Complement activation and inhibition: a delicate balance.
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[1] K. Reid,et al. Location of the binding site in subcomponent C1q for plasma fibronectin. , 1984, Acta pathologica, microbiologica, et immunologica Scandinavica. Supplement.
[2] C. Mold,et al. C-reactive protein inhibits pneumococcal activation of the alternative pathway by increasing the interaction between factor H and C3b. , 1984, Journal of immunology.
[3] G. Laurie,et al. The C1q subunit of the first component of complement binds to laminin: a mechanism for the deposition and retention of immune complexes in basement membrane. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[4] C. Bristow,et al. Evidence for the binding of human serum amyloid P component to Clq and Fab gamma. , 1986, Molecular immunology.
[5] B. Dahlbäck,et al. Assembly of protein S and C4b-binding protein on membranes. , 1990, The Journal of biological chemistry.
[6] S. Ruddy,et al. Synovial fluid levels of complement SC5b-9 and fragment Bb are elevated in patients with rheumatoid arthritis. , 2010, Arthritis and rheumatism.
[7] C. Mold,et al. Serum amyloid P component binds to histones and activates the classical complement pathway. , 1992, Journal of immunology.
[8] B. Moffatt,et al. Complement factor I and cofactors in control of complement system convertase enzymes. , 1993, Methods in enzymology.
[9] H. Gewurz,et al. Human serum amyloid P component oligomers bind and activate the classical complement pathway via residues 14-26 and 76-92 of the A chain collagen-like region of C1q. , 1993, Journal of Immunology.
[10] B. Dahlbäck,et al. Serum Amyloid P Component Binding to C4b-binding Protein (*) , 1995, The Journal of Biological Chemistry.
[11] O. Andersen,et al. Binding of Complement Proteins C1q and C4bp to Serum Amyloid P Component (SAP) in Solid Contra Liquid Phase , 1996, Scandinavian journal of immunology.
[12] J. Ahearn,et al. C1q binds directly and specifically to surface blebs of apoptotic human keratinocytes: complement deficiency and systemic lupus erythematosus revisited. , 1997, Journal of immunology.
[13] C. Mold,et al. Regulation of complement activation by C-reactive protein. , 1999, Immunopharmacology.
[14] S. Meri,et al. Regulation of complement activation by C-reactive protein: targeting the complement inhibitory activity of factor H by an interaction with short consensus repeat domains 7 and 8-11. , 1999, Journal of immunology.
[15] N. Brot,et al. C-Reactive Protein Binds to Apoptotic Cells, Protects the Cells from Assembly of the Terminal Complement Components, and Sustains an Antiinflammatory Innate Immune Response , 2000, The Journal of experimental medicine.
[16] S. Carsons,et al. Fibronectin fragments and their role in inflammatory arthritis. , 2000, Seminars in arthritis and rheumatism.
[17] Simon C Watkins,et al. The Globular Heads of C1q Specifically Recognize Surface Blebs of Apoptotic Vascular Endothelial Cells1 , 2001, The Journal of Immunology.
[18] A. Blom,et al. Structural Requirements for the Complement Regulatory Activities of C4BP* , 2001, The Journal of Biological Chemistry.
[19] M. Walport,et al. Temporary depletion of complement component C3 or genetic deficiency of C1q significantly delays onset of scrapie , 2001, Nature Medicine.
[20] V. Fadok,et al. C1q and Mannose Binding Lectin Engagement of Cell Surface Calreticulin and Cd91 Initiates Macropinocytosis and Uptake of Apoptotic Cells , 2001, The Journal of experimental medicine.
[21] I. Xenarios,et al. Complement facilitates early prion pathogenesis , 2001, Nature Medicine.
[22] P. Tacnet-Delorme,et al. Beta-amyloid fibrils activate the C1 complex of complement under physiological conditions: evidence for a binding site for A beta on the C1q globular regions. , 2001, Journal of immunology.
[23] P. Tacnet-Delorme,et al. β-Amyloid Fibrils Activate the C1 Complex of Complement Under Physiological Conditions: Evidence for a Binding Site for Aβ on the C1q Globular Regions1 , 2001, The Journal of Immunology.
[24] J. Volanakis,et al. Topology and Structure of the C1q-Binding Site on C-Reactive Protein1 , 2001, The Journal of Immunology.
[25] N. Thielens,et al. Structural biology of the C1 complex of complement unveils the mechanisms of its activation and proteolytic activity. , 2002, Molecular immunology.
