based cohort study of cardiovascular disease incidence and mortality in Toronto, Canada. , 2020, International journal of epidemiology.
[6] G. Ferns,et al. Association of Zinc and Copper Status with Cardiovascular Diseases and their Assessment Methods: A Review Study. , 2020, Mini reviews in medicinal chemistry.
[7] Ji Chen,et al. The molecular mechanisms of copper metabolism and its roles in human diseases , 2020, Pflügers Archiv - European Journal of Physiology.
[8] J. Martínez-González,et al. The Role of Lysyl Oxidase Enzymes in Cardiac Function and Remodeling , 2019, Cells.
[9] D. Vassallo,et al. Long-term Mercury Exposure Accelerates the Development of Hypertension in Prehypertensive Spontaneously Hypertensive Rats Inducing Endothelial Dysfunction: the Role of Oxidative Stress and Cyclooxygenase-2 , 2019, Biological Trace Element Research.
[10] C. Exley,et al. Egg White Hydrolysate: A new putative agent to prevent vascular dysfunction in rats following long-term exposure to aluminum. , 2019, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.
[11] F. Barile. Cardiovascular Toxicology , 2019, Barile’s Clinical Toxicology.
[12] F. Chaves,et al. The association of urine metals and metal mixtures with cardiovascular incidence in an adult population from Spain: the Hortega Follow-Up Study. , 2019, International journal of epidemiology.
[13] M. Pohanka. Copper and copper nanoparticles toxicity and their impact on basic functions in the body. , 2019, Bratislavske lekarske listy.
[14] Hourieh Alkadi. A Review On Free Radicals and Antioxidants. , 2020, Infectious disorders drug targets.
[15] A. Relling,et al. Effects of Copper and Zinc Supplementation on Weight Gain and Hematological Parameters in Pre-weaning Calves , 2018, Biological Trace Element Research.
[16] D. Vassallo,et al. Egg white-derived peptides prevent cardiovascular disorders induced by mercury in rats: Role of angiotensin-converting enzyme (ACE) and NADPH oxidase. , 2017, Toxicology letters.
[17] P. Mello-Carpes,et al. Ameliorative effects of egg white hydrolysate on recognition memory impairments associated with chronic exposure to low mercury concentration , 2016, Neurochemistry International.
[18] Renata Andrade Ávila,et al. Mechanisms involved in the in vitro contractile dysfunction induced by different concentrations of ferrous iron in the rat myocardium. , 2016, Toxicology in vitro : an international journal published in association with BIBRA.
[19] R. López-Fandiño,et al. Pepsin Egg White Hydrolysate Ameliorates Obesity-Related Oxidative Stress, Inflammation and Steatosis in Zucker Fatty Rats , 2016, PloS one.
[20] Kristin Decker. Dietary Reference Intakes For Thiamin Riboflavin Niacin Vitamin B6 Folate Vitamin B12 Pantothenic Acid Biotin And Choline , 2016 .
[21] Y. J. Kang,et al. Role of copper in regression of cardiac hypertrophy. , 2015, Pharmacology & therapeutics.
[22] A. Briones,et al. MAPK pathway activation by chronic lead-exposure increases vascular reactivity through oxidative stress/cyclooxygenase-2-dependent pathways. , 2015, Toxicology and applied pharmacology.
[23] Samiksha Singh,et al. Arsenic contamination, consequences and remediation techniques: a review. , 2015, Ecotoxicology and environmental safety.
[24] G. Filippatos,et al. Clinical and echocardiographic correlates of serum copper and zinc in acute and chronic heart failure , 2014, Clinical Research in Cardiology.
[25] D. Vassallo,et al. Chronic Lead Exposure Increases Blood Pressure and Myocardial Contractility in Rats , 2014, PloS one.
[26] R. F. Ribeiro,et al. Acute exposure to lead increases myocardial contractility independent of hypertension development , 2013, Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas.
[27] M. Valko,et al. Advances in metal-induced oxidative stress and human disease. , 2011, Toxicology.
[28] E. Salehifar,et al. Serum Zinc and Copper Levels in Ischemic Cardiomyopathy , 2009, Biological Trace Element Research.
[29] E. Salehifar,et al. The Study of Cu and Zn Serum Levels in Idiopathic Dilated Cardiomyopathy (IDCMP) Patients and its Comparison with Healthy Volunteers , 2008, Biological Trace Element Research.
[30] R. López-Fandiño,et al. Effect of the long-term intake of an egg white hydrolysate on the oxidative status and blood lipid profile of spontaneously hypertensive rats. , 2008, Food chemistry.
[31] R. López-Fandiño,et al. Vasodilator effects of peptides derived from egg white proteins , 2007, Regulatory Peptides.
[32] H. Taskapan,et al. Trace element status (Se, Zn, Cu) in heart failure. , 2006, Anadolu kardiyoloji dergisi : AKD = the Anatolian journal of cardiology.
[33] Paula Aracena,et al. Possible mechanisms underlying copper-induced damage in biological membranes leading to cellular toxicity. , 2005, Chemico-biological interactions.
[34] R. López-Fandiño,et al. Angiotensin I-converting enzyme inhibitory activity of peptides derived from egg white proteins by enzymatic hydrolysis. , 2004, Journal of food protection.
[35] R. Doughty,et al. Regeneration of the heart in diabetes by selective copper chelation. , 2004, Diabetes.
[36] C. Bonan,et al. Effects of mercury on myosin ATPase in the ventricular myocardium of the rat. , 2003, Comparative biochemistry and physiology. Toxicology & pharmacology : CBP.
[37] Exhibit. Copper in Drinking-water Background document for development of WHO Guidelines for Drinking-water Quality , 2003 .
[38] D. Bers. Cardiac excitation–contraction coupling , 2002, Nature.
[39] M. Reiter,et al. Effects of inhibition of calcium and potassium currents in guinea‐pig cardiac contraction: comparison of β‐caryophyllene oxide, eugenol, and nifedipine , 2000, British journal of pharmacology.
[40] R A Bassani,et al. Relaxation in rabbit and rat cardiac cells: species‐dependent differences in cellular mechanisms. , 1994, The Journal of physiology.
[41] J. Mill,et al. Effects of isoproterenol on the mechanical activity of isolated papillary muscles and perfused rat hearts in various calcium concentrations. , 1994, Pharmacological research.
[42] J. Salonen,et al. Serum copper and the risk of acute myocardial infarction: a prospective population study in men in eastern Finland. , 1991, American journal of epidemiology.
[43] N. Dhalla,et al. Modification of cardiac adrenergic receptors by oxygen free radicals. , 1991, The American journal of physiology.
[44] J. Mill,et al. Post-rest contractions of amphibian cardiac muscle. , 1991, Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas.
[45] A. Howard,et al. Studies in copper status and atherosclerosis. , 1990, Biochemical Society transactions.
[46] J. Mill,et al. Mechanical behavior of rest contractions in cardiac muscle. , 1988, Acta physiologica et pharmacologica latinoamericana : organo de la Asociacion Latinoamericana de Ciencias Fisiologicas y de la Asociacion Latinoamericana de Farmacologia.
[47] B. Halliwell,et al. Oxygen free radicals and iron in relation to biology and medicine: some problems and concepts. , 1986, Archives of biochemistry and biophysics.
[48] G. Czapski,et al. Unusual copper-induced sensitization of the biological damage due to superoxide radicals. , 1981, The Journal of biological chemistry.
[49] N. Misra,et al. Serum copper, ceruloplasmin & iron in ischaemic heart disease. , 1978, Indian heart journal.