p66Shc Mediates Mitochondrial Dysfunction Dependent on PKC Activation in Airway Epithelial Cells Induced by Cigarette Smoke
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Jingjing Tang | Jie Zhang | H. Shan | Ming Zhang | Xia Yang | Yali Li | Qiuhong Zhang
[1] Min Chen,et al. Cigarette smoke extract induces placental growth factor release from human bronchial epithelial cells via ROS/MAPK (ERK-1/2)/Egr-1 axis , 2016, International journal of chronic obstructive pulmonary disease.
[2] R. Yan,et al. Curcumin ameliorates alveolar epithelial injury in a rat model of chronic obstructive pulmonary disease. , 2016, Life sciences.
[3] M. Hermann,et al. Novel Insights into the PKCβ-dependent Regulation of the Oxidoreductase p66Shc* , 2016, The Journal of Biological Chemistry.
[4] M. Hermann,et al. cJun N-terminal kinase (JNK) phosphorylation of serine 36 is critical for p66Shc activation , 2016, Scientific Reports.
[5] W. Zou,et al. Nicotine reduces the levels of surfactant proteins A and D via Wnt/β-catenin and PKC signaling in human airway epithelial cells , 2016, Respiratory Physiology & Neurobiology.
[6] E. Vagdatli,et al. Effects of acute cigarette smoking on total blood count and markers of oxidative stress in active and passive smokers. , 2015, Hippokratia.
[7] T. Soldati,et al. Reactive oxygen species and mitochondria: A nexus of cellular homeostasis , 2015, Redox biology.
[8] Cuk-Seong Kim,et al. Trichostatin A Modulates Angiotensin II-induced Vasoconstriction and Blood Pressure Via Inhibition of p66shc Activation , 2015, The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology.
[9] Wei Yang,et al. High-dose alcohol induces reactive oxygen species-mediated apoptosis via PKC-β/p66Shc in mouse primary cardiomyocytes. , 2015, Biochemical and biophysical research communications.
[10] Yi-Xiong Tan,et al. The expression of p66shc in peripheral blood monocytes is increased in patients with coronary heart disease and correlated with endothelium-dependent vasodilatation , 2015, Heart and Vessels.
[11] Lin Sun,et al. PKCδ Promotes High Glucose Induced Renal Tubular Oxidative Damage via Regulating Activation and Translocation of p66Shc , 2014, Oxidative medicine and cellular longevity.
[12] H. Tse,et al. Mitochondrial transfer of induced pluripotent stem cell-derived mesenchymal stem cells to airway epithelial cells attenuates cigarette smoke-induced damage. , 2014, American journal of respiratory cell and molecular biology.
[13] Shui-juan Zhang,et al. Cigarette smoke extract-induced BEAS-2B cell apoptosis and anti-oxidative Nrf-2 up-regulation are mediated by ROS-stimulated p38 activation , 2014, Toxicology mechanisms and methods.
[14] G. Borchard,et al. Toll-like receptor 2 regulates the barrier function of human bronchial epithelial monolayers through atypical protein kinase C zeta, and an increase in expression of claudin-1 , 2014, Tissue barriers.
[15] Jianping Zhou,et al. The Expression of p66Shc Protein in Benign, Premalignant, and Malignant Gastrointestinal Lesions , 2014, Pathology & Oncology Research.
[16] R. Foronjy,et al. Cigarette smoke activates the proto-oncogene c-src to promote airway inflammation and lung tissue destruction. , 2014, American journal of respiratory cell and molecular biology.
[17] Y. Tesfaigzi,et al. Molecular processes that drive cigarette smoke-induced epithelial cell fate of the lung. , 2014, American journal of respiratory cell and molecular biology.
[18] Sina Zarrintan,et al. Prolonged cigarette smoke exposure alters mitochondrial structure and function in airway epithelial cells , 2013, Respiratory Research.
[19] Michael A Thompson,et al. Mitochondria in lung diseases , 2013, Expert Review of Respiratory Medicine.
[20] R. Yan,et al. Calreticulin-STAT3 Signaling Pathway Modulates Mitochondrial Function in a Rat Model of Furazolidone-Induced Dilated Cardiomyopathy , 2013, PloS one.