[26] A. Mantovani,et al. Direct binding of C1q to apoptotic cells and cell blebs induces complement activation , 2002, European journal of immunology.
[27] Christine Gaboriaud,et al. The Crystal Structure of the Globular Head of Complement Protein C1q Provides a Basis for Its Versatile Recognition Properties* , 2003, Journal of Biological Chemistry.
[28] A. Blom,et al. CCP1-4 of the C4b-binding protein alpha-chain are required for factor I mediated cleavage of complement factor C3b. , 2003, Molecular immunology.
[29] Cees van Kooten,et al. Recognition and clearance of apoptotic cells: a role for complement and pentraxins. , 2003, Trends in immunology.
[30] S. Ranganathan,et al. A common site within factor H SCR 7 responsible for binding heparin, C‐reactive protein and streptococcal M protein , 2003 .
[31] S. Ranganathan,et al. A common site within factor H SCR 7 responsible for binding heparin, C-reactive protein and streptococcal M protein. , 2000, European Journal of Immunology.
[32] H. Budka,et al. Complement activation in human prion disease , 2004, Neurobiology of Disease.
[33] D. Heinegård,et al. Cleavage of Fibromodulin in Cartilage Explants Involves Removal of the N-terminal Tyrosine Sulfate-rich Region by Proteolysis at a Site That Is Sensitive to Matrix Metalloproteinase-13* , 2004, Journal of Biological Chemistry.
[34] Anna M. Blom,et al. The Extracellular Matrix and Inflammation , 2005, Journal of Biological Chemistry.
[35] R. Veerhuis,et al. Amyloid associated proteins in Alzheimer's and prion disease. , 2005, Current drug targets. CNS and neurological disorders.
[36] R. T. Smith,et al. A common haplotype in the complement regulatory gene factor H (HF1/CFH) predisposes individuals to age-related macular degeneration. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[37] J. Atkinson,et al. Targeted and restricted complement activation on acrosome-reacted spermatozoa. , 2005, The Journal of clinical investigation.
[38] P. Gasque,et al. CD46 Plays a Key Role in Tailoring Innate Immune Recognition of Apoptotic and Necrotic Cells* , 2005, Journal of Biological Chemistry.
[39] J. Gilbert,et al. Complement Factor H Variant Increases the Risk of Age-Related Macular Degeneration , 2005, Science.
[40] L. Marnell,et al. C-reactive protein: ligands, receptors and role in inflammation. , 2005, Clinical immunology.
[41] M. Daha,et al. Interactions of the Extracellular Matrix Proteoglycans Decorin and Biglycan with C1q and Collectins1 , 2005, The Journal of Immunology.
[42] W. Janssen,et al. Cell-Surface Calreticulin Initiates Clearance of Viable or Apoptotic Cells through trans-Activation of LRP on the Phagocyte , 2005, Cell.
[43] A. Blom,et al. C4b-binding protein binds to necrotic cells and DNA, limiting DNA release and inhibiting complement activation , 2005, The Journal of experimental medicine.
[44] Gérard J Arlaud,et al. Complement protein C1q recognizes a conformationally modified form of the prion protein. , 2005, Biochemistry.
[45] N. Mabbott,et al. Prions and their lethal journey to the brain , 2006, Nature Reviews Microbiology.
[46] Fabian D. G. McGrath,et al. Evidence That Complement Protein C1q Interacts with C-Reactive Protein through Its Globular Head Region1 , 2006, The Journal of Immunology.
[47] J. Köhl. Self, non-self, and danger: a complementary view. , 2006, Advances in experimental medicine and biology.
[48] H. Cook,et al. Mechanisms of Disease: the complement system and the pathogenesis of systemic lupus erythematosus , 2006, Nature Clinical Practice Rheumatology.
[49] A. Blom,et al. Regulation of complement activation by C-reactive protein: targeting of the inhibitory activity of C4b-binding protein. , 2006, Journal of immunology.
[50] A. Blom,et al. Regulation of Complement Activation by C-Reactive Protein: Targeting of the Inhibitory Activity of C4b-Binding Protein1 , 2006, The Journal of Immunology.
[51] A. Mantovani,et al. Interaction of C1q with IgG1, C-reactive protein and pentraxin 3: mutational studies using recombinant globular head modules of human C1q A, B, and C chains. , 2006, Biochemistry.
[52] R. T. Smith,et al. Variation in factor B (BF) and complement component 2 (C2) genes is associated with age-related macular degeneration , 2006, Nature Genetics.
