Matrilysin [MMP-7] expression selects for cells with reduced sensitivity to apoptosis.
暂无分享,去创建一个
[1] G. Bergers,et al. Matrix metalloproteinases as emerging targets in anticancer therapy: status and prospects , 2000 .
[2] J. Trapani,et al. Proapoptotic functions of cytotoxic lymphocyte granule constituents in vitro and in vivo. , 2000, Current opinion in immunology.
[3] N. Restifo. Not so Fas: Re-evaluating the mechanisms of immune privilege and tumor escape , 2000, Nature Medicine.
[4] W. Earnshaw,et al. Induction of apoptosis by cancer chemotherapy. , 2000, Experimental cell research.
[5] D. Green,et al. Regulation of FasL by NF-κB and AP-1 in Fas-dependent Thymineless Death of Human Colon Carcinoma Cells* , 2000, The Journal of Biological Chemistry.
[6] S. Moe,et al. Opposing Effects of Transmembrane and Soluble FAS Ligand Expression on Inflammation and Tumor Cell Survival , 2000, The Journal of experimental medicine.
[7] S. Lowe,et al. Apoptosis in cancer. , 2000, Carcinogenesis.
[8] B. Fingleton,et al. Matrix metalloproteinases: biologic activity and clinical implications. , 2000, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[9] B. Fingleton,et al. Matrix metalloproteinase-7-dependent release of tumor necrosis factor-alpha in a model of herniated disc resorption. , 2000, The Journal of clinical investigation.
[10] A. Strasser,et al. Ionizing Radiation and Chemotherapeutic Drugs Induce Apoptosis in Lymphocytes in the Absence of FAS or Fadd/Mort1 Signaling Implications for Cancer Therapy , 2000 .
[11] B. Fingleton,et al. The metalloproteinase matrilysin proteolytically generates active soluble Fas ligand and potentiates epithelial cell apoptosis , 1999, Current Biology.
[12] A. Dobrovic,et al. Promoter region methylation does not account for the frequent loss of expression of the Fas gene in colorectal carcinoma , 1999, British Journal of Cancer.
[13] I. Svane,et al. The role of cytotoxic T-lymphocytes in the prevention and immune surveillance of tumors— lessons from normal and immunodeficient mice , 1999, Medical oncology.
[14] A. Younes,et al. Overexpression of Fas ligand (FasL) during malignant transformation in the large bowel and in Barrett's metaplasia of the esophagus. , 1999, Human pathology.
[15] R. Offringa,et al. Immune Escape of Tumors in Vivo by Expression of Cellular Flice-Inhibitory Protein , 1999, The Journal of experimental medicine.
[16] Ingo Schmitz,et al. Differential Modulation of Apoptosis Sensitivity in CD95 Type I and Type II Cells* , 1999, The Journal of Biological Chemistry.
[17] D. Smith,et al. Differential activity of soluble versus cellular Fas ligand: regulation by an accessory molecule. , 1999, Cellular immunology.
[18] F. Shanahan,et al. Expression of Fas (CD95/APO-1) Ligand by Human Breast Cancers: Significance for Tumor Immune Privilege , 1999, Clinical Diagnostic Laboratory Immunology.
[19] W. Dalton,et al. Myeloma cells selected for resistance to CD95-mediated apoptosis are not cross-resistant to cytotoxic drugs: evidence for independent mechanisms of caspase activation. , 1999, Blood.
[20] H. S. Kim,et al. Alterations of Fas (Apo-1/CD95) gene in cutaneous malignant melanoma. , 1999, American Journal of Pathology.
[21] W. Stetler-Stevenson,et al. Matrix metalloproteinases in angiogenesis: a moving target for therapeutic intervention. , 1999, The Journal of clinical investigation.
[22] F. Dallegri,et al. Soluble Fas ligand is chemotactic for human neutrophilic polymorphonuclear leukocytes. , 1999, Journal of immunology.
[23] L. Matrisian,et al. Differential expression of matrilysin and cyclooxygenase‐2 in intestinal and colorectal neoplasms , 1999, Molecular carcinogenesis.
[24] L. Matrisian,et al. The matrix metalloproteinase matrilysin influences early-stage mammary tumorigenesis. , 1998, Cancer research.
[25] L. Owen-Schaub,et al. Fas and Fas Ligand Interactions Suppress Melanoma Lung Metastasis , 1998, The Journal of experimental medicine.
[26] F. Shanahan,et al. Fas ligand expression in primary colon adenocarcinomas: evidence that the Fas counterattack is a prevalent mechanism of immune evasion in human colon cancer , 1998, The Journal of pathology.
[27] P. Walker,et al. Tumor expression of Fas ligand (CD95L) and the consequences. , 1998, Current opinion in immunology.
