Establishment of sandwich ELISA for soluble alpha-Klotho measurement: Age-dependent change of soluble alpha-Klotho levels in healthy subjects.
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M. Econs | Y. Nabeshima | N. Okamoto | N. Kurumatani | K. Ozono | T. Shimada | A. Imura | Yoshihiko Saito | N. Namba | S. Ichikawa | Y. Yamazaki | I. Urakawa | Yukiko Aono | Hisashi Hasegawa | T. Yamashita | T. Sakai | H. Hataya | K. Nakatani | E. Imel | T. Kitaoka | J. Murakami | H. Hasegawa
[1] K. Nakao,et al. Relevant use of Klotho in FGF19 subfamily signaling system in vivo , 2010, Proceedings of the National Academy of Sciences.
[2] E. Sprecher,et al. Familial tumoral calcinosis and the role of O-glycosylation in the maintenance of phosphate homeostasis. , 2009, Biochimica et biophysica acta.
[3] K. White,et al. A homozygous missense mutation in human KLOTHO causes severe tumoral calcinosis. , 2007, Journal of musculoskeletal & neuronal interactions.
[4] Shinzo Tanaka,et al. α-Klotho as a Regulator of Calcium Homeostasis , 2007, Science.
[5] K. Okawa,et al. Klotho converts canonical FGF receptor into a specific receptor for FGF23 , 2006, Nature.
[6] T. Shimada,et al. Vitamin D receptor-independent FGF23 actions in regulating phosphate and vitamin D metabolism. , 2005, American journal of physiology. Renal physiology.
[7] Animesh Nandi,et al. Suppression of Aging in Mice by the Hormone Klotho , 2005, Science.
[8] H. Yamada,et al. Immunohistochemical localization of Klotho protein in brain, kidney, and reproductive organs of mice. , 2004, Cell Structure and Function.
[9] K. Nozaki,et al. Secreted Klotho protein in sera and CSF: implication for post‐translational cleavage in release of Klotho protein from cell membrane , 2004, FEBS letters.
[10] K. Kamiya,et al. Sinoatrial Node Dysfunction and Early Unexpected Death of Mice With a Defect of klotho Gene Expression , 2004, Circulation.
[11] Y. Takeuchi,et al. FGF‐23 Is a Potent Regulator of Vitamin D Metabolism and Phosphate Homeostasis , 2003, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[12] K. Fukuda,et al. Klotho, a gene related to a syndrome resembling human premature aging, functions in a negative regulatory circuit of vitamin D endocrine system. , 2003, Molecular endocrinology.
[13] K. White,et al. FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting. , 2003, The Journal of clinical investigation.
[14] Y. Takeuchi,et al. Increased circulatory level of biologically active full-length FGF-23 in patients with hypophosphatemic rickets/osteomalacia. , 2002, The Journal of clinical endocrinology and metabolism.
[15] S. Takeda,et al. Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[16] A. Tamori,et al. Severely reduced production of klotho in human chronic renal failure kidney. , 2001, Biochemical and biophysical research communications.
[17] T. Meitinger,et al. Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23 , 2000, Nature Genetics.
[18] K. Nakamura,et al. Establishment of the anti-Klotho monoclonal antibodies and detection of Klotho protein in kidneys. , 2000, Biochemical and biophysical research communications.
[19] Tadashi Kaname,et al. Mutation of the mouse klotho gene leads to a syndrome resembling ageing , 1997, Nature.