[53] M. Fonseca,et al. The double-edged flower: roles of complement protein C1q in neurodegenerative diseases. , 2006, Advances in experimental medicine and biology.
[54] J. Thurman,et al. Alternative Complement Pathway Activation Is Essential for Inflammation and Joint Destruction in the Passive Transfer Model of Collagen-Induced Arthritis1 , 2006, The Journal of Immunology.
[55] John D Lambris,et al. The role of complement in inflammatory diseases from behind the scenes into the spotlight. , 2007, The American journal of pathology.
[56] A. Blom,et al. C4b-binding Protein and Factor H Compensate for the Loss of Membrane-bound Complement Inhibitors to Protect Apoptotic Cells against Excessive Complement Attack* , 2007, Journal of Biological Chemistry.
[57] Robert B Sim,et al. Prion protein activates and fixes complement directly via the classical pathway: implications for the mechanism of scrapie agent propagation in lymphoid tissue. , 2007, Molecular immunology.
[58] Don H. Anderson,et al. Y402H Polymorphism of Complement Factor H Affects Binding Affinity to C-Reactive Protein1 , 2007, The Journal of Immunology.
[59] J. Alexander,et al. The simple design of complement factor H: Looks can be deceiving. , 2007, Molecular immunology.
[60] A. Blom,et al. Rheumatoid arthritis and the complement system , 2007, Annals of medicine.
[61] U. Schlötzer-Schrehardt,et al. Defective complement control of factor H (Y402H) and FHL-1 in age-related macular degeneration. , 2007, Molecular immunology.
[62] A. Blom,et al. The factor H variant associated with age-related macular degeneration (His-384) and the non-disease-associated form bind differentially to C-reactive protein, fibromodulin, DNA, and necrotic cells. , 2007, The Journal of biological chemistry.
[63] D. Uhrín,et al. Structure Shows That a Glycosaminoglycan and Protein Recognition Site in Factor H Is Perturbed by Age-related Macular Degeneration-linked Single Nucleotide Polymorphism* , 2007, Journal of Biological Chemistry.
[64] L. Truedsson,et al. Complement deficiencies and systemic lupus erythematosus , 2007, Autoimmunity.
[65] A. Blom,et al. C4b-Binding Protein Is Present in Affected Areas of Myocardial Infarction during the Acute Inflammatory Phase and Covers a Larger Area than C3 , 2008, PloS one.
[66] G. Rivas,et al. Complement Factor H Binds to Denatured Rather than to Native Pentameric C-reactive Protein* , 2008, Journal of Biological Chemistry.
[67] E. Londos,et al. C4b-binding protein in Alzheimer's disease: binding to Abeta1-42 and to dead cells. , 2008, Molecular immunology.
[68] P. Zipfel,et al. P84Complement factor H binds to monomeric CRP which enhances the complement inactivation and mediates factor H binding to apoptotic particles and necrotic cells , 2008 .
[69] P. Tacnet-Delorme,et al. C1q Binds Phosphatidylserine and Likely Acts as a Multiligand-Bridging Molecule in Apoptotic Cell Recognition1 , 2008, The Journal of Immunology.
[70] N. Thielens,et al. The lectin‐like activity of human C1q and its implication in DNA and apoptotic cell recognition , 2008, FEBS letters.
[71] P. Gasque,et al. Role of complement and complement regulators in the removal of apoptotic cells. , 2008, Molecular immunology.
[72] D. Hourcade,et al. The complement protein properdin binds apoptotic T cells and promotes complement activation and phagocytosis , 2008, Proceedings of the National Academy of Sciences.
[73] Jun Zhou,et al. Complement C3 and C4 expression in C1q sufficient and deficient mouse models of Alzheimer’s disease , 2008, Journal of neurochemistry.
[74] A. Blom,et al. Structural basis and functional effects of the interaction between complement inhibitor C4b-binding protein and DNA. , 2008, Molecular immunology.
[75] A. Blom,et al. Native, amyloid fibrils and beta-oligomers of the C-terminal domain of human prion protein display differential activation of complement and bind C1q, factor H and C4b-binding protein directly. , 2008, Molecular immunology.
[76] Kutty Selva Nandakumar,et al. C4b-binding protein (C4BP) inhibits development of experimental arthritis in mice , 2008, Annals of the rheumatic diseases.
[77] A. Blom,et al. Short leucine-rich glycoproteins of the extracellular matrix display diverse patterns of complement interaction and activation. , 2009, Molecular immunology.