[21] J. Schauer,et al. Mitochondrial Genetic Background Modifies the Relationship between Traffic-Related Air Pollution Exposure and Systemic Biomarkers of Inflammation , 2013, PloS one.
[22] P. Pelicci,et al. The P66Shc/Mitochondrial Permeability Transition Pore Pathway Determines Neurodegeneration , 2013, Oxidative medicine and cellular longevity.
[23] Y. Katayama,et al. Protein kinase C (PKC) isozyme-specific substrates and their design. , 2012, Biotechnology advances.
[24] T. Hibi,et al. p53/p66Shc-mediated signaling contributes to the progression of non-alcoholic steatohepatitis in humans and mice. , 2012, Journal of hepatology.
[25] A. Azghani,et al. Inhibition of protein kinase C attenuates Pseudomonas aeruginosa elastase-induced epithelial barrier disruption. , 2011, American journal of respiratory cell and molecular biology.
[26] Wen-bin Tang,et al. p66Shc mediates high-glucose and angiotensin II-induced oxidative stress renal tubular injury via mitochondrial-dependent apoptotic pathway. , 2010, American journal of physiology. Renal physiology.
[27] John D. Taylor. COPD and the response of the lung to tobacco smoke exposure. , 2010, Pulmonary pharmacology & therapeutics.
[28] C. Steegborn,et al. The Lifespan-regulator p66Shc in mitochondria: redox enzyme or redox sensor? , 2010, Antioxidants & redox signaling.
[29] Yi Na Wang,et al. Genistein protects against UVB-induced senescence-like characteristics in human dermal fibroblast by p66Shc down-regulation. , 2010, Journal of dermatological science.
[30] M. Profita,et al. Chronic obstructive pulmonary disease and neutrophil infiltration: role of cigarette smoke and cyclooxygenase products. , 2010, American journal of physiology. Lung cellular and molecular physiology.
[31] M. Rajendran,et al. p66Shc—a longevity redox protein in human prostate cancer progression and metastasis , 2010, Cancer and Metastasis Reviews.
[32] H. Matsuzaki,et al. Epidermal growth factor receptor phosphorylates protein kinase C {delta} at Tyr332 to form a trimeric complex with p66Shc in the H2O2-stimulated cells. , 2007, Journal of biochemistry.
[33] C. Cool,et al. Lung disease and PKCs. , 2007, Pharmacological research.
[34] S. Minucci,et al. Protein Kinase C ß and Prolyl Isomerase 1 Regulate Mitochondrial Effects of the Life-Span Determinant p66Shc , 2007, Science.
[35] S. Menini,et al. Deletion of p66Shc Longevity Gene Protects Against Experimental Diabetic Glomerulopathy by Preventing Diabetes-Induced Oxidative Stress , 2006, Diabetes.
[36] B. Chorley,et al. Human neutrophil elastase induces hypersecretion of mucin from well-differentiated human bronchial epithelial cells in vitro via a protein kinase C{delta}-mediated mechanism. , 2005, The American journal of pathology.
[37] S. Minucci,et al. Electron Transfer between Cytochrome c and p66Shc Generates Reactive Oxygen Species that Trigger Mitochondrial Apoptosis , 2005, Cell.
[38] S. Shen,et al. Mitochondrial-dependent, reactive oxygen species-independent apoptosis by myricetin: roles of protein kinase C, cytochrome c, and caspase cascade. , 2005, Biochemical pharmacology.
[39] James D Crapo,et al. The Role of Oxidative Stress in Chronic Obstructive Pulmonary Disease , 2004, COPD.
[40] H. Gottlieb,et al. Superoxide organic chemistry within the liposomal bilayer, part II: a correlation between location and chemistry. , 2002, Free radical biology & medicine.
[41] S. Alkan,et al. Caspase activation and disruption of mitochondrial membrane potential during UV radiation-induced apoptosis of human keratinocytes requires activation of protein kinase C , 2002, Cell Death and Differentiation.
[42] B S Polla,et al. Effects of tobacco smoke and benzo[a]pyrene on human endothelial cell and monocyte stress responses. , 2001, American journal of physiology. Heart and circulatory physiology.
[43] Pier Paolo Pandolfi,et al. The p66shc adaptor protein controls oxidative stress response and life span in mammals , 1999, Nature.