[28] D. Ferrari,et al. Apoptosis signaling by death receptors. , 1998, European journal of biochemistry.
[29] C. Brady,et al. The Fas counterattack in vivo: apoptotic depletion of tumor-infiltrating lymphocytes associated with Fas ligand expression by human esophageal carcinoma. , 1998, Journal of immunology.
[30] J. Tschopp,et al. Conversion of Membrane-bound Fas(CD95) Ligand to Its Soluble Form Is Associated with Downregulation of Its Proapoptotic Activity and Loss of Liver Toxicity , 1998, The Journal of experimental medicine.
[31] S. Nagata,et al. Downregulation of Fas ligand by shedding , 1998, Nature Medicine.
[32] D. Green,et al. Fas ligand- mediated killing by intestinal intraepithelial lymphocytes. Participation in intestinal graft-versus-host disease. , 1998, The Journal of clinical investigation.
[33] R. Nagle,et al. Cleavage of β4 Integrin by Matrilysin , 1997 .
[34] L. Matrisian,et al. Changing views of the role of matrix metalloproteinases in metastasis. , 1997, Journal of the National Cancer Institute.
[35] T. Ishikawa,et al. Matrilysin gene expression in sporadic and familial colorectal adenomas , 1997, Molecular carcinogenesis.
[36] P. Walker,et al. Role of Fas ligand (CD95L) in immune escape: the tumor cell strikes back. , 1997, Journal of immunology.
[37] B. Hogan,et al. Intestinal tumorigenesis is suppressed in mice lacking the metalloproteinase matrilysin. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[38] P. Galle,et al. Lymphocyte apoptosis induced by CD95 (APO–1/Fas) ligand–expressing tumor cells — A mechanism of immune evasion? , 1996, Nature Medicine.
[39] J. Tschopp,et al. Melanoma Cell Expression of Fas(Apo-1/CD95) Ligand: Implications for Tumor Immune Escape , 1996, Science.
[40] C. Kitanaka,et al. Apoptosis in cancer. , 1996, Human cell.
[41] L. Matrisian,et al. Expression of most matrix metalloproteinase family members in breast cancer represents a tumor-induced host response. , 1996, The American journal of pathology.
[42] A. Mes-Masson,et al. Decreased Fas antigen receptor expression in testicular tumor cell lines derived from polyomavirus large T-antigen transgenic mice. , 1996, Oncogene.
[43] L. Matrisian,et al. Matrilysin: an epithelial matrix metalloproteinase with potentially novel functions. , 1996, The international journal of biochemistry & cell biology.
[44] R. Nagle,et al. Matrilysin expression in human prostate carcinoma , 1996, Molecular carcinogenesis.
[45] Jack P. Witty,et al. Expression and localization of matrix‐degrading metalloproteinases during colorectal tumorigenesis , 1994, Molecular carcinogenesis.
[46] H Hengartner,et al. Fas and perforin pathways as major mechanisms of T cell-mediated cytotoxicity. , 1994, Science.
[47] K. Koretz,et al. Expression of APO‐1 (CD95), a member of the NGF/TNF receptor superfamily, in normal and neoplastic colon epithelium , 1994, International journal of cancer.
[48] S. Goff,et al. Construction and use of a safe and efficient amphotropic packaging cell line. , 1988, Virology.
[49] J. Abecassis,et al. The collagenase gene family in humans consists of at least four members. , 1988, The Biochemical journal.
[50] H. Smith,et al. Epithelial cell cultures from normal glandular tissue of mice. , 1974, Journal of the National Cancer Institute.
[51] M. Mareel,et al. Release of an invasion promoter E-cadherin fragment by matrilysin and stromelysin-1. , 2001, Journal of cell science.
[52] I. Stamenkovic,et al. Matrix metalloproteinase-7-mediated cleavage of Fas ligand protects tumor cells from chemotherapeutic drug cytotoxicity. , 2001, Cancer research.
[53] T. Gajewski,et al. The immune system as anti-tumor sentinel: molecular requirements for an anti-tumor immune response. , 1999, Critical reviews in oncogenesis.
[54] R. Nagle,et al. Cleavage of beta 4 integrin by matrilysin. , 1997, Experimental cell research.
[55] J. Ritz,et al. Functional consequences of APO-1/Fas (CD95) antigen expression by normal and neoplastic hematopoietic cells. , 1995, Leukemia & lymphoma.
[56] L. Matrisian,et al. Expression and localization of the matrix metalloproteinase pump‐1 (MMP‐7) in human gastric and colon carcinomas , 1991, Molecular carcinogenesis.
[57] Luzhe Sun,et al. Isolation and characterization of a spontaneously transformed malignant mouse mammary epithelial cell line in culture. , 1998, Carcinogenesis.